Maintenance of acinar cell organization is critical to preventing Kras-induced acinar-ductal metaplasia

Shi, G, DiRenzo, D, Qu, C, Barney, D, Miley, D, Konieczny, S F·2013·Oncogene

Cited by (163)

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  13. Activated K-RAS and its effect on morphological appearance
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  17. Acinar Ductal Metaplasia: Yap Fills a Gap
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  19. Endoplasmic Reticulum Stress and the Unfolded Protein Response in Exocrine Pancreas Physiology and Pancreatitis
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  20. Anti-proteolytic regulation of KRAS by USP9X/NDRG3 in KRAS-driven cancer development
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  21. Unraveling pancreatic ductal adenocarcinoma at single-cell resolution with spatial insights: From mechanisms to clinical translation
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  23. Stabilization of HIF-2α impacts pancreas growth
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  24. Chapter 1A – Embryologic development of the liver, biliary tract, and pancreas
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  28. Cellular Plasticity and Heterogeneity in Pancreatic Regeneration and Malignancy
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  34. Klf4-Tymp axis promotes inflammation-driven early tumorigenesis by enhancing kras mutation-induced acinar-to-ductal metaplasia through Pi3k/Akt and Mek/Erk pathways
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  35. Levels of retinol and retinoic acid in pancreatic cancer, type-2 diabetes and chronic pancreatitis
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  36. Animal Modeling of Pancreatitis-to-Cancer Progression
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  37. Embryologic development of the liver, biliary tract, and pancreas
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  38. Developmental Molecular Biology of the Pancreas
    Lewis Charles Murtaugh, Ondine B. Cleaver, Raymond J. MacDonald · 2016 · Springer eBooks
  39. Inactivation of HIPK2 attenuates KRASG12D activity and prevents pancreatic tumorigenesis
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  40. Mechanisms and targeted prevention of chronic pancreatitis-acinar to ductal metaplasia caused by a low concentration of di-(2-ethylhexyl)-phthalate
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  41. Pancreatic Carcinogenesis
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  42. Animal Modeling of Pancreatitis-to-Cancer Progression
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  43. Epigenetics of PNLIPRP1 in human pancreas reveals a molecular path between type 2 diabetes and pancreatic cancer
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  45. Somatostatin Receptor Subtype 2 as Pancreatic Tumorigenesis Suppressor: Identification of a New Targetable Signaling Node
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  46. JB Review Activated K-RAS and its effect on morphological appearance
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  47. The role of PAK4 in pancreatic cancer
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  48. Somatostatin Receptor Subtype 2 as Pancreatic Tumorigenesis Suppressor: Identification of a New Targetable Signaling Node
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  50. Lineage Tracing of Primary Human Pancreatic Acinar and Ductal Cells for Studying Acinar-to-Ductal Metaplasia
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  52. Interplay of ARID1A and EGFR signaling in controlling acinar cell reprogramming in the pancreas
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  53. The Loss of the E3 ubiquitin ligase TRIP12 inhibits Pancreatic Acinar Cell Plasticity and Tumor Cell Metastatic Capacity
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  54. The Normal Pancreas
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  56. Additional file 1 of Inactivation of HIPK2 attenuates KRASG12D activity and prevents pancreatic tumorigenesis
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