Saleem, Moin A.

Transgenic expression of human APOL1 risk variants in podocytes induces kidney disease in mice

Beckerman, P., Bi-Karchin, J., Park, A. S. D., Qiu, C., Dummer, P. D., Soomro, I., Boustany-Kari, C. M., Pullen, S. S., Miner, J. H., Hu, C.-A. A., et al. (2017) · Nature Medicine

Renal Diseases and GlomerulopathiesChronic Kidney Disease and Diabetes

Cited by 344

Prorenin Receptor Is Essential for Normal Podocyte Structure and Function

Oshima, Y., Kinouchi, K., Ichihara, A., Sakoda, M., Kurauchi-Mito, A., Bokuda, K., Narita, T., Kurosawa, H., Sun-Wada, G.-H., Wada, Y., et al. (2011) · Journal of the American Society of Nephrology

Renal Diseases and GlomerulopathiesRenin-Angiotensin System Studies

Cited by 173

VEGF regulates local inhibitory complement proteins in the eye and kidney

Keir, L. S., Firth, R., Aponik, L., Feitelberg, D., Sakimoto, S., Aguilar, E., Welsh, G. I., Richards, A., Usui, Y., Satchell, S. C., et al. (2016) · Journal of Clinical Investigation

Complement system in diseasesRenal Diseases and Glomerulopathies

Cited by 154

Evidence for activation of the unfolded protein response in collagen IV nephropathies

Pieri, M., Stefanou, C., Zaravinos, A., Erguler, K., Stylianou, K., Lapathitis, G., Karaiskos, C., Savva, I., Paraskeva, R., Dweep, H., et al. (2014) · Journal of the American Society of Nephrology

Renal Diseases and GlomerulopathiesEndoplasmic Reticulum Stress and Disease

Cited by 94

MAGI2 Mutations Cause Congenital Nephrotic Syndrome

Bierzynska, A., Soderquest, K., Dean, P., Colby, E., Rollason, R., Jones, C., Inward, C. D., McCarthy, H. J., Simpson, M. A., Lord, G. M., et al. (2017) · Journal of the American Society of Nephrology

Renal Diseases and GlomerulopathiesRenal and related cancers

Cited by 82

Expression of CCN1 (CYR61) in developing, normal, and diseased human kidney

Sawai, K., Mukoyama, M., Mori, K., Kasahara, M., Koshikawa, M., Yokoi, H., Yoshioka, T., Ogawa, Y., Sugawara, A., Nishiyama, H., et al. (2007) · American Journal of Physiology-Renal Physiology

Renal Diseases and GlomerulopathiesConnective Tissue Growth Factor Research

Cited by 33

Disrupted apolipoprotein L1-miR193a axis dedifferentiates podocytes through autophagy blockade in an APOL1 risk milieu

Kumar, V., Ayasolla, K., Jha, A., Mishra, A., Vashistha, H., Lan, X., Qayyum, M., Chinnapaka, S., Purohit, R., Mikulak, J., et al. (2019) · American Journal of Physiology-Cell Physiology

Renal Diseases and GlomerulopathiesAutophagy in Disease and Therapy

Cited by 29