The DOI matches its Crossref record.
Author Correction: Single-cell transcriptomics identifies Mcl-1 as a target for senolytic therapy in cancer
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First-generation senolytics face resistance mechanisms such as MCL-1 upregulation.
“resistance mechanisms such as MCL-1 upregulation, and negative outcomes in some models21”
From Emerging strategies in senotherapeutics: from broad-spectrum senolysis to precision reprogramming (2026) · cites this paper as [55]
The Author Correction (page not specified) describes image and figure-legend errors, including one involving an ABT263 image. It does not address MCL-1 upregulation as a resistance mechanism or negative outcomes in models; searches for those points returned only the correction text. (LLM, conf=0.76)
Experimental — accuracy not yet measuredSenescent cells can evade elimination by upregulating alternative survival pathways such as MCL-1 or modulating immune checkpoints.
“the capacity of SnCs to evade elimination by upregulating alternative survival pathways (such as MCL-1)54,55 or modulating immune checkpoints underscore the need for more sophisticated therapeutic approaches56.”
From Emerging strategies in senotherapeutics: from broad-spectrum senolysis to precision reprogramming (2026) · cites this paper as [55]
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Emerging strategies in senotherapeutics: from broad-spectrum senolysis to precision reprogramming
Zhang, W., Song, S., Zhang, Y., Pan, Y., Hu, D., & Wang, Y. (2026) · npj Aging · doi:10.1038/s41514-026-00355-z · Cited by 6
Metadata
- Authors
- Troiani, M., Colucci, M., D’Ambrosio, M., Guccini, I., Pasquini, E., Varesi, A., Valdata, A., Mosole, S., Revandkar, A., Attanasio, G., Rinaldi, A., Rinaldi, A., Bolis, M., Cippà, P., & Alimonti, A.
- Venue
- Nature Communications
- Published
- 2023-07-20
- Volume
- 14
- Issue
- 1
- ISSN
- 2041-1723
- Cited by
- 5
- References
- 0
- Language
- en
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