The DOI matches its Crossref record; 74 of the 74 references it cites were found in a registry or an index.
Epidemiology, clinical features, and immunology of the “intrinsic” (non‐IgE‐mediated) type of atopic dermatitis (constitutional dermatitis)
Open accessAbstract
Atopic dermatitis (AD) is a common, chronic relapsing, inflammatory skin disease, characterized by typically distributed eczematous skin lesions, dry skin, intense pruritus, and a wide variety of pathophysiologic aspects (1-4).The current knowledge of the genetic background and the immunopathogenesis of AD, as well as the role of environmental and other major provocation factors in this disorder, was recently reviewed in Allergy (3, 4).Thus, AD is a genetically determined, IgE-mediated, delayed-type hypersensitivity reaction of the skin (5, 6) that can be triggered by multiple factors, varying in their intensity in each individual patient and during different life phases (1-7).The term ''atopic dermatitis'' was first proposed by Wise & Sulzberger in the 1930s, because of the close association with other atopic diseases such as bronchial asthma (AB) and allergic rhinitis (AR) (8).The various other names, such as atopic eczema, intrinsic allergic dermatitis; neurodermitis constitutionalis, endogenous eczema, eczema flexurarum, Besnier's prurigo, asthmaeczema, or hay fever-eczema, reflect the uncertainty in the understanding of the pathophysiology of this disease (9).The debate about the appropriate name is still going on, as all these definitions emphasize some aspects but may not cover all pathomechanisms or abnormalities.The revised nomenclature of allergic diseases, which appears in this issue of Allergy, proposes the term ''constitutional dermatitis'' for AD; however, in this review, the generally accepted term ''AD'' will still be used. Definition and diagnostic criteria of the intrinsic type of AD (IAD)Although most patients with AD have high concentrations of total and allergen-specific serum IgE levels and positive skin prick and intracutaneous test reactions of the immediate type to common environmental aller-Abbreviations.
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Diagnostic features of atopic dermatitis
Hanifin, J. M., & Rajka, G. (1980) · Acta Dermato-Venereologica · doi:10.2340/00015555924447 · Cited by 5,086
Williams, H. C., Leung, D. Y. M., Thestrup‐Pedersen, K., & David, T. J. (2000) · Cited by 4,596
Marsh, D. G., Neely, J. D., Breazeale, D. R., Ghosh, B., Freidhoff, L. R., Ehrlich-Kautzky, E., Schou, C., Krishnaswamy, G., & Beaty, T. H. (1994) · Science · doi:10.1126/science.8178175 · Cited by 1,003
Pène, J., Rousset, F., Brière, F., Chrétien, I., Bonnefoy, J. Y., Spits, H., Yokota, T., Arai, N., Arai, K., & Banchereau, J. (1988) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.85.18.6880 · Cited by 924
Mechanisms of eosinophil-associated inflammation
Gleich, G. J. (2000) · Journal of Allergy and Clinical Immunology · doi:10.1067/mai.2000.105712 · Cited by 785
Differential in situ cytokine gene expression in acute versus chronic atopic dermatitis.
Hamid, Q., Boguniewicz, M., & Leung, D. Y. (1994) · Journal of Clinical Investigation · doi:10.1172/jci117408 · Cited by 778
Atopic dermatitis: New insights and opportunities for therapeutic intervention
Leung, D. Y. M. (2000) · Journal of Allergy and Clinical Immunology · doi:10.1067/mai.2000.106484 · Cited by 721
Fahy, J. V., Fleming, H. E., Wong, H. H., Liu, J. T., Su, J. Q., Reimann, J., Fick, R. B., & Boushey, H. A. (1997) · American Journal of Respiratory and Critical Care Medicine · doi:10.1164/ajrccm.155.6.9196082 · Cited by 713
Atopic dermatitis: A genetic-epidemiologic study in a population-based twin sample
Larsen, F. S. (1993) · Journal of the American Academy of Dermatology · doi:10.1016/0190-9622(93)70099-f · Cited by 498
Direct demonstration of delayed eosinophil apoptosis as a mechanism causing tissue eosinophilia
Simon, H. U., Yousefi, S., Schranz, C., Schapowal, A., Bachert, C., & Blaser, K. (1997) · The Journal of Immunology · doi:10.4049/jimmunol.158.8.3902 · Cited by 468
Self-citation
Cited by
Johansson, S. G. O., Bieber, T., Dahl, R., Friedmann, P. S., Lanier, B. Q., Lockey, R. F., Motala, C., Ortega Martell, J. A., Platts-Mills, T. A. E., Ring, J., et al. (2004) · Journal of Allergy and Clinical Immunology · doi:10.1016/j.jaci.2003.12.591 · Cited by 2,391
Johansson, S. G. O., Hourihane, J. O., Bousquet, J., Bruijnzeel-Koomen, C., Dreborg, S., Haahtela, T., Kowalski, M. L., Mygind, N., Ring, J., van Cauwenberge, P., et al. (2001) · Allergy · doi:10.1034/j.1398-9995.2001.t01-1-00001.x · Cited by 1,723
Dupilumab Treatment in Adults with Moderate-to-Severe Atopic Dermatitis
Beck, L. A., Thaçi, D., Hamilton, J. D., Graham, N. M., Bieber, T., Rocklin, R., Ming, J. E., Ren, H., Kao, R., Simpson, E., et al. (2014) · New England Journal of Medicine · doi:10.1056/nejmoa1314768 · Cited by 1,421
Akdis, C. A., Akdis, M., Bieber, T., Bindslev‐Jensen, C., Boguniewicz, M., Eigenmann, P., Hamid, Q., Kapp, A., Leung, D. Y. M., Lipozencic, J., et al. (2006) · Allergy · doi:10.1111/j.1398-9995.2006.01153.x · Cited by 733
Self-citationCork, M. J., Robinson, D. A., Vasilopoulos, Y., Ferguson, A., Moustafa, M., MacGowan, A., Duff, G. W., Ward, S. J., & Tazi-Ahnini, R. (2006) · Journal of Allergy and Clinical Immunology · doi:10.1016/j.jaci.2006.04.042 · Cited by 525
Metadata
- Authors
- Schmid(‐Grendelmeier), P., Simon, D., Simon, H., Akdis, C. A., & Wüthrich, B.
- Venue
- Allergy
- Publisher
- Wiley
- Published
- 2001-09
- Volume
- 56
- Issue
- 9
- Pages
- 841-849
- ISSN
- 0105-4538 (ISSN-L)
- 1398-9995
- OpenAlex
- W1999387064
- Cited by
- 279
- References
- 74
- FWCI
- 11.15
- Type
- journal article
- Language
- en
- Open access
- bronze
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
How to cite
Cite CoChat for the checks on this page, and the paper for its own findings.
- This page
- CoChat. (2026). Citation check: Epidemiology, clinical features, and immunology of the “intrinsic” (non‐IgE‐mediated) type of atopic dermatitis (constitutional dermatitis). https://app.cochat.ai/papers/10.1034/j.1398-9995.2001.00144.x
- The paper
- Schmid(‐Grendelmeier), P., Simon, D., Simon, H., Akdis, C. A., & Wüthrich, B. (2001). Epidemiology, clinical features, and immunology of the “intrinsic” (non‐IgE‐mediated) type of atopic dermatitis (constitutional dermatitis). Allergy. https://doi.org/10.1034/j.1398-9995.2001.00144.x
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
