The Effect of Insulin on Oxidative Phosphorylation in Normal and Diabetic Mitochondria
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In liver and muscle mitochondria isolated from rodents or cats with insulin-deficient diabetes induced by pancreatectomy, alloxan, or streptozotocin, respiration on complex I or complex II substrates is decreased.
“Early studies revealed decreased respiration on complex I or complex II substrates in liver and muscle mitochondria isolated from rodents or cats made diabetic by pancreatectomy, alloxan, or streptozotocin (38, 119, 127, 322).”
From Mitochondrial Dysfunction in Diabetes: From Molecular Mechanisms to Functional Significance and Therapeutic Opportunities (2010) · cites this paper as [127]
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References 12 · Cited by 90
- Authors
- Hall, J. C., Sordahl, L. A., & Stefko, P. L.
- Affiliations
- Rutgers, The State University of New Jersey (US)
- Venue
- Journal of Biological Chemistry
- Publisher
- Elsevier BV
- Published
- 1960-05
- Volume
- 235
- Issue
- 5
- Pages
- 1536-1539
- ISSN
- 0021-9258 (ISSN-L)
- OpenAlex
- W2183021025
- Cited by
- 90
- References
- 12
- Citations by year
- 2025: 1 · 2021: 1 · 2020: 1 · 2019: 1 · 2017: 2 · 2016: 1 · 2015: 2 · 2013: 2
- FWCI
- 2.28
- Type
- journal article
- Language
- en
- Open access
- hybrid
- doi.org
- Licence
- Locations
- Journal of Biological Chemistry (publishedVersion, cc-by)
- PubMed (publishedVersion)
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Mitochondrial Function and Pathology
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Metabolism, Diabetes, and Cancer
- Health Sciences › Medicine › Epidemiology › Autophagy in Disease and Therapy
- Keywords
- insulin, oxidative phosphorylation, mitochondria, diabetes mellitus, mitochondrial respiration, insulin action
- MeSH terms
- Diabetes Mellitus, Humans, Insulin, Liver, Mitochondria, Oxidative Phosphorylation
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