Cited by 119Lipoproteins and Cardiovascular HealthGenetic Associations and EpidemiologyLipid metabolism and disorders

Apolipoprotein B levels, APOB alleles, and risk of ischemic cardiovascular disease in the general population, a review

Benn, Marianne·2009·Atherosclerosis

Abstract

Apolipoprotein B is a key component in lipid metabolism. Subendothelial retention of apolipoprotein B containing lipoproteins is a necessary initiating event in atherogenesis, and high plasma levels of apolipoprotein B is a risk factor for atherosclerosis, whereas low levels may provide protection. The present review examines, with focus on general population studies, apolipoprotein B levels as a predictor of ischemic cardiovascular disease, as well as the association of mutations and polymorphisms in APOB with plasma apolipoprotein B levels, and risk of ischemic cardiovascular disease. The studies can be summarized as follows: (1) apolipoprotein B predicts ischemic cardiovascular events in both genders, and is better than LDL cholesterol in this respect; (2) linkage disequilibrium structure in APOB is more complex than expected from HapMap data, because a minimal set of tag single nucleotide polymorphisms capturing the entire variation in APOB cannot be identified, and thus most polymorphisms must be evaluated separately in association studies; (3) APOB mutations and polymorphisms are associated with a range of apolipoprotein B and LDL cholesterol levels, although the magnitude of effect sizes of common polymorphisms are modest; (4) both mutations and polymorphisms are associated with LDL metabolism in vivo; (5) association of APOB mutations and polymorphisms with lipid and disease phenotype cannot be predicted in silico using evolutionary conservation or existing prediction programs; and finally, (6) except for the E4154K polymorphism that possibly predicts a reduction in risk of ischemic cerebrovascular disease and ischemic stroke, common APOB polymorphisms with modest effect sizes on lipid levels do not predict risk of ischemic heart disease, myocardial infarction, ischemic cerebrovascular disease, or ischemic stroke in the general population.

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Crossref · 10.1016/j.atherosclerosis.2009.01.004

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  • In humans and mice, genetic APOB variants associated with increased levels of apoB are also associated with unusually high plasma concentrations of cholesterol and LDL, which contribute to hypercholesterolemia and heart disease in humans.

    “In humans and mice, genetic APOB variants associated with increased levels of apoB are also associated with unusually high plasma concentrations of cholesterol and LDL, which in turn contribute to hypercholesterolemia and heart disease in humans (Benn, 2009; Hegele, 2009).”
  • ApoB enables the transport of fat molecules in blood plasma and lymph and acts as a ligand for LDL receptors, facilitating movement of molecules such as cholesterol into cells.

    “ApoB enables the transport of fat molecules in blood plasma and lymph and acts as a ligand for LDL receptors, facilitating the movement of molecules such as cholesterol into cells (Benn, 2009).”

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References 92 · Cited by 119

Authors
Benn, M.
Affiliations
Copenhagen University Hospital (DK)
Corresponding author
Benn, Marianne
Venue
Atherosclerosis
Publisher
Elsevier BV
Published
2009-09
Volume
206
Issue
1
Pages
17-30
ISSN
0021-9150 (ISSN-L)
OpenAlex
W1963682711
Cited by
119
References
92
Citations by year
2026: 4 · 2025: 3 · 2024: 5 · 2023: 5 · 2022: 8 · 2021: 8 · 2020: 5 · 2019: 5 · 2018: 5 · 2017: 9 · 2016: 5 · 2015: 10 · 2014: 11 · 2013: 3 · 2012: 14
FWCI
1.76
Type
journal article
Language
en
Open access
closed
Locations
  • Atherosclerosis (publishedVersion)
  • PubMed (publishedVersion)
  • Research at the University of Copenhagen (University of Copenhagen) (submittedVersion)
Topics
  • Health Sciences › Medicine › Surgery › Lipoproteins and Cardiovascular Health
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genetic Associations and Epidemiology
  • Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Lipid metabolism and disorders
Keywords
apolipoprotein B, APOB mutations, ischemic cardiovascular disease, LDL cholesterol, linkage disequilibrium, tag single nucleotide polymorphisms, LDL metabolism, ischemic stroke, ischemic heart disease, cardiovascular risk prediction, general population
MeSH terms
Alleles, Apolipoproteins B, Cardiovascular Diseases, Female, Humans, Ischemia, Cholesterol, LDL, Male, Polymorphism, Genetic, Risk Factors, Linkage Disequilibrium

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