The DOI matches its Crossref record; 175 of the 175 references it cites were found in a registry or an index.
Central role of dysregulation of TGF-β/Smad in CKD progression and potential targets of its treatment
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Citations
Cites
2 retracted
Chung, A. C. K., Dong, Y., Yang, W., Zhong, X., Li, R., & Lan, H. Y. (2012) · Molecular Therapy · doi:10.1038/mt.2012.251 · Cited by 150
RetractedAi, J., Nie, J., He, J., Guo, Q., Li, M., Lei, Y., Liu, Y., Zhou, Z., Zhu, F., Liang, M., et al. (2014) · Journal of the American Society of Nephrology · doi:10.1681/asn.2014040363 · Cited by 78
RetractedMechanisms of TGF-β Signaling from Cell Membrane to the Nucleus
Shi, Y., & Massagué, J. (2003) · Cell · doi:10.1016/s0092-8674(03)00432-x · Cited by 5,857
Webster, A. C., Nagler, E. V., Morton, R. L., & Masson, P. (2017) · The Lancet · doi:10.1016/s0140-6736(16)32064-5 · Cited by 3,842
Mechanisms of fibrosis: therapeutic translation for fibrotic disease
Wynn, T. A., & Ramalingam, T. R. (2012) · Nature Medicine · doi:10.1038/nm.2807 · Cited by 3,541
TGF-β: the master regulator of fibrosis
Meng, X.-M., Nikolic-Paterson, D. J., & Lan, H. Y. (2016) · Nature Reviews Nephrology · doi:10.1038/nrneph.2016.48 · Cited by 3,528
Origin and function of myofibroblasts in kidney fibrosis
LeBleu, V. S., Taduri, G., O'Connell, J., Teng, Y., Cooke, V. G., Woda, C., Sugimoto, H., & Kalluri, R. (2013) · Nature Medicine · doi:10.1038/nm.3218 · Cited by 1,263
Partnership between DPC4 and SMAD proteins in TGF-β signalling pathways
Lagna, G., Hata, A., Hemmati-Brivanlou, A., & Massagué, J. (1996) · Nature · doi:10.1038/383832a0 · Cited by 905
Li, M. O., Sanjabi, S., & Flavell, R. A. (2006) · Immunity · doi:10.1016/j.immuni.2006.07.011 · Cited by 805
Li, M. O., Wan, Y. Y., & Flavell, R. A. (2007) · Immunity · doi:10.1016/j.immuni.2007.03.014 · Cited by 733
Cited by
TGF-β as A Master Regulator of Aging-Associated Tissue Fibrosis
Ren, L.-L., Miao, H., Wang, Y.-N., Liu, F., Li, P., & Zhao, Y.-Y. (2023) · Aging and disease · doi:10.14336/ad.2023.0222 · Cited by 139
Self-citationSong, G., Han, P., Sun, H., Shao, M., Yu, X., Wang, W., Wang, D., Yi, W., Ge, N., Li, S., et al. (2018) · Journal of International Medical Research · doi:10.1177/0300060518778711 · Cited by 34
Miao, Y., Wang, M., Cai, X., Zhu, Q., & Mao, L. (2022) · Bioengineered · doi:10.1080/21655979.2022.2048775 · Cited by 22
Metadata
- Authors
- Chen, L., Yang, T., Lu, D.-W., Zhao, H., Feng, Y.-L., Chen, H., Chen, D.-Q., Vaziri, N. D., & Zhao, Y.-Y.
- Venue
- Biomedicine & Pharmacotherapy
- Publisher
- Elsevier BV
- Published
- 2018-05
- Volume
- 101
- Pages
- 670-681
- ISSN
- 0753-3322 (ISSN-L)
- OpenAlex
- W2789927805
- Cited by
- 359
- References
- 175
- FWCI
- 11.93
- Type
- journal article
- Language
- en
- Open access
- closed
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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- CoChat. (2026). Citation check: Central role of dysregulation of TGF-β/Smad in CKD progression and potential targets of its treatment. https://app.cochat.ai/papers/10.1016/j.biopha.2018.02.090
- The paper
- Chen, L., Yang, T., Lu, D.-W., Zhao, H., Feng, Y.-L., Chen, H., Chen, D.-Q., Vaziri, N. D., & Zhao, Y.-Y. (2018). Central role of dysregulation of TGF-β/Smad in CKD progression and potential targets of its treatment. Biomedicine & Pharmacotherapy. https://doi.org/10.1016/j.biopha.2018.02.090
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
