Pyroptosis: An Inflammatory Link Between NAFLD and NASH With Potential Therapeutic Implications
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Lytic cell death programs including necrosis, necroptosis, and pyroptosis are important inflammatory drivers in NAFLD pathogenesis.
“There is an increasing appreciation that lytic cell death programs, including necrosis, necroptosis, and pyroptosis are important inflammatory drivers in the pathogenesis of NALFD (11).”
From Hepatic Stellate Cell-Immune Interactions in NASH (2022) · cites this paper as [11]
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References 20 · Cited by 101
- Authors
- Beier, J. I., & Banales, J. M.
- Affiliations
- University of Louisville (US)
- Corresponding author
- Beier, Juliane I.
- ORCID
- Beier, Juliane I.
- Venue
- Journal of Hepatology
- Publisher
- Elsevier BV
- Published
- 2018-04
- Volume
- 68
- Issue
- 4
- Pages
- 643-645
- ISSN
- 0168-8278 (ISSN-L)
- PMC
- PMC6185810
- OpenAlex
- W2794352179
- Cited by
- 101
- References
- 20
- Citations by year
- 2026: 6 · 2025: 13 · 2024: 19 · 2023: 16 · 2022: 12 · 2021: 17 · 2020: 11 · 2019: 4 · 2018: 3
- Type
- journal article
- Language
- en
- Open access
- green
- europepmc.org
- Locations
- Journal of Hepatology (publishedVersion)
- PubMed (publishedVersion)
- Europe PMC (PubMed Central) (acceptedVersion)
- PubMed Central (acceptedVersion)
- Funders
- National Institute on Alcohol Abuse and Alcoholism (P50AA024337)
- National Institute of General Medical Sciences (P20GM113226)
- National Institute of Diabetes and Digestive and Kidney Diseases (K01DK096042, R03DK107912)
- Topics
- Health Sciences › Medicine › Epidemiology › Liver Disease Diagnosis and Treatment
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Inflammasome and immune disorders
- Health Sciences › Medicine › Hepatology › Liver Diseases and Immunity
- Keywords
- pyroptosis, non-alcoholic fatty liver disease, nonalcoholic steatohepatitis, inflammation, metabolic syndrome, liver fibrosis, hepatocellular carcinoma, liver cirrhosis, therapeutic target
- MeSH terms
- Pyroptosis, Animals, Humans, Apoptosis Regulatory Proteins, Mice, Inflammasomes, Non-alcoholic Fatty Liver Disease
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