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Role of intracellular tyrosines in activating KIT-induced myeloproliferative disease
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“p85α, the regulatory subunit of class IA PI3 kinase (PI3K), is required for KITD814V-induced (murine homolog) transformation (10,11).”
From Pak and Rac GTPases promote oncogenic KIT–induced neoplasms (2013) · cites this paper as [11]
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Research works printed but not linked: 35
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Cited by
Pak and Rac GTPases promote oncogenic KIT–induced neoplasms
Martin, H., Mali, R. S., Ma, P., Chatterjee, A., Ramdas, B., Sims, E., Munugalavadla, V., Ghosh, J., Mattingly, R. R., Visconte, V., et al. (2013) · Journal of Clinical Investigation · doi:10.1172/jci67509 · Cited by 45
Self-citation
Metadata
- Authors
- Ma, P., Mali, R. S., Martin, H., Ramdas, B., Sims, E., & Kapur, R.
- Venue
- Leukemia
- Published
- 2012-02-02
- Volume
- 26
- Issue
- 7
- Pages
- 1499-1506
- ISSN
- 0887-6924
- 1476-5551
- PMC
- PMC4378686
- Cited by
- 13
- References
- 0
- Language
- en
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- CoChat. (2026). Citation check: Role of intracellular tyrosines in activating KIT-induced myeloproliferative disease. https://app.cochat.ai/papers/10.1038/leu.2012.22
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- Ma, P., Mali, R. S., Martin, H., Ramdas, B., Sims, E., & Kapur, R. (2012). Role of intracellular tyrosines in activating KIT-induced myeloproliferative disease. Leukemia. https://doi.org/10.1038/leu.2012.22