Harada, Takayuki
Tokyo Metropolitan Institute of Medical Science
6 papers on CoChatORCID 0000-0001-6167-0997
Most cited papers
- Verified
Microglia-Müller glia cell interactions control neurotrophic factor production during light-induced retinal degeneration
Harada, T., Harada, C., Kohsaka, S., Wada, E., Yoshida, K., Ohno, S., Mamada, H., Tanaka, K., Parada, L. F., & Wada, K. (2002) · The Journal of Neuroscience
Retinal Development and DisordersRetinal Diseases and Treatments
Cited by 397
- Verified
Dock3 induces axonal outgrowth by stimulating membrane recruitment of the WAVE complex
Namekata, K., Harada, C., Taya, C., Guo, X., Kimura, H., Parada, L. F., & Harada, T. (2010) · Proceedings of the National Academy of Sciences
Axon Guidance and Neuronal SignalingNerve injury and regeneration
Cited by 84
- Verified
Dock GEFs and their therapeutic potential: Neuroprotection and axon regeneration
Namekata, K., Kimura, A., Kawamura, K., Harada, C., & Harada, T. (2014) · Progress in Retinal and Eye Research
Glaucoma and retinal disordersProtein Kinase Regulation and GTPase Signaling
Cited by 57
- Verified
DOCK8 is expressed in microglia, and it regulates microglial activity during neurodegeneration in murine disease models
Namekata, K., Guo, X., Kimura, A., Arai, N., Harada, C., & Harada, T. (2019) · Journal of Biological Chemistry
Neuroinflammation and Neurodegeneration MechanismsMultiple Sclerosis Research Studies
Cited by 40
- Verified
Roles of the DOCK-D family proteins in a mouse model of neuroinflammation
Namekata, K., Guo, X., Kimura, A., Azuchi, Y., Kitamura, Y., Harada, C., & Harada, T. (2020) · Journal of Biological Chemistry
Multiple Sclerosis Research StudiesNeuroinflammation and Neurodegeneration Mechanisms
Cited by 23
- Verified
Variants in DOCK3 cause developmental delay and hypotonia
Wiltrout, K., Ferrer, A., van de Laar, I., Namekata, K., Harada, T., Klee, E. W., Zimmerman, M. T., Cousin, M. A., Kempainen, J. L., Babovic-Vuksanovic, D., et al. (2019) · European Journal of Human Genetics
Genetics and Neurodevelopmental DisordersGenomics and Rare Diseases
Cited by 17
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