The DOI matches its Crossref record.
Obesity and Essential Hypertension: Hemodynamics, Intravascular Volume, Sodium Excretion, and Plasma Renin Activity
Abstract
Systemic hemodynamics, intravascular volume, and plasma renin activity were determined in 135 lean, mildly obese, or distinctly overweight subjects who were normotensive or had borderline or established essential hypertension. Cardiac output (but not index) was higher and peripheral resistance lower in obese than in lean subjects, except in borderline hypertension. intravascular volume was increased in obese patients, and more so when corrected for body height; correction for body weight led to relative volume contraction. Intravascular volume correlated directly with cardiac output in the entire population, as well as in the subgroups. Intravascular volume correlated inversely with total peripheral resistance in all subjects and in each subgroup. Both correlations remained significant when an approximation was used to correct influences of obesity on total blood volume. Sodium excretion was higher in obese than in lean subjects. Thus, despite the expanded intravascular volume in obesity, the pathophysiologic relationship between systemic hemodynamics and intravascular volumes remains unchanged. Relatively low peripheral resistance in obesity may decrease the risk of systemic vascular disease. Nevertheless, since circulating volume is increased, the greater venous return adds an additional load to a left ventricle that is already burdened by a high afterload caused by arterial hypertension. (Arch Intern Med141:81-85, 1981)
Overview
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
In the citing paper
“The metabolic, endocrine, cardiovascular, and renal changes caused by dietary-induced obesity in these experimental animals closely mimic the changes observed in obese humans5,13–16 (Table 1).”
From Obesity-Induced Hypertension: Interaction of Neurohumoral and Renal Mechanisms (2015) · cites this paper as [16]
Citations
Cited by
Sorted by citations
Items 101–200 of 207
Obesity in the pediatric patient: cardiovascular complications
Daniels, S. R. (2001) · Progress in Pediatric Cardiology · doi:10.1016/s1058-9813(00)00069-2 · Cited by 30
Hop (Humulus lupulus L.) extract inhibits obesity in mice fed a high-fat diet over the long term
Sumiyoshi, M., & Kimura, Y. (2012) · British Journal Of Nutrition · doi:10.1017/s000711451200061x · Cited by 31
“Furthermore, obesity is associated with expanded circulatory volume and an increased extracellular fluid ratio, and enhanced body Na content (29,30) .”
Berton, M., Bettonte, S., Stader, F., Battegay, M., & Marzolini, C. (2022) · Clinical Pharmacokinetics · doi:10.1007/s40262-022-01132-3 · Cited by 33
Impact of age, race, and obesity on hypertensive mechanisms and therapy
Weir, M. R. (1991) · The American Journal of Medicine · doi:10.1016/0002-9343(91)90479-h · Cited by 29
Prabhu, S., Deng, H., Cross, T.-W., Shahoei, S. H., Konopka, C. J., Medina, N. G., Applegate, C. C., Wallig, M. A., Dobrucki, L. W., Nelson, E. R., et al. (2020) · Biomaterials Science · doi:10.1039/d0bm01142h · Cited by 28
Matsuda, A., Matsutani, T., Sasajima, K., Furukawa, K., Tajiri, T., Tamura, K., & Kogo, H. (2008) · Journal of Surgical Research · doi:10.1016/j.jss.2008.09.007 · Cited by 28
“It is well known that increased body weight, namely, the accumulation of adipose tissue, is a risk factor for the development of various metabolic disorders, including hypertension, diabetes mellitus, dyslipidemia, and the metabolic syndrome [10, 11].”
BackgroundManagement of obesity cardiomyopathy
Alpert, M., Fraley, M. A., Birchem, J. A., & Senkottaiyan, N. (2005) · Expert Review of Cardiovascular Therapy · doi:10.1586/14779072.3.2.225 · Cited by 26
Engeli, S., Boschmann, M., Frings, P., et al. (2006) · Hypertension · doi:10.1161/01.hyp.0000248837.88749.18 · Cited by 28
Stress response pattern in obesity and systemic hypertension
Rockstroh, J. K., Schmieder, R. E., Schächinger, H., & Messerli, F. H. (1992) · The American Journal of Cardiology · doi:10.1016/0002-9149(92)90356-4 · Cited by 28
Self-citationImpaired Pressure Natriuresis in Obese Youths
Barbeau, P., Litaker, M. S., & Harshfield, G. A. (2003) · Obesity Research · doi:10.1038/oby.2003.104 · Cited by 27
Tanaka, K. A., Alejo, D. E., Ghoreishi, M., Salenger, R., Fonner, C. E., Ad, N., Whitman, G. J. R., Taylor, B. S., & Mazzeffi, M. A. (2022) · Journal of Cardiothoracic and Vascular Anesthesia · doi:10.1053/j.jvca.2022.03.034 · Cited by 26
The metabolic syndrome and related cardiovascular risk
Ramos, F., Baglivo, H. P., Ramirez, A. J., & Sanchez, R. A. (2001) · Current Hypertension Reports · doi:10.1007/s11906-001-0020-z · Cited by 26
Adiposity and stroke among older adults of low socioeconomic status: the Chicago Stroke Study.
DiPietro, L., Ostfeld, A. M., & Rosner, G. L. (1994) · American Journal of Public Health · doi:10.2105/ajph.84.1.14 · Cited by 26
Schmieder, R. E., & Messerli, F. H. (1987) · Medical Clinics of North America · doi:10.1016/s0025-7125(16)30822-7 · Cited by 29
Self-citationA meta-analysis of the haemodynamics of primary hypertension in children and adults
Li, Y., Haseler, E., McNally, R., et al. (2022) · Journal of Hypertension · doi:10.1097/hjh.0000000000003326 · Cited by 24
“…4.424 0.000 2.00 Safar et al. 1975 -2.420 5.792 33.542 -13.771 8.931 -0.418 0.676 Messerli et al. 1981 1.540 4.988 24.885 -8.237 11.317 0.309 0.758 Temmar et al. 1981 7.950 6.298 39.663 -4.394 20.294 1.262 0.207 London et al. 1984 -1.800 3.130 9.795 -7.934 4.334 -0.575 0.565 Bouthier et al. 1985…”
InfluentialMR Imaging of the Lumbar Spine: Relation of Posterior Soft-Tissue Edema-Like Signal and Body Weight
Shi, H., Schweitzer, M. E., Carrino, J. A., & Parker, L. (2003) · American Journal of Roentgenology · doi:10.2214/ajr.180.1.1800081 · Cited by 24
“Second, obesity is associated with expanded circulatory volume [ 12 ], which in turn could translate to increased intravascular pressure.”
BackgroundHall, M. E., & Hall, J. E. (2017) · Elsevier eBooks · doi:10.1016/b978-0-323-42973-3.00005-6 · Cited by 36
Individualization of Antihypertensive Therapy: An Approach Based on Hemodynamics and Age
Messerli, F. H. (1981) · The Journal of Clinical Pharmacology · doi:10.1002/j.1552-4604.1981.tb05659.x · Cited by 24
Self-citationPhysiologic considerations in left ventricular hypertrophy
Frohlich, E. D. (1983) · The American Journal of Medicine · doi:10.1016/0002-9343(83)90112-2 · Cited by 24
Clinical utility of postoperative hemoglobin level testing following total laparoscopic hysterectomy
Chamsy, D. J., Louie, M. Y., Lum, D. A., Phelps, A. L., & Mansuria, S. M. (2014) · American Journal of Obstetrics and Gynecology · doi:10.1016/j.ajog.2014.04.003 · Cited by 23
Whaley‐Connell, A. T., Pavey, B. S., Chaudhary, K. R., Saab, G., & Sowers, J. R. (2007) · Therapeutic Advances in Cardiovascular Disease · doi:10.1177/1753944707082697 · Cited by 22
“In obesity, there is a state of dysregulation and resultant abnormal vascular responsiveness modulated by insulin, which works as a vasodilator by inhibiting voltagegated Ca2+ influx [Doflou et al. 1995; Messerli et al. 1981; Frohlich et al. 1983].”
BackgroundObesity, Hypertension, and the Heart
Good, D., Morse, S. A., Ventura, H. O., & Reisin, E. (2008) · Journal of the CardioMetabolic Syndrome · doi:10.1111/j.1559-4572.2008.00011.x · Cited by 22
Evaluation of Arterial Compliance in Humans
Giannattasio, C., Failla, M., Mangoni, A. A., et al. (1996) · Clinical and Experimental Hypertension · doi:10.3109/10641969609088968 · Cited by 23
Cardiac adaptation to obesity and hypertension after heart transplantation
Ventura, H. O., Johnson, M. R., Grusk, B., et al. (1992) · Journal of the American College of Cardiology · doi:10.1016/0735-1097(92)90051-n · Cited by 23
Kennedy, H., Haynes, S. L., & Shelton, C. (2022) · British Journal of Anaesthesia · doi:10.1016/j.bja.2022.08.011 · Cited by 22
BUSKIRK, E. R. (1994) · Elsevier eBooks · doi:10.1016/b978-1-4831-7818-9.50024-1 · Cited by 22
Relation Between Lead Exposure and Trends in Blood Pressure in Children
Zachariah, J. P. V., Wang, Y., Penny, D. J., et al. (2018) · The American Journal of Cardiology · doi:10.1016/j.amjcard.2018.08.033 · Cited by 21
Sex Differences in Blood Pressure Hemodynamics in Middle-Aged Adults With Overweight and Obesity
Syme, C. A., Shin, J., Richer, L., et al. (2019) · Hypertension · doi:10.1161/hypertensionaha.119.13058 · Cited by 21
The Impact of Weight Loss on Cardiac Structure and Function in Obese Patients
Syed, M., Rosati, C., Torosoff, M. T., El-Hajjar, M. C., Feustel, P. J., Alger, S. A., Singh, P. T., & Fein, S. (2008) · Obesity Surgery · doi:10.1007/s11695-008-9645-1 · Cited by 20
Frohlich, E. D., & Sušić, D. (2008) · Current Hypertension Reports · doi:10.1007/s11906-008-0028-8 · Cited by 20
Obesity and Preclinical Changes of Cardiac Geometry and Function
Sung, J. K., & Kim, J. (2010) · Korean Circulation Journal · doi:10.4070/kcj.2010.40.2.55 · Cited by 18
Bonafini, S., Giontella, A., Tagetti, A., Montagnana, M., Benati, M., Danese, E., Minuz, P., Maffeis, C., Antoniazzi, F., & Fava, C. (2019) · Hypertension Research · doi:10.1038/s41440-018-0173-7 · Cited by 20
van der Meer, N. J. M., de Vries, J. P. P. M., Schreuder, W. O., Bulder, E. R., Eysman, L., & VRIES, P. M. J. M. D. (1997) · Acta Anaesthesiologica Scandinavica · doi:10.1111/j.1399-6576.1997.tb04770.x · Cited by 19
The importance of hemodynamic considerations in essential hypertension
Egan, B. M., & Schmouder, R. (1988) · American Heart Journal · doi:10.1016/0002-8703(88)90556-x · Cited by 19
Dustan, H. P. (1991) · Primary Care Clinics in Office Practice · doi:10.1016/s0095-4543(21)00342-0 · Cited by 19
Pathophysiology of Obesity—Induced Hypertension and Target Organ Damage
Hall, J. E., da Silva, A. A., Brandon, E. L., Stec, D. E., Ying, Z., & Jones, D. W. (2007) · Elsevier eBooks · doi:10.1016/b978-0-323-03961-1.50041-6 · Cited by 18
Cardiometabolic crosstalk in obesity-associated arterial hypertension
Jordan, J., & Birkenfeld, A. L. (2016) · Reviews in Endocrine and Metabolic Disorders · doi:10.1007/s11154-016-9348-1 · Cited by 17
“[16] Obese individuals also exhibited an increase in intravascular volume.[16] Cardiac output determined by the Fick technique [17] or radionuclide ventriculography [18] was also increased in obese individuals.”
BackgroundMethodInfluentialKikuchi, K., Iimura, O., Yamaji, I., Shibata, S., Nishimura, M., Aoki, K., Nozawa, A., Hasegawa, T., Honma, C., Kobayakawa, H., et al. (1988) · American Journal of Hypertension · doi:10.1093/ajh/1.1.31 · Cited by 17
Achievements in hypertension: A 25 year overview
Frohlich, E. D. (1983) · Journal of the American College of Cardiology · doi:10.1016/s0735-1097(83)80024-2 · Cited by 16
Treatment of Hypertension in Obese Patients
Kidambi, S., & Kotchen, T. A. (2013) · American Journal of Cardiovascular Drugs · doi:10.1007/s40256-013-0008-5 · Cited by 16
Non-invasive assessment of maternal hemodynamics in early pregnancy
van der Graaf, A. M., Zeeman, G. G., Groen, H., Roberts, C. T., & Dekker, G. (2013) · Pregnancy Hypertension · doi:10.1016/j.preghy.2013.07.005 · Cited by 16
“Body weight According to various studies the hemodynamic profile of obese subjects is characterized by high CO and inappropriately normal peripheral resistance [36,37].”
BackgroundSchutte, R., Huisman, H. W., Schutte, A. E., & Malan, N. T. (2005) · Journal of Human Hypertension · doi:10.1038/sj.jhh.1001922 · Cited by 16
Patel, D. A., Srinivasan, S. R., Chen, W., et al. (2009) · The American Journal of Cardiology · doi:10.1016/j.amjcard.2009.02.063 · Cited by 16
Santos, J. L. F., Salemi, V. M. C., Picard, M. H., Mady, C., & Coelho, O. R. (2010) · Obesity · doi:10.1038/oby.2010.253 · Cited by 15
Beyond Weight Loss: the Emerging Role of Incretin-Based Treatments in Cardiometabolic HFpEF
Capone, F., Nambiar, N., & Schiattarella, G. G. (2024) · Current Opinion in Cardiology · doi:10.1097/hco.0000000000001117 · Cited by 15
“Specific to obese patients with HFpEF is plasma volume expansion, a condition caused by excessive systemic vasodilation and sodium retention [28], which is directly correlated with elevation in cardiac filling pressures [23].”
BackgroundTherapeutic Considerations in the Treatment of Obesity Hypertension
Wofford, M. R., Davis, M. M., Harkins, K. G., King, D. S., Wyatt, S. B., & Jones, D. W. (2002) · Journal of Clinical Hypertension · doi:10.1111/j.1524-6175.2002.00502.x · Cited by 15
Cao, J. (2018) · Annals of the New York Academy of Sciences · doi:10.1111/nyas.13936 · Cited by 16
“Other major findings of this study are (1) CR decreased serum leptin concentration and was more effective in reducing adiposity compared to Ex without CR; (2) CR decreased serum concentrations of TRAP, IGF-1, and osteocalcin, and negatively affected bone structure; (3) Ex decreased serum concentration of TRAP; and (4)…”
ResultSex-Dependence of Body Fat Distribution in Patients with Obesity and Hypertension
Raison, J., Achimastos, A. D., & Safar, M. (1992) · Clinical and Experimental Hypertension · doi:10.3109/10641969209036203 · Cited by 15
Impaired blood pressure compensation following hemorrhage in conscious obese Zucker rats
Xiang, L., Clemmer, J. S., Lu, S., & Mittwede, P. N. (2013) · Life Sciences · doi:10.1016/j.lfs.2013.06.006 · Cited by 14
“However, hypertension is not always present in obese subjects, and TPR can be normal or even decreased despite an overall elevated sympathetic activity (Messerli et al., 1981).”
Obesity and heart failure: exploring the cardiometabolic axis
Rayner, J. J., Abdesselam, I., Pan, J., Lewis, A., & Rider, O. J. (2025) · Cardiovascular Research · doi:10.1093/cvr/cvaf090 · Cited by 15
“Preclinical obesity, and in particular increased visceral adiposity, is associated with increased circulating volume but also disproportionate systemic vasodilatation, 64 due in part to secretion of adipose tissue derived hormones such as leptin.”
Risks for Arterial Hypertension
Schmieder, R. E., Messerli, F. H., & Rüddel, H. (1986) · Cardiology Clinics · doi:10.1016/s0733-8651(18)30635-0 · Cited by 13
Self-citationMetabolic fuel utilization and subclinical atherosclerosis in overweight/obese subjects
Montalcini, T., Gazzaruso, C., Ferro, Y., Migliaccio, V., Rotundo, S., Castagna, A., & Pujia, A. (2012) · Endocrine · doi:10.1007/s12020-012-9845-2 · Cited by 12
Gillum, R. F., Prineas, R. J., Sopko, G. S., et al. (1983) · American Heart Journal · doi:10.1016/0002-8703(83)90533-1 · Cited by 12
Use of diuretic agents in obese or black patients with systemic hypertension
Messerli, F. H., & Schmieder, R. E. (1986) · The American Journal of Cardiology · doi:10.1016/0002-9149(86)90876-3 · Cited by 12
Self-citationBiological Pathways in Adolescent Aortic Stiffness
Zachariah, J. P. V., Wang, Y., Newburger, J. W., de Ferranti, S. D., Mitchell, G. F., & Vasan, R. S. (2021) · Journal of the American Heart Association · doi:10.1161/jaha.120.018419 · Cited by 11
Sex Hormones in Postmenopausal Women Receiving Low‐Dose Hormone Therapy: The Effect of BMI
Lambrinoudaki, I. V., Armeni, E., Rizos, D., Deligeoroglou, E., Kofinakos, P., Kaparos, G., Alexandrou, A. P., Creatsa, M. G., Logothetis, E., & Kouskouni, E. E. (2010) · Obesity · doi:10.1038/oby.2010.232 · Cited by 11
“It is known that intravascular volume is increased in overweight subjects (25,26).”
BackgroundWeir, M. R., & Sowers, J. R. (1988) · The American Journal of Cardiology · doi:10.1016/0002-9149(88)91107-1 · Cited by 11
Bayauli, P. M., M’Buyamba-Kayamba, J.-R., Lemogoum, D., Fagard, R. H., Degaute, J.-P., Ditu, M., Lepira, B. F., & M’Buyamba-Kabangu, J.-R. (2014) · World Journal of Cardiovascular Diseases · doi:10.4236/wjcd.2014.48049 · Cited by 10
“Overweight/obesity is characterized by sympathetic overload responsible for elevated systemic vascular resistance [29] [30] and expanded extracellular volume through which cardiac output and blood pressure can be increased [31] [32].”
BackgroundPathophysiology of Hypertension
Carethers, M., & Blanchette, P. L. (1989) · Clinics in Geriatric Medicine · doi:10.1016/s0749-0690(18)30649-9 · Cited by 10
Feeling the pressure: (patho) physiological mechanisms of weight gain and weight loss in humans
Middlemiss, J. E., & McEniery, C. M. (2016) · Hypertension Research · doi:10.1038/hr.2016.142 · Cited by 9
Kwakernaak, A. J., Roksnoer, L. C. W., Heerspink, H. J. L., Garrelds, I. M., Lochorn, G. A., Andres, J. H. V. E., Klijn, M. A., Kobori, H., Danser, A. H. J., Laverman, G. D., et al. (2017) · PLoS ONE · doi:10.1371/journal.pone.0169258 · Cited by 9
Cardiovascular adaptations to obesity and arterial hypertension: detrimental or beneficial?
Messerli, F. H. (1983) · International Journal of Cardiology · doi:10.1016/0167-5273(83)90069-4 · Cited by 9
Self-citationEliahou, H. E., Laufer, J., Blau, A., et al. (1992) · American Journal of Clinical Nutrition · doi:10.1093/ajcn/56.1.175sa · Cited by 9
Valensi, P. E., L’Hermite, F., Behar, A., et al. (2000) · International Journal of Obesity · doi:10.1038/sj.ijo.0801096 · Cited by 8
Aristizábal, D., Gallo‐Villegas, J., Fernández, R., Restrepo, M. A., Zapata, N., & Correa, M. (2008) · Journal of the CardioMetabolic Syndrome · doi:10.1111/j.1559-4572.2008.00018.x · Cited by 7
Obesity and the kidney connection
Reisin, E. (2001) · American Journal of Kidney Diseases · doi:10.1053/ajkd.2001.28620 · Cited by 7
Ling, H. Z., Jara, P. G., Nicolaides, K. H., & Kametas, N. A. (2022) · Ultrasound in Obstetrics and Gynecology · doi:10.1002/uog.24858 · Cited by 6
Antihypertensive treatment in patients with class 3 obesity
Jordan, J., Boye, S. W., Breton, S. L., Keefe, D. L., Engeli, S., & PRESCOTT, M. F. (2012) · Therapeutic Advances in Endocrinology and Metabolism · doi:10.1177/2042018812445573 · Cited by 6
“Consequently, sodium retention, volume expansion, and increases in cardiac output may ensue [Messerli et al. 1981; Stelfox et al. 2006; Strazzullo et al. 2001].”
BackgroundThe Relationship of Childhood Obesity with Cardiomyopathy and Heart Failure
Qureshi, M. Y., Wilkinson, J. D., & Lipshultz, S. E. (2012) · Springer eBooks · doi:10.1007/978-1-4471-2366-8_10 · Cited by 5
Abe, C., Nagai, Y., Yamaguchi, A., Aoki, H., Shimizu, S., Akiyama, T., Kawada, T., Sugimachi, M., & Morita, H. (2015) · American Journal of Physiology-Heart and Circulatory Physiology · doi:10.1152/ajpheart.00697.2014 · Cited by 5
Cao, J., Gregoire, B. R., Michelsen, K. G., et al. (2021) · Journal of Nutrition · doi:10.1093/jn/nxab173 · Cited by 5
Heterogeneous pathophysiology of essential hypertension: Implications for therapy
Messerli, F. H., Schmieder, R. E., & Núñez, B. D. (1986) · American Heart Journal · doi:10.1016/0002-8703(86)90498-9 · Cited by 5
Self-citationFrohlich, E. D. (1988) · The American Journal of the Medical Sciences · doi:10.1097/00000441-198804000-00032 · Cited by 4
Cardiovascular Physiology in Obesity
Chan, E. J., & Alpert, M. (2012) · doi:10.1002/9781119962083.ch1 · Cited by 4
Nonpharmacologic approaches to the treatment of systemic hypertension
Frohlich, E. D. (1987) · The American Journal of Cardiology · doi:10.1016/0002-9149(87)90590-x · Cited by 3
Individuals with hypertension have lower plasma volume regardless of weight status
Spitz, R. W., Loprinzi, P. D., & Loenneke, J. P. (2022) · Journal of Human Hypertension · doi:10.1038/s41371-022-00705-w · Cited by 2
Belly fat and resistant hypertension
Jordan, J., & Grassi, G. M. (2010) · Journal of Hypertension · doi:10.1097/hjh.0b013e328339b8d9 · Cited by 2
Izzo, J. L. (2007) · Elsevier eBooks · doi:10.1016/b978-0-323-03961-1.50014-3 · Cited by 3
Adipose Tissue and Blood Pressure Regulation
Cassis, L. A., & Police, S. B. (2010) · Wiley eBooks · doi:10.1002/9783527629527.ch13 · Cited by 2
Blood Volume Changes After Weight Reduction
Beretta-Piccoli, C. (1983) · Annals of Internal Medicine · doi:10.7326/0003-4819-99-4-568_2 · Cited by 2
Nonpharmacologic treatment of hypertension: A review
Lehmann, J. W., & Benson, H. (1982) · General Hospital Psychiatry · doi:10.1016/0163-8343(82)90025-1 · Cited by 2
Environmental factors as a risk for future hypertension
Schmieder, R. E., & Messerli, F. H. (1986) · Stress Medicine · doi:10.1002/smi.2460020310 · Cited by 2
Self-citationHypertension Update for the 1980s
Lowenthal, D. T., & Swartz, C. D. (1985) · Primary Care Clinics in Office Practice · doi:10.1016/s0095-4543(21)01243-4 · Cited by 2
Dustan, H. P. (2009) · Clinical Cardiology · doi:10.1002/clc.4960121313 · Cited by 1
Obesity can break your heart and more so when you are a woman
Haufe, S., & Jordan, J. (2011) · Journal of Hypertension · doi:10.1097/hjh.0b013e32834905ee · Cited by 1
Obesity and Hypertension: Impact on Cardiovascular and Renal Systems
Hall, J. E., Jones, D. W., Kuo, J. J., da Silva, A. A., Liu, J., & Tallam, L. S. (2005) · Elsevier eBooks · doi:10.1016/b978-0-7216-0258-5.50134-4 · Cited by 1
Potnuri, A. G., Kallamadi, P. R., Suresh, P., Husain, G. M., Kazmi, M. H., & Nemani, H. (2021) · Cardiovascular Toxicology · doi:10.1007/s12012-021-09675-w · Cited by 1
Writers, A. M. (2013) · Drugs & Therapy Perspectives · doi:10.1007/s40267-013-0077-1 · Cited by 1
Menopausal hypertension in the Nigerian female:Role of psycho-social stress
Eferakeya, A. E., & Imasuen, E. J. (1985) · Public Health · doi:10.1016/s0033-3506(85)80026-3 · Cited by 1
Evaluation of Left Ventricular Circumferential Contraction Functions in Obese Patients
Sürücü, H., Tatlı, E., Boz, H., & Meriç, M. (2010) · Echocardiography · doi:10.1111/j.1540-8175.2009.01036.x · Cited by 1
Evaluation and Treatment of Hypertension
Nguyen, Q. P. H., & Plodkowski, R. A. (2018) · Springer eBooks · doi:10.1007/978-3-319-95655-8_14 · Cited by 1
Antihypertensive Therapy in the Elderly
Carter, B. L. (1989) · Primary Care Clinics in Office Practice · doi:10.1016/s0095-4543(21)01097-6 · Cited by 1
Hemodynamics in patients with overweight and hypertension
Reisin, E., & Frohlich, E. D. (1989) · Developments in cardiovascular medicine · doi:10.1007/978-94-009-0941-0_11 · Cited by 1
Resolución de las patologías comórbidas bariátricas
MacDonald, K. G., & Pender, J. R. (2009) · Elsevier eBooks · doi:10.1016/b978-84-8086-428-2.50043-5
Weir, M. R. (2001) · Humana Press eBooks · doi:10.1007/978-1-59259-008-7_18
Role of socio-economic factor on menopausalhypertension
Eferakeya, A. E., & Imasuen, E. J. (1986) · Public Health · doi:10.1016/s0033-3506(86)80006-3
Gn, O., Левичева, Е. Н., Chernyavskiy, A. М., & Мироненко, С. П. (2006) · Human Physiology · doi:10.1134/s0362119706030030
“Numerous authors have already noted that the functions of body systems depend on BM. An increase in the activity of the sympathetic nervous system (SNS) is observed in subjects with excessive BM, causing an increase in the heart rate and cardiac output at rest, although with unchanged stroke volume [ 24 ].”
BackgroundClinical and Haemodynamic Effects of Ketanserin in Lean and Obese Hypertensive Patients
Licata, G., Scaglione, R., Parrinello, G., et al. (1990) · Journal of International Medical Research · doi:10.1177/030006059001800206
Is gravitation your friend or foe?
Haufe, S., & Jordan, J. (2013) · Hypertension Research · doi:10.1038/hr.2013.118
Messerli, F. H. (2005) · The American Journal of Cardiology · doi:10.1016/j.amjcard.2005.03.022
Self-citation
Metadata
- Authors
- Messerli, F. H.
- Corresponding author
- Messerli, Franz H.
- ORCID
- Messerli, Franz H.
- Venue
- Archives of Internal Medicine
- Publisher
- American Medical Association (AMA)
- Published
- 1981-01-01
- Volume
- 141
- Issue
- 1
- Pages
- 81
- ISSN
- 0003-9926 (ISSN-L)
- OpenAlex
- W4246843923
- Cited by
- 210
- References
- 0
- Citations by year
- 2025: 1 · 2024: 2 · 2023: 1 · 2022: 8 · 2021: 4 · 2020: 3 · 2019: 7 · 2018: 4 · 2017: 3 · 2016: 8 · 2015: 5 · 2014: 7 · 2013: 7 · 2012: 10
- FWCI
- 5.34
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Archives of Internal Medicine (publishedVersion)
- Topics
- Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Blood Pressure and Hypertension Studies
- Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Cardiovascular Function and Risk Factors
- Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Renin-Angiotensin System Studies
- Keywords
- obesity, essential hypertension, systemic hemodynamics, intravascular volume, plasma renin activity, cardiac output, systemic vascular resistance, sodium excretion, blood volume, left ventricular afterload, venous return
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
