The DOI matches its Crossref record; 34 of the 34 references it cites were found in a registry or an index.
FGFR activation in skeletal disorders: Too much of a good thing
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Citations
Cites
Signal transduction by receptors with tyrosine kinase activity
Ullrich, A., & Schlessinger, J. (1990) · Cell · doi:10.1016/0092-8674(90)90801-k · Cited by 5,435
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The Fgf Family of Growth Factors and Oncogenes
(1992) · Advances in Cancer Research · doi:10.1016/s0065-230x(08)60305-x · Cited by 1,145
Fibroblast Growth Factor Receptor 3 Is a Negative Regulator of Bone Growth
Deng, C., Wynshaw-Boris, A., Zhou, F., Kuo, A., & Leder, P. (1996) · Cell · doi:10.1016/s0092-8674(00)81069-7 · Cited by 1,070
Bargmann, C. I., Hung, M.-C., & Weinberg, R. A. (1986) · Cell · doi:10.1016/0092-8674(86)90779-8 · Cited by 1,006
Mutations in the gene encoding fibroblast growth factor receptor-3 in achondroplasia
Rousseau, F., Bonaventure, J., Legeai-Mallet, L., Pelet, A., Rozet, J.-M., Maroteaux, P., Merrer, M. L., & Munnich, A. (1994) · Nature · doi:10.1038/371252a0 · Cited by 928
Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3
Colvin, J. S., Bohne, B. A., Harding, G. W., McEwen, D. G., & Ornitz, D. M. (1996) · Nature Genetics · doi:10.1038/ng0496-390 · Cited by 873
Activation of RET as a Dominant Transforming Gene by Germline Mutations of MEN2A and MEN2B
Santoro, M., Carlomagno, F., Romano, A., Bottaro, D. P., Dathan, N. A., Grieco, M., Fusco, A., Vecchio, G., Matoskova, B., Kraus, M. H., et al. (1995) · Science · doi:10.1126/science.7824936 · Cited by 841
Spivak-Kroizman, T., Lemmon, M. A., Dikic, I., Ladbury, J. E., Pinchasi, D., Huang, J., Jaye, M., Crumley, G., Schlessinger, J., & Lax, I. (1994) · Cell · doi:10.1016/0092-8674(94)90032-9 · Cited by 658
Fibroblast growth factor receptor tyrosine kinases: molecular analysis and signal transduction
Jaye, M., Schlessinger, J., & Dionne, C. A. (1992) · Biochimica et Biophysica Acta (BBA) - Molecular Cell Research · doi:10.1016/0167-4889(92)90136-y · Cited by 617
Cited by
The FGF family: biology, pathophysiology and therapy
Beenken, A., & Mohammadi, M. (2009) · Nature Reviews Drug Discovery · doi:10.1038/nrd2792 · Cited by 1,945
Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23
White, K. E., Evans, W. E., O'Riordan, J. L. H., Speer, M. C., Econs, M. J., Lorenz-Depiereux, B., Grabowski, M., Meitinger, T., & Strom, T. M. (2000) · Nature Genetics · doi:10.1038/81664 · Cited by 1,528
Johnson, D., & Wilkie, A. O. M. (2011) · European Journal of Human Genetics · doi:10.1038/ejhg.2010.235 · Cited by 499
The Promise of Comparative Genomics in Mammals
O'Brien, S. J., Menotti-Raymond, M., Murphy, W. J., Nash, W. G., Wienberg, J., Stanyon, R., Copeland, N. G., Jenkins, N. A., Womack, J. E., & Marshall Graves, J. A. (1999) · Science · doi:10.1126/science.286.5439.458 · Cited by 423
Naski, M. C., Colvin, J. S., Coffin, J. D., & Ornitz, D. M. (1998) · Development · doi:10.1242/dev.125.24.4977 · Cited by 420
Metadata
- Authors
- Webster, M.
- Venue
- Trends in Genetics
- Publisher
- Elsevier BV
- Published
- 1997-05
- Volume
- 13
- Issue
- 5
- Pages
- 178-182
- ISSN
- 0168-9525 (ISSN-L)
- OpenAlex
- W2055229479
- Cited by
- 317
- References
- 34
- FWCI
- 9.56
- Type
- journal article
- Language
- en
- Open access
- closed
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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- CoChat. (2026). Citation check: FGFR activation in skeletal disorders: Too much of a good thing. https://app.cochat.ai/papers/10.1016/s0168-9525(97)01131-1
- The paper
- Webster, M. (1997). FGFR activation in skeletal disorders: Too much of a good thing. Trends in Genetics. https://doi.org/10.1016/s0168-9525(97)01131-1
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.