Curcuminoids for the management of hypertriglyceridaemia
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Curcumin may modulate HDL production and HDL functionality biomarkers, including apolipoprotein AI, cholesteryl ester transfer protein, lecithin cholesterol acyl transferase, paraoxonase-1, myeloperoxidase, and lipoprotein-associated phospholipase A2.
“Curcumin may also modulate the production of HDL and biomarkers of HDL functionality, including apolipoprotein AI ([), cholesteryl ester transfer protein (Y), lecithin cholesterol acyl transferase ([), paraoxonase-1 (Y), myeloperoxidase (Y), and lipoprotein-associated phospholipase A2 (Y) (174-177).”
From The Role of Nutraceuticals in Statin Intolerant Patients (2018) · cites this paper as [176]
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References 15 · Cited by 172
- Authors
- Sahebkar, A.
- Affiliations
- Mashhad University of Medical Sciences (IR)
- Corresponding author
- Sahebkar, Amirhossein
- Venue
- Nature Reviews Cardiology
- Published
- 2014-01-07
- Volume
- 11
- Issue
- 2
- Pages
- 123-123
- ISSN
- 1759-5002 (ISSN-L)
- 1759-5010
- OpenAlex
- W2094564938
- Cited by
- 172
- References
- 15
- Citations by year
- 2026: 5 · 2025: 21 · 2024: 21 · 2023: 8 · 2022: 7 · 2021: 12 · 2020: 7 · 2019: 9 · 2018: 19 · 2017: 26 · 2016: 21 · 2015: 6 · 2014: 10
- Type
- journal article
- Language
- en
- Open access
- closed
- Licence
- Locations
- Nature Reviews Cardiology (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Medicine › Curcumin's Biomedical Applications
- Health Sciences › Medicine › Surgery › Natural Products and Biological Research
- Health Sciences › Medicine › Oncology › Saffron Plant Research Studies
- Keywords
- curcuminoids, hypertriglyceridemia, dyslipidemia, obesity, type 2 diabetes, hepatic steatosis, AMPK, atherosclerosis, C-reactive protein, NF-κB, anti-inflammatory activity, dietary supplementation, free fatty acids, lipoprotein metabolism, diabetic nephropathy, macrophage infiltration
- MeSH terms
- Hypolipidemic Agents, Cardiovascular Diseases, Humans, Triglycerides, Hypertriglyceridemia, Genetic Therapy, Risk Reduction Behavior
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