The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.
Retinopathy of prematurity
Open accessAbstract
The immature retinas of preterm neonates are susceptible to insults that disrupt neurovascular growth, leading to retinopathy of prematurity. Suppression of growth factors due to hyperoxia and loss of the maternal–fetal interaction result in an arrest of retinal vascularisation (phase 1). Subsequently, the increasingly metabolically active, yet poorly vascularised, retina becomes hypoxic, stimulating growth factor-induced vasoproliferation (phase 2), which can cause retinal detachment. In very premature infants, controlled oxygen administration reduces but does not eliminate retinopathy of prematurity. Identification and control of factors that contribute to development of retinopathy of prematurity is essential to prevent progression to severe sight-threatening disease and to limit comorbidities with which the disease shares modifiable risk factors. Strategies to prevent retinopathy of prematurity will depend on optimisation of oxygen saturation, nutrition, and normalisation of concentrations of essential factors such as insulin-like growth factor 1 and ω-3 polyunsaturated fatty acids, as well as curbing of the effects of infection and inflammation to promote normal growth and limit suppression of neurovascular development.
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Cites
Epidemiology and causes of preterm birth
Goldenberg, R. L., Culhane, J. F., Iams, J. D., & Romero, R. (2008) · The Lancet · doi:10.1016/s0140-6736(08)60074-4 · Cited by 7,801
the GAPPS Review Group, Lawn, J. E., Gravett, M. G., Nunes, T. M., Rubens, C. E., & Stanton, C. (2010) · BMC Pregnancy and Childbirth · doi:10.1186/1471-2393-10-s1-s1 · Cited by 784
Blood gases, pH, and lactate in appropriate- and small-for-gestational-age fetuses
Nicolaides, K. H., Economides, D. L., & Soothill, P. W. (1989) · American Journal of Obstetrics and Gynecology · doi:10.1016/0002-9378(89)90770-9 · Cited by 390
Cited by
Digital technology, tele-medicine and artificial intelligence in ophthalmology: A global perspective
Li, J.-P. O., Liu, H., Ting, D. S. J., Jeon, S., Chan, R. V. P., Kim, J. E., Sim, D. A., Thomas, P. B. M., Lin, H., Chen, Y., et al. (2021) · Progress in Retinal and Eye Research · doi:10.1016/j.preteyeres.2020.100900 · Cited by 626
Tin, W., Milligan, D. W. A., Pennefather, P., & Hey, E. (2001) · Archives of Disease in Childhood - Fetal and Neonatal Edition · doi:10.1136/fn.84.2.f106 · Cited by 432
Neutrophil extracellular traps target senescent vasculature for tissue remodeling in retinopathy
Binet, F., Cagnone, G., Crespo-Garcia, S., Hata, M., Neault, M., Dejda, A., Wilson, A. M., Buscarlet, M., Mawambo, G. T., Howard, J. P., et al. (2020) · Science · doi:10.1126/science.aay5356 · Cited by 283
Senescence-associated secretory phenotype contributes to pathological angiogenesis in retinopathy
Oubaha, M., Miloudi, K., Dejda, A., Guber, V., Mawambo, G., Germain, M.-A., Bourdel, G., Popovic, N., Rezende, F. A., Kaufman, R. J., et al. (2016) · Science Translational Medicine · doi:10.1126/scitranslmed.aaf9440 · Cited by 259
Preterm birth and neurodevelopmental outcome: a review
Arpino, C., Compagnone, E., Montanaro, M. L., Cacciatore, D., De Luca, A., Cerulli, A., Di Girolamo, S., & Curatolo, P. (2010) · Child's Nervous System · doi:10.1007/s00381-010-1125-y · Cited by 195
Metadata
- Authors
- Hellström, A., Smith, L. E., & Dammann, O.
- Venue
- The Lancet
- Publisher
- Elsevier BV
- Published
- 2013-10
- Volume
- 382
- Issue
- 9902
- Pages
- 1445-1457
- ISSN
- 0099-5355 (ISSN-L)
- 0140-6736
- OpenAlex
- W1998726711
- Cited by
- 1,304
- References
- 3
- FWCI
- 45.87
- Type
- journal article
- Language
- en
- Open access
- green
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
