Cited by 401Retinal Development and DisordersRetinal Diseases and TreatmentsGlaucoma and retinal disorders

Cellular responses following retinal injuries and therapeutic approaches for neurodegenerative diseases

Cuenca, Nicolás, Fernández-Sánchez, Laura, Campello, Laura, Maneu, Victoria, De la Villa, Pedro, Lax, Pedro, Pinilla, Isabel·2014·Progress in Retinal and Eye Research

Abstract

Retinal neurodegenerative diseases like age-related macular degeneration, glaucoma, diabetic retinopathy and retinitis pigmentosa each have a different etiology and pathogenesis. However, at the cellular and molecular level, the response to retinal injury is similar in all of them, and results in morphological and functional impairment of retinal cells. This retinal degeneration may be triggered by gene defects, increased intraocular pressure, high levels of blood glucose, other types of stress or aging, but they all frequently induce a set of cell signals that lead to well-established and similar morphological and functional changes, including controlled cell death and retinal remodeling. Interestingly, an inflammatory response, oxidative stress and activation of apoptotic pathways are common features in all these diseases. Furthermore, it is important to note the relevant role of glial cells, including astrocytes, Müller cells and microglia, because their response to injury is decisive for maintaining the health of the retina or its degeneration. Several therapeutic approaches have been developed to preserve retinal function or restore eyesight in pathological conditions. In this context, neuroprotective compounds, gene therapy, cell transplantation or artificial devices should be applied at the appropriate stage of retinal degeneration to obtain successful results. This review provides an overview of the common and distinctive features of retinal neurodegenerative diseases, including the molecular, anatomical and functional changes caused by the cellular response to damage, in order to establish appropriate treatments for these pathologies.

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Crossref · 10.1016/j.preteyeres.2014.07.001

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  • The presence of reactive astrocytes, microglial activation, and release of inflammatory mediators such as cytokines, reactive oxygen species, nitric oxide, and TNF-α could cause a state of chronic inflammation that may exert neurotoxic effects.

    “The presence of reactive astrocytes, the microglial activation, and the release of inflammatory mediators such as cytokines, reactive oxygen species (ROS), nitric oxide (NO), and Tumor Necrosis Factor-α (TNF-α) could cause a state of chronic inflammation that may exert neurotoxic effects (Cuenca et al., 2014).”

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References 727 · Cited by 401

Authors
Cuenca, N., Fernández-Sánchez, L., Campello, L., Maneu, V., De la Villa, P., Lax, P., & Pinilla, I.
Affiliations
  • University of Alicante (ES)
  • Hospital Clínico Universitario Lozano Blesa (ES)
Corresponding author
Cuenca, Nicolás
Venue
Progress in Retinal and Eye Research
Publisher
Elsevier BV
Published
2014-11
Volume
43
Pages
17-75
ISSN
1350-9462 (ISSN-L)
OpenAlex
W2005433264
Cited by
401
References
727
Citations by year
2026: 16 · 2025: 21 · 2024: 33 · 2023: 42 · 2022: 40 · 2021: 38 · 2020: 41 · 2019: 29 · 2018: 30 · 2017: 33 · 2016: 36 · 2015: 37 · 2014: 5
FWCI
3.48
Type
journal article
Language
en
Open access
closed
Locations
  • Progress in Retinal and Eye Research (publishedVersion)
  • PubMed (publishedVersion)
  • RUA, Repositorio Institucional de la Universidad de Alicante (Universidad de Alicante) (submittedVersion)
Funders
  • Spanish Ministry of Economy and Competitiveness-FEDER (BFU2012-36845)
  • Instituto de Salud Carlos III, (RETICS RD07/0062/0008-0012, RETICS RD12/0034/0006-0010, PS0901854, PI13/01124)
  • ONCE
  • FUNDALUCE
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Retinal Development and Disorders
  • Health Sciences › Medicine › Ophthalmology › Retinal Diseases and Treatments
  • Health Sciences › Medicine › Ophthalmology › Glaucoma and retinal disorders
Keywords
retinal damage, age-related macular degeneration, glaucoma, diabetic retinopathy, retinitis pigmentosa, retinal remodeling, apoptosis, oxidative stress, neuroinflammation, Müller cells, astrocytes, microglia, glial cells, neuroprotection, gene therapy, cell transplantation, retinal prosthesis
MeSH terms
Anti-Inflammatory Agents, Antioxidants, Clinical Trials as Topic, Humans, Retinal Degeneration, Apoptosis, Oxidative Stress, Neuroprotective Agents, Neurodegenerative Diseases, Stem Cell Transplantation, Retinal Neurons

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