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Pure adult-onset Spastic Paraplegia caused by a novel mutation in the KIAA0196 (SPG8) gene
Abstract
SPG8 is a rare autosomal dominant hereditary spastic paraplegia (AD-HSP), with only six SPG8 families described so far. Our purpose was to screen for KIAA0196 (SPG8) mutations in AD-HSP patients and to investigate their phenotype. Extensive family investigation was performed after positive KIAA0196 mutation analysis, which was part of an on-going mutation screening effort in AD-HSP patients. A novel pathogenic KIAA0196 mutation p.(Gly696Ala) was identified in two AD-HSP patients, who subsequently were shown to belong to a single large Dutch pedigree with more than 10 affected family members. The phenotype consisted of a pure HSP with ages at onset between 20 and 60 years, distally reduced vibration sense in the legs in all, and urinary urgency in seven out of 10 patients. Frequent features were exercise- or emotion-induced increase of spasticity and gait problems and chronic nonspecific lower back and joint pains. We have identified a fourth pathogenic KIAA0196 mutation in a Dutch HSP-family, the seventh family worldwide, with a less severe clinical course than described before.
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SPG8 is a rare autosomal dominant HSP characterized by onset of progressive lower limb spasticity and hyperreflexia in adults, resulting in impaired gait and difficulty in walking.
“SPG8 is a rare autosomal dominant HSP characterized by onset of progressive lower limb spasticity and hyperreflexia in adults, resulting in impaired gait and difficulty in walking.”
From Case report: A novel WASHC5 variant altering mRNA splicing causes spastic paraplegia in a patient (2023) · cites this paper as de Bot et al., 2013
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Research works printed but not linked: 12
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Case report: A novel WASHC5 variant altering mRNA splicing causes spastic paraplegia in a patient
Gao, S.-Y., Liu, Y.-X., Dong, Y., Fan, L.-L., Ding, Q., & Liu, L. (2023) · Frontiers in Genetics · doi:10.3389/fgene.2023.1205052 · Cited by 2
CCDC22 mutations that impair COMMD binding cause attenuated 3C/Ritscher-Schinzel syndrome
Singla, A., Rogers, C., Touma, M.-J., El-Najjar, Y., Colley, A., Boesch, D. J., Billadeau, D. D., Gecz, J., Chen, B., & Burstein, E. (2025) · BMC Medical Genomics · doi:10.1186/s12920-025-02168-7 · Cited by 1
Metadata
- Authors
- Bot, S. T., Vermeer, S., Buijsman, W., Heister, A., Voorendt, M., Verrips, A., Scheffer, H., Kremer, H. P. H., Warrenburg, B. P. C., & Kamsteeg, E.-J.
- Venue
- Journal of Neurology
- Published
- 2013-03-02
- Volume
- 260
- Issue
- 7
- Pages
- 1765-1769
- ISSN
- 0340-5354
- 1432-1459
- Cited by
- 39
- References
- 0
- Language
- en
- Licence
- www.springer.com/tdm (tdm)
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