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Cited by 200

The DOI matches its Crossref record; 21 of the 21 references it cites were found in a registry or an index.

The multifaceted role of astrocytes in regulating myelination

Open access
Kıray, Hülya, Lindsay, Susan L., Hosseinzadeh, Sara, Barnett, Susan C.·2016·Experimental Neurology

Abstract

Astrocytes are the major glial cell of the central nervous system (CNS), providing both metabolic and physical support to other neural cells. After injury, astrocytes become reactive and express a continuum of phenotypes which may be supportive or inhibitory to CNS repair. This review will focus on the ability of astrocytes to influence myelination in the context of specific secreted factors, cytokines and other neural cell targets within the CNS. In particular, we focus on how astrocytes provide energy and cholesterol to neurons, influence synaptogenesis, affect oligodendrocyte biology and instigate cross-talk between the many cellular components of the CNS.

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Registry

Crossref · 10.1016/j.expneurol.2016.03.009

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Metadata

Authors
Kıray, H., Lindsay, S. L., Hosseinzadeh, S., & Barnett, S. C.
Venue
Experimental Neurology
Publisher
Elsevier BV
Published
2016-09
Volume
283
Issue
Pt B
Pages
541-549
ISSN
0014-4886 (ISSN-L)
OpenAlex
W2301137789
Cited by
200
References
21
FWCI
7.80
Type
journal article
Language
en
Open access
hybrid

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CoChat. (2026). Citation check: The multifaceted role of astrocytes in regulating myelination. https://app.cochat.ai/papers/10.1016/j.expneurol.2016.03.009
The paper
Kıray, H., Lindsay, S. L., Hosseinzadeh, S., & Barnett, S. C. (2016). The multifaceted role of astrocytes in regulating myelination. Experimental Neurology. https://doi.org/10.1016/j.expneurol.2016.03.009

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.