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Cited by 28Genomic variations and chromosomal abnormalitiesSchizophrenia research and treatmentGenetic Associations and Epidemiology

Schizophrenia: Genome, Interrupted

Open access
Cantor, Rita M., Geschwind, Daniel H.·2008·Neuron
DOI 10.1016/j.neuron.2008.04.007
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Abstract

Structural chromosomal variation is increasingly recognized as an important contributor to human diseases, particularly those of neurodevelopment, such as autism. A current paper makes a significant advance to schizophrenia genetics by establishing an association with rare copy number variants (CNV), which are over-represented in neurodevelopmental genes.

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Crossref · 10.1016/j.neuron.2008.04.007

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  • Direct genetic evidence links ASD and schizophrenia.

    “alongside direct genetic evidence that links ASD and schizophrenia (Cantor and Geschwind, 2008;Ellis et al, 2016;Zhou et al, 2016).”

    From The Role of the Immune System in Autism Spectrum Disorder (2017) · cites this paper as Cantor and Geschwind, 2008

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References 15 · Cited by 28

Authors
Cantor, R. M., & Geschwind, D. H.
Affiliations
University of California, Los Angeles (US)
Corresponding author
  • Cantor, Rita M.
  • Geschwind, Daniel H.
ORCID
Venue
Neuron
Publisher
Elsevier BV
Published
2008-04
Volume
58
Issue
2
Pages
165-167
ISSN
0896-6273 (ISSN-L)
DOI
10.1016/j.neuron.2008.04.007
OpenAlex
W1964148493
Cited by
28
References
15
Citations by year
2019: 2 · 2018: 1 · 2016: 1 · 2014: 1 · 2013: 1 · 2012: 3
FWCI
3.61
Type
journal article
Language
en
Open access
Licence
Locations
  • Neuron (publishedVersion)
  • PubMed (publishedVersion)
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genomic variations and chromosomal abnormalities
  • Health Sciences › Medicine › Psychiatry and Mental health › Schizophrenia research and treatment
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genetic Associations and Epidemiology
Keywords
schizophrenia, copy number variation, neurodevelopmental genes, schizophrenia genetics
MeSH terms
Humans, Schizophrenia, Genome

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