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Neuroprotective and antiamnesic effects of Semax during experimental ischemic infarction of the cerebral cortex
Abstract
Semax had a pronounced neuroprotective and antiamnesic effect during focal photoinduced ischemia of the prefrontal cortex. Intranasal administration of Semax for 6 days decreased the volume of cortical infarction and improved retention and performance of conditioned passive avoidance response.
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Semax decreases ischemic brain damage in clinical and animal studies by inducing the expression of neurotrophins and their receptors.
“It has been shown that Semax decreases ischemic brain damage in clinical and animal studies by inducing the expression of neurotrophins and their receptors [45,46,47,48,49,50].”
From Protective Effects of PGC-1α Activators on Ischemic Stroke in a Rat Model of Photochemically Induced Thrombosis (2021) · cites this paper as [45]
Intranasal administration of Semax over a 7-day treatment course resulted in a 25% decrease of infarct volume in the prefrontal cortex and cognitive function recovery over the long term.
“Intranasal administration of Semax over the 7-day treatment course resulted in a 25% decrease of infarct volume in the prefrontal cortex and cognitive function recovery over the long term [45,81].”
From Protective Effects of PGC-1α Activators on Ischemic Stroke in a Rat Model of Photochemically Induced Thrombosis (2021) · cites this paper as [45]
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References 5 · Cited by 17
Research works printed but not linked: 11
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- G. A. Romanova, Dysregulation Changes in Integrative Function of the Brain during Focal Cerebral Ischemia. Dysregulation Pathology [in Russian], Moscow (2002), pp. 605–615.
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Metadata
- Authors
- Romanova, G. A., Silachev, D. N., Shakova, F. M., Kvashennikova, Y. N., Viktorov, I. V., Shram, S. I., & Myasoedov, N. F.
- Affiliations
- Research Institute of General Pathology and Pathophysiology, the Russian Academy of Medical Sciences (RU)
- Russian Academy of Sciences (RU)
- Corresponding author
- Romanova, G. A.
- Venue
- Bulletin of Experimental Biology and Medicine
- Published
- 2006-12
- Volume
- 142
- Issue
- 6
- Pages
- 663-666
- ISSN
- 0007-4888 (ISSN-L)
- 1573-8221
- OpenAlex
- W2106078501
- Cited by
- 17
- References
- 5
- Citations by year
- 2022: 1 · 2021: 2 · 2020: 4 · 2018: 2 · 2017: 2 · 2016: 3 · 2013: 1
- FWCI
- 0.44
- Type
- journal article
- Language
- en
- Open access
- closed
- Licence
- www.springer.com/tdm (tdm)
- Locations
- Bulletin of Experimental Biology and Medicine (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Life Sciences › Neuroscience › Neurology › Neuroprotection in Cerebral Ischemia
- Life Sciences › Neuroscience › Neurology › Neurological Disease Mechanisms and Treatments
- Health Sciences › Medicine › Clinical Neurology › Acute Ischemic Stroke Management
- Keywords
- Semax, neuroprotection, anti-amnesic effect, cerebral ischemia, focal cerebral infarction, prefrontal cortex, intranasal administration, memory impairment, conditioned passive avoidance
- MeSH terms
- Administration, Intranasal, Adrenocorticotropic Hormone, Amnesia, Animals, Cerebral Cortex, Ischemic Attack, Transient, Male, Peptide Fragments, Treatment Outcome, Neuroprotective Agents, Brain Infarction, Animals, Outbred Strains, Rats
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