Brain expression level and activity of HDAC6 protein in neurodegenerative dementia
Abstract
Histone deacetylase 6 (HDAC6) is a multifunctional cytoplasmic protein that plays an especially critical role in the formation of aggresomes, where aggregates of excess protein are deposited. Previous immunohistochemical studies have shown that HDAC6 accumulates in Lewy bodies in Parkinson's disease and dementia with Lewy bodies (DLB) as well as in glial cytoplasmic inclusions in multiple system atrophy (MSA). However, it is uncertain whether the level and activity of HDAC6 are altered in the brains of patients with neurodegenerative dementia. In the present study, we demonstrated that the level of HDAC6 was not altered in the temporal cortex of patients with Alzheimer's disease and DLB in comparison with controls. In contrast, the level of HDAC6 was significantly increased in the temporal cortex of patients with frontotemporal lobar degeneration with TDP-43 inclusions (FTLD-TDP) and in the cerebellar white matter of patients with MSA. However, the level of acetylated α-tubulin, one of the substrates of HDAC6, was not altered in FTLD-TDP and MSA relative to controls. These findings suggest that the induced level of HDAC6 in the brain is insufficient for manifestation of its activity in FTLD-TDP and MSA.
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HDAC6 expression is increased in the cortex and hippocampus of AD patients.
“Expression of HDAC6 is increased in the cortex and hippocampus of AD patients [34–36].”
From Elevated SGK1 increases Tau phosphorylation and microtubule instability in Alzheimer’s patient-derived cortical neurons (2026) · cites this paper as [35]
Cites
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HDAC6 is a microtubule-associated deacetylase
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HDAC6 rescues neurodegeneration and provides an essential link between autophagy and the UPS.
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HDAC6 controls autophagosome maturation essential for ubiquitin‐selective quality‐control autophagy
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Saleem, K., Xiao, Z., Zhu, B., Ren, Y., Yan, Z., & Feng, J. (2026) · Molecular Psychiatry · doi:10.1038/s41380-025-03225-4 · Cited by 10 · see citing works
- Authors
- Odagiri, S., Tanji, K., Mori, F., Miki, Y., Kakita, A., Takahashi, H., & Wakabayashi, K.
- Affiliations
- Hirosaki University (JP)
- Brain Research Institute, Niigata University (JP)
- Corresponding author
- Tanji, Kunikazu
- Venue
- Biochemical and Biophysical Research Communications
- Publisher
- Elsevier BV
- Published
- 2013-01
- Volume
- 430
- Issue
- 1
- Pages
- 394-399
- ISSN
- 0006-291X (ISSN-L)
- OpenAlex
- W2072316845
- Cited by
- 43
- References
- 4
- Citations by year
- 2026: 3 · 2025: 3 · 2024: 4 · 2023: 3 · 2022: 5 · 2021: 3 · 2020: 5 · 2019: 2 · 2018: 2 · 2015: 5 · 2014: 5 · 2013: 3
- FWCI
- 2.79
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Biochemical and Biophysical Research Communications (publishedVersion)
- PubMed (publishedVersion)
- Funders
- JSPS KAKENHI (24700363, 23500425, 23500424, 24300131)
- President of Hirosaki University (2012-2209)
- Brain Research Institute, Niigata University
- Research Committee for Ataxic Disease, the Ministry of Health, Labor and Welfare, Japan (24-5)
- NCNP
- Topics
- Health Sciences › Medicine › Physiology › Alzheimer's disease research and treatments
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Histone Deacetylase Inhibitors Research
- Health Sciences › Medicine › Neurology › Amyotrophic Lateral Sclerosis Research
- Keywords
- HDAC6, neurodegenerative dementia, aggresomes, Lewy bodies, dementia with Lewy bodies, multiple system atrophy, frontotemporal lobar degeneration, TDP-43 inclusions, acetylated alpha-tubulin, temporal cortex, cerebellar white matter, protein aggregation
- MeSH terms
- Histone Deacetylase 6, Alzheimer Disease, Brain, DNA-Binding Proteins, Histone Deacetylases, Humans, Temporal Lobe, Multiple System Atrophy, Lewy Body Disease, Frontotemporal Lobar Degeneration
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