Metformin and the Intestine
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With a plasma half-life of approximately 3 to 5 hours and subject to dosing schedule, it is unlikely that other than in gut enterocytes a significant cellular accumulation of metformin occurs when used for T2DM.
“However, with a plasma half-life of approximately 3 to 5 hours, and subject to the schedule of dosing, it is unlikely, that other than in gut enterocytes, a significant cellular accumulation of metformin occurs when the drug is used for the treatment of T2DM (34, 155, 156, 159, 160).”
From A Critical Review of the Evidence That Metformin Is a Putative Anti-Aging Drug That Enhances Healthspan and Extends Lifespan (2021) · cites this paper as [159]
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References 11 · Cited by 275
Research works printed but not linked: 1
- Bailey CJ, Campbell IW, Chan JCN et al (eds) (2007) Metformin: the gold standard. Wiley, Chichester
- Authors
- Bailey, C. J., Wilcock, C., & Scarpello, J. H. B.
- Affiliations
- Aston University (GB)
- University Hospitals of North Midlands NHS Trust (GB)
- Corresponding author
- Bailey, C. J.
- ORCID
- Bailey, C. J.
- Venue
- Diabetologia
- Published
- 2008-06-05
- Volume
- 51
- Issue
- 8
- Pages
- 1552-1553
- ISSN
- 0012-186X (ISSN-L)
- 1432-0428
- OpenAlex
- W1995265458
- Cited by
- 275
- References
- 11
- Citations by year
- 2026: 11 · 2025: 12 · 2024: 24 · 2023: 30 · 2022: 28 · 2021: 30 · 2020: 23 · 2019: 18 · 2018: 20 · 2017: 17 · 2016: 13 · 2015: 9 · 2014: 9 · 2013: 7 · 2012: 3
- FWCI
- 4.83
- Type
- journal article
- Language
- en
- Open access
- bronze
- doi.org
- Licence
- www.springer.com/tdm (tdm)
- Locations
- Diabetologia (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes Treatment and Management
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Metabolism, Diabetes, and Cancer
- Health Sciences › Medicine › Gastroenterology › Gastrointestinal motility and disorders
- Keywords
- metformin, intestinal mucosa, lactic acid production, anaerobic glucose metabolism, intestinal glucose absorption, type 2 diabetes, jejunal mucosa, hypoglycemic effect, glucose turnover, insulin-stimulated glucose uptake, hepatic glucose production, obesity, SGLT2 inhibition
- MeSH terms
- Animals, Diabetes Mellitus, Type 2, Glucose, Humans, Hypoglycemic Agents, Intestinal Mucosa, Intestines, Liver, Male, Metformin
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