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188th ENMC International Workshop: Inclusion Body Myositis, 2–4 December 2011, Naarden, The Netherlands
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Construction and validation of a quality of life questionnaire for neuromuscular disease (INQoL)
Vincent, K. A., Carr, A. J., Walburn, J., Scott, D. L., & Rose, M. R. (2007) · Neurology · doi:10.1212/01.wnl.0000257819.47628.41 · Cited by 168
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Research works printed but not linked: 65
- Benveniste, 2010, International workshop on inclusion body myositis held at the Institute of Myology, Paris, on 29 May 2009, Neuromuscul Disord, 20, 414, 10.1016/j.nmd.2010.03.014 · doi:10.1016/j.nmd.2010.03.014
- Hilton-Jones, 2010, Inclusion body myositis: MRC centre for neuromuscular diseases, IBM workshop, London, 13 June 2008, Neuromuscul Disord, 20, 142, 10.1016/j.nmd.2009.11.003 · DOI
- Griggs, 1995, Inclusion body myositis and myopathies, Annal Neurol, 38, 705, 10.1002/ana.410380504 · DOI · Paper page
- Chahin, 2008, Correlation of muscle biopsy, clinical course, and outcome in PM and sporadic IBM, Neurology, 70, 418, 10.1212/01.wnl.0000277527.69388.fe · DOI · Paper page
- Meyer, 2012, Emerging functions of the VCP/p97 AAA-ATPase in the ubiquitin system, Nat Cell Biol, 14, 117, 10.1038/ncb2407 · DOI · Paper page
- Benjamin Larman H, Salajegheh M, Nazareno R, Lam T, Sauld J, Steen H, et al. Cytosolic 5′-nucleotidase 1A autoimmunity in sporadic inclusion body myositis. Ann Neurol 2013;73:408–18. · DOI · Paper page
- Pluk, 2013, Autoantibodies to cytosolic 5′-nucleotidase 1A in inclusion body myositis, Ann Neurol, 73, 397, 10.1002/ana.23822 · DOI · Paper page
- Schmidt, 2008, Interrelation of inflammation and APP in sIBM: IL-1 beta induces accumulation of beta-amyloid in skeletal muscle, Brain, 131, 1228, 10.1093/brain/awn053 · DOI
- Kitazawa, 2008, Inflammation induces tau pathology in inclusion body myositis model via glycogen synthase kinase-3beta, Ann Neurol, 64, 15, 10.1002/ana.21325 · DOI
- Muth, 2009, Proinflammatory cell stress in sporadic inclusion body myositis muscle: overexpression of aB-crystallin is associated with amyloid precursor protein and accumulation of bamyloid., J Neurol Neurosurg Psychiatry, 80, 1344, 10.1136/jnnp.2009.174276 · doi:10.1136/jnnp.2009.174276
- Keller, 2011, TNF-alpha induces macroautophagy and regulates MHC class II expression in human skeletal muscle cells, J Biol Chem, 286, 3970, 10.1074/jbc.M110.159392 · DOI
- Schmidt, 2010, Of amyloid and inflammation: causes of chronic muscle disease, e-Neuroforum, 1, 81, 10.1007/s13295-010-0010-1 · DOI
- Needham, 2008, Prevalence of sporadic inclusion body myositis and factors contributing to delayed diagnosis, J Clin Neurosci, 15, 1350, 10.1016/j.jocn.2008.01.011 · DOI
- Serdaroglu, 2007, Prevalence of sporadic inclusion body myositis (s-IBM) in Turkey: a muscle biopsy-based survey, Neuromuscul Disord, 17, 849, 10.1016/j.nmd.2007.06.293 · doi:10.1016/j.nmd.2007.06.293
- Scott, 2011, Recombination mapping of the susceptibility region for sporadic inclusion body myositis within the major histocompatibility complex, J Neuroimmunol, 235, 77, 10.1016/j.jneuroim.2011.02.011 · DOI
- Mastaglia, 2009, Sporadic inclusion body myositis: HLA-DRB1 allele interactions influence disease risk and clinical phenotype, Neuromuscul Disord, 19, 763, 10.1016/j.nmd.2009.07.015 · doi:10.1016/j.nmd.2009.07.015
- Sivakumar, 1995, Exons 16 and 17 of the amyloid precursor protein gene in familial inclusion body myopathy, Ann Neurol, 38, 267, 10.1002/ana.410380222 · DOI
- Orth, 2000, Sporadic inclusion body myositis not linked to prion protein codon 129 methionine homozygosity, Neurology, 55, 1235, 10.1212/WNL.55.8.1235 · doi:10.1212/wnl.55.8.1235
- Salajegheh, 2009, Sarcoplasmic redistribution of nuclear TDP-43 in inclusion body myositis, Muscle & Nerve, 40, 19, 10.1002/mus.21386 · DOI
- Gossrau, 2004, Apolipoprotein E and alpha-1-antichymotrypsin polymorphisms in sporadic inclusion body myositis, Eur Neurol, 51, 215, 10.1159/000078488 · DOI
- Needham, 2008, Apolipoprotein epsilon alleles in sporadic inclusion body myositis: a reappraisal, Neuromuscul Disord, 18, 150, 10.1016/j.nmd.2007.09.005 · DOI
- Askanas, 1996, Apolipoprotein E alleles in sporadic inclusion-body myositis and hereditary inclusion-body myopathy, Ann Neurol, 40, 264, 10.1002/ana.410400223 · DOI
- Harrington, 1995, Apolipoprotein E type epsilon 4 allele frequency is not increased in patients with sporadic inclusion-body myositis, Neurosci Lett, 183, 35, 10.1016/0304-3940(94)11108-U · DOI
- Love, 1996, Apolipoprotein E allele frequencies in sporadic inclusion body myositis, Muscle & Nerve, 19, 1605, 10.1002/(SICI)1097-4598(199612)19:12<1605::AID-MUS11>3.0.CO;2-Q · DOI
- Vasconcelos, 2002, GNE mutations in an American family with quadriceps-sparing IBM and lack of mutations in s-IBM, Neurology, 59, 1776, 10.1212/01.WNL.0000039780.13681.AD · DOI
- Machado, 2012, Valosin containing protein (VCP) and myofibrillar myopathies (MFM) genes’ mutations are not associated with sporadic inclusion body myositis (sIBM)., JNNP, 83, 3e1
- Ranque-Francois, 2005, Familial inflammatory inclusion body myositis, AnnRheumDis., 64, 634
- Amato, 1998, Inclusion body myositis in twins, Neurology, 51, 598, 10.1212/WNL.51.2.598 · DOI
- Naumann, 1999, Inclusion body myositis in twins; correspondence, Neurology, 53, 656, 10.1212/WNL.53.3.656-e · DOI
- Sivakumar, 1997, An inflammatory, familial, inclusion body myositis with autoimmune features and a phenotype identical to sporadic inclusion body myositis. Studies in three families, Brain, 120, 653, 10.1093/brain/120.4.653 · DOI
- Griggs, 1995, Inclusion body myositis and myopathies, Ann Neurol, 38, 705, 10.1002/ana.410380504 · DOI · Paper page
- Lotz, 1989, Inclusion body myositis. Observations in 40 patients, Brain, 112, 727, 10.1093/brain/112.3.727 · DOI · Paper page
- Lindberg, 1994, Inclusion body myositis: clinical, morphological, physiological and laboratory findings in 18 cases, Acta Neurol Scand, 89, 123, 10.1111/j.1600-0404.1994.tb01647.x · doi:10.1111/j.1600-0404.1994.tb01647.x
- Amato, 1996, Inclusion body myositis: clinical and pathological boundaries, Ann Neurol, 40, 581, 10.1002/ana.410400407 · DOI
- Badrising, 2005, Inclusion body myositis. Clinical features and clinical course of the disease in 64 patients, J Neurol, 252, 1448, 10.1007/s00415-005-0884-y · DOI
- Felice, 2001, Inclusion body myositis in Connecticut: observations in 35 patients during an 8-year period, Medicine (Baltimore), 80, 320, 10.1097/00005792-200109000-00006 · DOI
- Cox, 2011, A 12-year follow-up in sporadic inclusion body myositis: an end stage with major disabilities, Brain, 134, 3167, 10.1093/brain/awr217 · DOI
- Rose, 2001, A prospective natural history study of inclusion body myositis: implications for clinical trials, Neurology, 57, 548, 10.1212/WNL.57.3.548 · DOI
- Dalakas, 2009, Effect of Alemtuzumab (CAMPATH 1-H) in patients with inclusion-body myositis, Brain, 132, 1536, 10.1093/brain/awp104 · doi:10.1093/brain/awp104
- Crosby, 1994, Hand strength: normative values, J Hand Surg Am, 19, 665, 10.1016/0363-5023(94)90280-1 · DOI · Paper page
- Tawil, 2002, Inclusion body myositis, Curr Opin Rheumatol, 14, 653, 10.1097/00002281-200211000-00004 · DOI
- Hayter, 2012, Updated assessment of the prevalence, spectrum and case definition of autoimmune disease, Autoimmun Rev, 10.1016/j.autrev.2012.02.001 · DOI · Paper page
- Needham, 2007, Inclusion body myositis: current pathogenetic concepts and diagnostic and therapeutic approaches, Lancet Neurol, 6, 620, 10.1016/S1474-4422(07)70171-0 · DOI · Paper page
- Benveniste, 2011, Long-term observational study of sporadic inclusion body myositis, Brain, 134, 3176, 10.1093/brain/awr213 · doi:10.1093/brain/awr213
- Peng, 2000, Disease progression in sporadic inclusion body myositis: observations in 78 patients, Neurology, 55, 296, 10.1212/WNL.55.2.296 · doi:10.1212/wnl.55.2.296
- Rose MR, Griggs R, Dalakas M. Immunotherapy for inclusion body myositis (Protocol for a Cochrane Review). Issue 4 ed: Update software; 1999. · doi:10.1002/14651858.cd001555
- Barohn, 1995, Inclusion body myositis: explanation for poor response to immunosuppressive therapy, Neurology, 45, 1302, 10.1212/WNL.45.7.1302 · DOI
- Dalakas, 2001, A controlled study of intravenous immunoglobulin combined with prednisolone in the treatment of IBM, Neurology, 56, 323, 10.1212/WNL.56.3.323 · DOI
- Muscle Study Group. Randomized pilot trial of INF1a (Avonex) in patients with inclusion body myositis. Neurology 2001;57:1566–70. · DOI
- Skalsky, 2008, The relationship between regional body composition and quantitative strength in facioscapulohumeral muscular dystrophy (FSHD), Neuromuscul Disord, 18, 873, 10.1016/j.nmd.2008.07.005 · DOI
- Skalsky, 2009, Assessment of regional body composition with dual-energy X-ray absorptiometry in Duchenne muscular dystrophy: correlation of regional lean mass and quantitative strength, Muscle & Nerve, 39, 647, 10.1002/mus.21212 · doi:10.1002/mus.21212
- Vanhoutte, 2011, Modifying the Medical Research Council grading system through Rasch analyses, Brain
- Lowes, 2012, Knee extensor strength exhibits potential to predict function in sporadic inclusion-body myositis, Muscle & Nerve, 45, 163, 10.1002/mus.22321 · doi:10.1002/mus.22321
- Allenbach, 2012, Quadriceps strength is a sensitive marker of disease progression in sporadic inclusion body myositis, Neuromuscul Disord, 22, 980, 10.1016/j.nmd.2012.05.004 · doi:10.1016/j.nmd.2012.05.004
- Jackson, 2008, Inclusion body myositis functional rating scale: a reliable and valid measure of disease severity, Muscle & Nerve, 37, 473, 10.1002/mus.20958 · DOI
- Muscle Study Group. Randomized pilot trial of high-dose betaINF-1a in patients with inclusion body myositis. Neurology 2004;63:718–20. · DOI
- Benveniste, 2011, Long-term observational study of sporadic inclusion body myositis, Brain, 134, 3176, 10.1093/brain/awr213 · doi:10.1093/brain/awr213
- Sadjadi, 2010, What determines quality of life in inclusion body myositis?, J Neurol Neurosurg Psychiatry, 81, 1164, 10.1136/jnnp.2009.183863 · DOI · Paper page
- Badrising, 2000, Epidemiology of inclusion body myositis in the Netherlands: a nationwide study, Neurology, 55, 1385, 10.1212/WNL.55.9.1385 · DOI
- Eccles, 1996, North of England evidence based guidelines development project: methods of guideline development, BMJ, 312, 760, 10.1136/bmj.312.7033.760 · DOI
- Eccles, 1996, Developing valid guidelines: methodological and procedural issues from the North of England evidence based guideline development project, Qual Health Care, 5, 44, 10.1136/qshc.5.1.44 · DOI
- Dalakas, 1997, Treatment of inclusion-body myositis with IVIg: a double-blind, placebo-controlled study, Neurology., 48, 712, 10.1212/WNL.48.3.712 · DOI · Paper page
- Walter, 2000, High dose immunglobulin therapy in sporadic inclusion body myositis: a double blind, placebo-controlled study, J Neurol., 247, 22, 10.1007/s004150050005 · DOI
- Rutkove, 2002, A pilot randomized trial of oxandrolone in inclusion body myositis, Neurology., 58, 1081, 10.1212/WNL.58.7.1081 · DOI
- Badrising, 2002, Comparison of weakness progression in inclusion body myositis during treatment with methotrexate or placebo, Annal Neurol., 51, 369, 10.1002/ana.10121 · DOI
Cited by
Rose, M. R., Norton, S., Vari, C., Edwards, V., McCracken, L., Graham, C. D., Radunovic, A., & Chalder, T. (2018) · BMJ Open · doi:10.1136/bmjopen-2018-022083 · Cited by 15
Self-citationRose, M., Graham, C. D., O'Connell, N., Vari, C., Edwards, V., Taylor, E., McCracken, L. M., Radunovic, A., Rakowicz, W., Norton, S., et al. (2023) · Psychological Medicine · doi:10.1017/s0033291722000083 · Cited by 28
Self-citation
Metadata
- Authors
- Rose, M. R.
- Venue
- Neuromuscular Disorders
- Publisher
- Elsevier BV
- Published
- 2013-12
- Volume
- 23
- Issue
- 12
- Pages
- 1044-1055
- ISSN
- 0960-8966 (ISSN-L)
- OpenAlex
- W1995588340
- Cited by
- 388
- References
- 1
- FWCI
- 13.84
- Type
- journal article
- Language
- en
- Open access
- bronze
- Licence
- www.elsevier.com/tdm/userlicense/1.0/ (tdm)
- www.elsevier.com/legal/tdmrep-license (tdm)
- Funders
- Muskelsvindfonden (Denmark)
- Association Française contre les Myopathies (France)
- Deutsche Gesellschaft für Muskelkranke (Germany)
- Telethon Foundation (Italy)
- Drustvo Distrofikov Slovenije (Slovenia)
- , Schweizerische Stiftung für die Erforschung der Muskelkrankheiten (Switzerland)
- Prinses Beatrix Fonds (The Netherlands)
- Vereniging Spierziekten Nederland (The Netherlands)
- Muscular Dystrophy Campaign (UK)
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