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Cited by 1Maternal and Neonatal HealthcareEnvironmental Justice and Health DisparitiesBirth, Development, and Health

The DOI matches its Crossref record; 58 of the 58 references it cites were found in a registry or an index; none of the 84 sources its claims cite is checked for support yet.

Environmental roots of neonatal health: how social and structural exposures shape early-life outcomes

Open access

Abstract

Neonatal health in the United States is influenced by a combination of biological, social, environmental, nutritional, and healthcare factors, with structural exposures-such as historical redlining, air pollution, and neighborhood disadvantages-associated with differences in perinatal outcomes. This review synthesizes nearly a century of multidisciplinary evidence linking adverse exposures in utero to persistent disparities in neonatal survival and neurodevelopment. We outline biological mechanisms, notably stress-mediated alterations of the fetal HPA axis and epigenetic changes, that may mediate the effects of social and environmental adversity before birth. Historical and contemporary data highlight the enduring impact of discriminatory policies on neonatal outcomes, emphasizing how structural racism and environmental injustice have been linked to higher rates of preterm birth and low birth weight. In addition, disparities in prenatal care access, food insecurity, infection, and inflammatory conditions represent important pathways through which social conditions may affect fetal growth and neonatal outcomes. Advances in exposomics and biomarker science now allow more precise measurement of early-life exposures, while place-based policy interventions, including urban greening and zoning reform, show promise in mitigating harm. Integrating these scientific and policy approaches is essential to disrupt intergenerational cycles of risk and advance perinatal equity. Bridging socioeconomics, environmental, and biological research offers a path forward to ensuring that all infants- regardless of background- have the opportunity for a healthy start. IMPACT: Review linking social and structural exposures, especially in redlining, air pollution, and environmental injustice disparities in neonatal survival and neurodevelopment. Highlights novel advances in exposomics and place-based policy interventions that target root causes and offer promising strategies to improve perinatal equity. Provides an integrated biological, clinical, and policy framework for disrupting intergenerational cycles of risk and advancing neonatal health equity.

Overview

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TL;DR

This review synthesizes nearly a century of evidence that social and structural exposures—such as historical redlining, air pollution, and neighborhood disadvantage—are linked to neonatal health disparities through stress-mediated HPA axis and epigenetic pathways. It highlights exposomics and place-based policy interventions as promising for perinatal equity, while emphasizing observational limits and cautious causal interpretation.

Key findings

  • Historical redlining and environmental injustice are linked to adverse birth outcomes: a 2022 systematic review found 86% of included studies reported worse outcomes, including higher preterm birth and low birth weight, among residents of historically redlined neighborhoods; New York City 2013–2017 data showed higher odds of preterm birth in grade D versus grade A tracts (OR 1.6; 95% CI 1.4–1.7).
  • Social risk can rival medical risk: a NICHD Neonatal Research Network study of over 7,400 infants found a composite social risk measure—including race as a social construct, insurance status, and maternal education—was a stronger predictor of post-discharge mortality and neurodevelopmental impairment than many traditional medical risk factors.
  • Biological embedding is supported by stress-pathway evidence: maternal stress may downregulate placental 11β-HSD2, increasing fetal glucocorticoid exposure and programming a hyper-reactive HPA axis; a 2015 meta-analysis found a small but significant increase in NR3C1 promoter methylation with prenatal maternal stress, and natural experiments (Dutch Hunger Winter, Quebec ice storm, COVID-19) link prenatal stress to HPA dysregulation, DNA methylation changes, and neonatal brain alterations.
  • Emerging exposomics and interventions show measurable signals: an NYC study identified 1,524 chemical features (36 novel) in maternal and cord sera; cord blood adductomics identified 51 oxidative stress-related adducts predicting bronchopulmonary dysplasia in preterm infants; and a 10% increase in tree canopy was associated with a 7% reduction in low birth weight risk in formerly redlined tracts.

Methods

  • Narrative review synthesizing nearly a century—ninety years—of multidisciplinary evidence spanning biological, clinical, historical, and policy perspectives on socioenvironmental stressors and neonatal health in the United States.
  • Draws on observational cohort studies, systematic reviews, meta-analyses, animal models, natural experiments, and emerging exposomics/neuroimaging methods; includes conceptual figures and tables summarizing evidence.
  • No primary data collection or human participants; the authors state the article is a review based exclusively on previously published literature.

Limitations

  • The authors state most available data are observational, subject to confounding, and cannot definitively establish causation; residual confounding and heterogeneity across studies limit strong causal inference.
  • The authors note that identifying U.S.-based longitudinal cohorts from the 1930s to 1980s that capture molecular pathways remains difficult because epigenetic assays did not become available until the 1990s, so understanding of earlier periods relies on extrapolation.
  • The authors state neuroimaging provides only a limited snapshot and does not reliably predict long-term outcomes, and that AI-based studies are largely observational with high-dimensional data requiring transparency, validation, and caution about prediction and clinical actionability; as evident from the text, no systematic search or quality appraisal is described for this narrative review.

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Integrity signals

Registry

Crossref · 10.1038/s41390-026-05139-5

Cites

58 of 58 references verified0 citation errors · 0 not checkable

Statistics

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Figures

Fig. 1 Conceptual illustration of Barker ' s hypothesis and DOHaD pathways relevant to neonatal outcomes. Maternal factors (e.g., stress, nutrition, infection, environmental exposure) act via placental mediators (e.g., blood fl ow, 11 B-HSD2, in fl ammation) to in fl uence fetal growth, HPA axis development, immune programming, and neonatal outcomes such as preterm birth, small-for-gestational-age birth, and low birth weight. The fi gure is schematic and not based on a single data set.
Fig. 1 Conceptual illustration of Barker ' s hypothesis and DOHaD pathways relevant to neonatal outcomes. Maternal factors (e.g., stress, nutrition, infection, environmental exposure) act via placental mediators (e.g., blood fl ow, 11 B-HSD2, in fl ammation) to in fl uence fetal growth, HPA axis development, immune programming, and neonatal outcomes such as preterm birth, small-for-gestational-age birth, and low birth weight. The fi gure is schematic and not based on a single data set.CC-BY-NC-ND · From the PDF
Fig. 2 Maternal stress and placental mediation of fetal HPA axis programming.
Fig. 2 Maternal stress and placental mediation of fetal HPA axis programming.CC-BY-NC-ND · From the PDF
Fig. 3 Environmental and structural determinants of neonatal health.
Fig. 3 Environmental and structural determinants of neonatal health.CC-BY-NC-ND · From the PDF

Verification

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Citations

Cites

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Metadata

Authors
Nawaz, K., Babata, K., Scheid, L., Bitar, L., Ortiz, N. V., Kota, S., & Chalak, L. F.
Affiliations
Children's Medical Center (US)
Corresponding author
Chalak, Lina F.
Venue
Pediatric Research
Published
2026-06-13
Volume
100
Issue
4
Pages
1527-1533
ISSN
  • 0031-3998 (ISSN-L)
  • 1530-0447
OpenAlex
W7164607221
Cited by
1
References
58
Citations by year
2026: 1
FWCI
1.26
Type
journal article
Language
en
Open access
Locations
  • Pediatric Research (publishedVersion, cc-by-nc-nd)
  • PubMed (publishedVersion)
Topics
  • Health Sciences › Medicine › Pediatrics, Perinatology and Child Health › Maternal and Neonatal Healthcare
  • Social Sciences › Sociology and Political Science › Environmental Justice and Health Disparities
  • Health Sciences › Medicine › Pediatrics, Perinatology and Child Health › Birth, Development, and Health
Keywords
newborn health, perinatal outcomes, social determinants of health, structural racism, environmental injustice, historical redlining, air pollution, neighborhood disadvantage, preterm birth, low birth weight, neonatal survival, brain development, epigenetics, prenatal care access, food insecurity, exposomics, biomarkers, place-based policy, urban greening, zoning reform

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Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.