The DOI matches its Crossref record; 33 of the 33 references it cites were found in a registry or an index.
Cancer predisposition caused by elevated mitotic recombination in Bloom mice
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Citations
Cites
Mutations of bacteria from virus sensitivity to virus resistance
Luria, S. E., & Delbrück, M. (1943) · Genetics · doi:10.1093/genetics/28.6.491 · Cited by 3,894
Positional Cloning of the Werner's Syndrome Gene
Yu, C.-E., Oshima, J., Fu, Y.-H., Wijsman, E. M., Hisama, F., Alisch, R., Matthews, S., Nakura, J., Miki, T., Ouais, S., et al. (1996) · Science · doi:10.1126/science.272.5259.258 · Cited by 1,722
Growth differentiation factor-9 is required during early ovarian folliculogenesis
Dong, J., Albertini, D. F., Nishimori, K., Kumar, T. R., Lu, N., & Matzuk, M. M. (1996) · Nature · doi:10.1038/383531a0 · Cited by 1,646
The Bloom's syndrome gene product is homologous to RecQ helicases
Ellis, N. A., Groden, J., Ye, T.-Z., Straughen, J., Lennon, D. J., Ciocci, S., Proytcheva, M., & German, J. (1995) · Cell · doi:10.1016/0092-8674(95)90105-1 · Cited by 1,403
An Fgf8 mutant allelic series generated by Cre- and Flp-mediated recombination
Meyers, E. N., Lewandoski, M., & Martin, G. R. (1998) · Nature Genetics · doi:10.1038/ng0298-136 · Cited by 1,031
Gangloff, S., McDonald, J. P., Bendixen, C., Arthur, L., & Rothstein, R. (1994) · Molecular and Cellular Biology · doi:10.1128/mcb.14.12.8391 · Cited by 677
Mutations in RECQL4 cause a subset of cases of Rothmund-Thomson syndrome
Kitao, S., Shimamoto, A., Goto, M., Miller, R. W., Smithson, W. A., Lindor, N. M., & Furuichi, Y. (1999) · Nature Genetics · doi:10.1038/8788 · Cited by 666
Dietrich, W. (1993) · Cell · doi:10.1016/0092-8674(93)90484-8 · Cited by 654
EPSTKIN, C. J., MARTIN, G. M., SCHULTZ, A. L., & MOTULSKYS, A. G. (1966) · Medicine · doi:10.1097/00005792-196605000-00001 · Cited by 636
Bloom Syndrome: A Mendelian Prototype of Somatic Mutational Disease
German, J. (1993) · Medicine · doi:10.1097/00005792-199311000-00003 · Cited by 593
Cited by
Playing the End Game: DNA Double-Strand Break Repair Pathway Choice
Chapman, J. R., Taylor, M. R. G., & Boulton, S. J. (2012) · Molecular Cell · doi:10.1016/j.molcel.2012.07.029 · Cited by 1,625
The Bloom's syndrome helicase suppresses crossing over during homologous recombination
Wu, L., & Hickson, I. D. (2003) · Nature · doi:10.1038/nature02253 · Cited by 1,085
Mammalian Telomeres Resemble Fragile Sites and Require TRF1 for Efficient Replication
Sfeir, A., Kosiyatrakul, S. T., Hockemeyer, D., MacRae, S. L., Karlseder, J., Schildkraut, C. L., & de Lange, T. (2009) · Cell · doi:10.1016/j.cell.2009.06.021 · Cited by 990
Alternative-NHEJ Is a Mechanistically Distinct Pathway of Mammalian Chromosome Break Repair
Bennardo, N., Cheng, A., Huang, N., & Stark, J. M. (2008) · PLoS Genetics · doi:10.1371/journal.pgen.1000110 · Cited by 870
Endogenous DNA double-strand breaks: production, fidelity of repair, and induction of cancer
Vilenchik, M. M., & Knudson, A. G. (2003) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.2135498100 · Cited by 717
Metadata
- Authors
- Luo, G., Santoro, I. M., McDaniel, L. D., Nishijima, I., Mills, M., Youssoufian, H., Vogel, H., Schultz, R. A., & Bradley, A.
- Venue
- Nature Genetics
- Published
- 2000-12
- Volume
- 26
- Issue
- 4
- Pages
- 424-429
- ISSN
- 1061-4036 (ISSN-L)
- 1546-1718
- OpenAlex
- W1522566189
- Cited by
- 372
- References
- 33
- FWCI
- 8.11
- Type
- journal article
- Language
- en
- Open access
- closed
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- CoChat. (2026). Citation check: Cancer predisposition caused by elevated mitotic recombination in Bloom mice. https://app.cochat.ai/papers/10.1038/82548
- The paper
- Luo, G., Santoro, I. M., McDaniel, L. D., Nishijima, I., Mills, M., Youssoufian, H., Vogel, H., Schultz, R. A., & Bradley, A. (2000). Cancer predisposition caused by elevated mitotic recombination in Bloom mice. Nature Genetics. https://doi.org/10.1038/82548
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
