Katholnig, Karl

Chronic signaling via the metabolic checkpoint kinase mTORC1 induces macrophage granuloma formation and marks sarcoidosis progression

Linke, M., Pham, H. T. T., Katholnig, K., Schnöller, T., Miller, A., Demel, F., Schütz, B., Rosner, M., Kovacic, B., Sukhbaatar, N., et al. (2017) · Nature Immunology

Sarcoidosis and Beryllium Toxicity ResearchTuberous Sclerosis Complex Research

Cited by 286

Serum Amyloid A in Uremic HDL Promotes Inflammation

Weichhart, T., Kopecky, C., Kubicek, M., Haidinger, M., Döller, D., Katholnig, K., Suarna, C., Eller, P., Tölle, M., Gerner, C., et al. (2012) · Journal of the American Society of Nephrology

Dialysis and Renal Disease ManagementCholesterol and Lipid Metabolism

Cited by 201

Inhibition of mTOR blocks the anti-inflammatory effects of glucocorticoids in myeloid immune cells

Weichhart, T., Haidinger, M., Katholnig, K., Kopecky, C., Poglitsch, M., Lassnig, C., Rosner, M., Zlabinger, G. J., Hengstschläger, M., Müller, M., et al. (2011) · Blood

PI3K/AKT/mTOR signaling in cancerImmune Response and Inflammation

Cited by 141

Inverse data-driven modeling and multiomics analysis reveals Phgdh as a metabolic checkpoint of macrophage polarization and proliferation

Wilson, J. L., Nägele, T., Linke, M., Demel, F., Fritsch, S. D., Mayr, H. K., Cai, Z., Katholnig, K., Sun, X., Fragner, L., et al. (2020) · Cell Reports

Immune cells in cancerPI3K/AKT/mTOR signaling in cancer

Cited by 97