Apolipoprotein B levels, APOB alleles, and risk of ischemic cardiovascular disease in the general population, a review
Abstract
Apolipoprotein B is a key component in lipid metabolism. Subendothelial retention of apolipoprotein B containing lipoproteins is a necessary initiating event in atherogenesis, and high plasma levels of apolipoprotein B is a risk factor for atherosclerosis, whereas low levels may provide protection. The present review examines, with focus on general population studies, apolipoprotein B levels as a predictor of ischemic cardiovascular disease, as well as the association of mutations and polymorphisms in APOB with plasma apolipoprotein B levels, and risk of ischemic cardiovascular disease. The studies can be summarized as follows: (1) apolipoprotein B predicts ischemic cardiovascular events in both genders, and is better than LDL cholesterol in this respect; (2) linkage disequilibrium structure in APOB is more complex than expected from HapMap data, because a minimal set of tag single nucleotide polymorphisms capturing the entire variation in APOB cannot be identified, and thus most polymorphisms must be evaluated separately in association studies; (3) APOB mutations and polymorphisms are associated with a range of apolipoprotein B and LDL cholesterol levels, although the magnitude of effect sizes of common polymorphisms are modest; (4) both mutations and polymorphisms are associated with LDL metabolism in vivo; (5) association of APOB mutations and polymorphisms with lipid and disease phenotype cannot be predicted in silico using evolutionary conservation or existing prediction programs; and finally, (6) except for the E4154K polymorphism that possibly predicts a reduction in risk of ischemic cerebrovascular disease and ischemic stroke, common APOB polymorphisms with modest effect sizes on lipid levels do not predict risk of ischemic heart disease, myocardial infarction, ischemic cerebrovascular disease, or ischemic stroke in the general population.
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In humans and mice, genetic APOB variants associated with increased levels of apoB are also associated with unusually high plasma concentrations of cholesterol and LDL, which contribute to hypercholesterolemia and heart disease in humans.
“In humans and mice, genetic APOB variants associated with increased levels of apoB are also associated with unusually high plasma concentrations of cholesterol and LDL, which in turn contribute to hypercholesterolemia and heart disease in humans (Benn, 2009; Hegele, 2009).”
From Population genomics reveal recent speciation and rapid evolutionary adaptation in polar bears (2014) · cites this paper as Benn, 2009
ApoB enables the transport of fat molecules in blood plasma and lymph and acts as a ligand for LDL receptors, facilitating movement of molecules such as cholesterol into cells.
“ApoB enables the transport of fat molecules in blood plasma and lymph and acts as a ligand for LDL receptors, facilitating the movement of molecules such as cholesterol into cells (Benn, 2009).”
From Population genomics reveal recent speciation and rapid evolutionary adaptation in polar bears (2014) · cites this paper as Benn, 2009
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References 92 · Cited by 119
- Authors
- Benn, M.
- Affiliations
- Copenhagen University Hospital (DK)
- Corresponding author
- Benn, Marianne
- ORCID
- Benn, Marianne
- Venue
- Atherosclerosis
- Publisher
- Elsevier BV
- Published
- 2009-09
- Volume
- 206
- Issue
- 1
- Pages
- 17-30
- ISSN
- 0021-9150 (ISSN-L)
- OpenAlex
- W1963682711
- Cited by
- 119
- References
- 92
- Citations by year
- 2026: 4 · 2025: 3 · 2024: 5 · 2023: 5 · 2022: 8 · 2021: 8 · 2020: 5 · 2019: 5 · 2018: 5 · 2017: 9 · 2016: 5 · 2015: 10 · 2014: 11 · 2013: 3 · 2012: 14
- FWCI
- 1.76
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Atherosclerosis (publishedVersion)
- PubMed (publishedVersion)
- Research at the University of Copenhagen (University of Copenhagen) (submittedVersion)
- Topics
- Health Sciences › Medicine › Surgery › Lipoproteins and Cardiovascular Health
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genetic Associations and Epidemiology
- Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Lipid metabolism and disorders
- Keywords
- apolipoprotein B, APOB mutations, ischemic cardiovascular disease, LDL cholesterol, linkage disequilibrium, tag single nucleotide polymorphisms, LDL metabolism, ischemic stroke, ischemic heart disease, cardiovascular risk prediction, general population
- MeSH terms
- Alleles, Apolipoproteins B, Cardiovascular Diseases, Female, Humans, Ischemia, Cholesterol, LDL, Male, Polymorphism, Genetic, Risk Factors, Linkage Disequilibrium
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