Dillmann, Wolfgang H.

Myofibroblasts revert to an inactive phenotype during regression of liver fibrosis

Kisseleva, T., Cong, M., Paik, Y., Scholten, D., Jiang, C., Benner, C., Iwaisako, K., Moore-Morris, T., Scott, B., Tsukamoto, H., et al. (2012) · Proceedings of the National Academy of Sciences

Liver physiology and pathologyLiver Disease Diagnosis and Treatment

Cited by 805

Biochemical basis of thyroid hormone action in the heart.

Dillmann, W. H. (1990) · The American Journal of Medicine

Thyroid Disorders and TreatmentsCardiac electrophysiology and arrhythmias

Cited by 338

Mitochondrial fragmentation and superoxide anion production in coronary endothelial cells from a mouse model of type 1 diabetes

Makino, A., Scott, B. T., & Dillmann, W. H. (2010) · Diabetologia

Mitochondrial Function and PathologyAutophagy in Disease and Therapy

Cited by 223

Alterations in mitochondrial function and cytosolic calcium induced by hyperglycemia are restored by mitochondrial transcription factor A in cardiomyocytes

Suarez, J., Hu, Y., Makino, A., Fricovsky, E., Wang, H., & Dillmann, W. H. (2008) · American Journal of Physiology-Cell Physiology

Mitochondrial Function and PathologyCardiac Ischemia and Reperfusion

Cited by 77

Dilated cardiomyopathy in transgenic mice expressing a mutant A subunit of protein phosphatase 2A

Brewis, N., Ohst, K., Fields, K., Rapacciuolo, A., Chou, D., Bloor, C., Dillmann, W., Rockman, H., & Walter, G. (2000) · American Journal of Physiology-Heart and Circulatory Physiology

Cardiomyopathy and Myosin StudiesSignaling Pathways in Disease

Cited by 46

Thyroid hormone inhibits ERK phosphorylation in pressure overload-induced hypertrophied mouse hearts through a receptor-mediated mechanism

Suarez, J., Scott, B. T., Suarez-Ramirez, J. A., Chavira, C. V., & Dillmann, W. H. (2010) · American Journal of Physiology-Cell Physiology

Cardiac Fibrosis and RemodelingThyroid Disorders and Treatments

Cited by 30