Cited by 11Neuroinflammation and Neurodegeneration MechanismsTryptophan and brain disordersStress Responses and Cortisol

Neurotherapeutic Implications of Brain-Immune Interactions

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Felger, Jennifer C, Miller, Andrew H·2014·Neuropsychopharmacology

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Crossref · 10.1038/npp.2013.213

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References 6 · Cited by 11

Authors
Felger, J. C., & Miller, A. H.
Affiliations
Emory University (US)
Corresponding author
Miller, Andrew H
Venue
Neuropsychopharmacology
Published
2013-12-09
Volume
39
Issue
1
Pages
242-243
ISSN
  • 0893-133X (ISSN-L)
  • 1740-634X
OpenAlex
W1966721217
Cited by
11
References
6
Citations by year
2025: 1 · 2023: 1 · 2020: 1 · 2019: 1 · 2018: 1 · 2017: 2 · 2016: 3 · 2015: 1
Type
other
Language
en
Open access
Locations
  • Neuropsychopharmacology (publishedVersion)
  • Europe PMC (PubMed Central) (publishedVersion)
  • PubMed Central (publishedVersion)
  • PubMed (publishedVersion)
Funders
  • Abbott Laboratories
  • Pfizer
  • AstraZeneca
  • GlaxoSmithKline
  • Emory University (UL1TR000454, KL2TR000455)
  • F. Hoffmann-La Roche
  • H. Lundbeck A/S
  • National Institutes of Health (KL2TR000455, UL1TR000454)
  • School of Medicine, Emory University (UL1TR000454)
  • Winship Cancer Institute
  • National Institute of Mental Health (R21MH077172, R21 MH0771172, R03MH100273, R01MH087604)
  • National Cancer Institute (P30CA138292)
  • National Center for Research Resources (UL1RR025008)
  • National Center for Advancing Translational Sciences (UL1TR000454 and KL2TR000455)
Topics
  • Life Sciences › Neuroscience › Neurology › Neuroinflammation and Neurodegeneration Mechanisms
  • Life Sciences › Neuroscience › Biological Psychiatry › Tryptophan and brain disorders
  • Life Sciences › Neuroscience › Behavioral Neuroscience › Stress Responses and Cortisol
Keywords
neuroimmune interactions, neuroimmunology, neuroinflammation, meningeal immunity, microglia, Rett syndrome, infliximab, treatment-resistant depression, cytokines, neurogenesis, spatial learning, inflammatory markers, TNF-α
MeSH terms
Infliximab, Antibodies, Monoclonal, Mental Disorders, Brain, Clinical Trials as Topic, Humans, Receptors, Immunologic, T-Lymphocytes, Tumor Necrosis Factor-alpha, Biomarkers, Neurogenesis, Depressive Disorder, Treatment-Resistant

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