The DOI matches its Crossref record; 100 of the 100 references it cites were found in a registry or an index.
Bronchopulmonary dysplasia: A review of pathogenesis and pathophysiology
Open accessAbstract
Bronchopulmonary dysplasia (BPD) is a chronic lung disease of primarily premature infants that results from an imbalance between lung injury and repair in the developing lung. BPD is the most common respiratory morbidity in preterm infants, which affects nearly 10, 000 neonates each year in the United States. Over the last two decades, the incidence of BPD has largely been unchanged; however, the pathophysiology has changed with the substantial improvement in the respiratory management of extremely low birth weight (ELBW) infants. Here we have attempted to comprehensively review and summarize the current literature on the pathogenesis and pathophysiology of BPD. Our goal is to provide insight to help further progress in preventing and managing severe BPD in the ELBW infants.
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Cites
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Neonatal Outcomes of Extremely Preterm Infants From the NICHD Neonatal Research Network
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Ventilator-Induced Lung Injury
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Defining dysbiosis and its influence on host immunity and disease
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Chorioamnionitis and Early Lung Inflammation in Infants in Whom Bronchopulmonary Dysplasia Develops
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Cited by
Asiaticoside attenuates hyperoxia-induced lung injury in vitro and in vivo.
(2019) · PubMed Central · doi:10.22038/ijbms.2019.35913.8556 · Cited by 12
Metadata
- Authors
- Kalikkot Thekkeveedu, R., Guaman, M. C., & Shivanna, B.
- Venue
- Respiratory Medicine
- Publisher
- Elsevier BV
- Published
- 2017-11
- Volume
- 132
- Pages
- 170-177
- ISSN
- 0954-6111 (ISSN-L)
- OpenAlex
- W2765990633
- Cited by
- 409
- References
- 100
- FWCI
- 18.83
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
