Cited by 285Single-cell and spatial transcriptomicsNeuroscience and Neuropharmacology ResearchNeurogenesis and neuroplasticity mechanisms

Human neocortical expansion involves glutamatergic neuron diversification

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Berg, Jim, Sorensen, Staci A., Ting, Jonathan T., Miller, Jeremy A., Chartrand, Thomas, Buchin, Anatoly, Bakken, Trygve E., Budzillo, Agata +139 more·2021·Nature

Abstract

Abstract The neocortex is disproportionately expanded in human compared with mouse 1,2 , both in its total volume relative to subcortical structures and in the proportion occupied by supragranular layers composed of neurons that selectively make connections within the neocortex and with other telencephalic structures. Single-cell transcriptomic analyses of human and mouse neocortex show an increased diversity of glutamatergic neuron types in supragranular layers in human neocortex and pronounced gradients as a function of cortical depth 3 . Here, to probe the functional and anatomical correlates of this transcriptomic diversity, we developed a robust platform combining patch clamp recording, biocytin staining and single-cell RNA-sequencing (Patch-seq) to examine neurosurgically resected human tissues. We demonstrate a strong correspondence between morphological, physiological and transcriptomic phenotypes of five human glutamatergic supragranular neuron types. These were enriched in but not restricted to layers, with one type varying continuously in all phenotypes across layers 2 and 3. The deep portion of layer 3 contained highly distinctive cell types, two of which express a neurofilament protein that labels long-range projection neurons in primates that are selectively depleted in Alzheimer’s disease 4,5 . Together, these results demonstrate the explanatory power of transcriptomic cell-type classification, provide a structural underpinning for increased complexity of cortical function in humans, and implicate discrete transcriptomic neuron types as selectively vulnerable in disease.

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  • BICCN and BICAN reference classifications provide a foundational reference to understand the cellular, molecular and epigenomic underpinnings of AD.

    “These reference classifications provide an extremely powerful foundational reference to understand the cellular, molecular and epigenomic underpinnings of AD.”

    From Integrated multimodal cell atlas of Alzheimer’s disease (2024) · cites this paper as [20]

    The Discussion (page unavailable) says the paper’s cell classification may help identify the cellular locus of disease and may guide future studies of neuronal vulnerability, including in Alzheimer’s disease. The Results link transcriptomically defined cell types to vulnerable neurons. Our copy does not establish the claimed epigenomic underpinnings of AD or identify the classifications as BICCN and BICAN references. (LLM, conf=0.86)

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References 70 · Cited by 285

Authors
Berg, J., Sorensen, S. A., Ting, J. T., Miller, J. A., Chartrand, T., Buchin, A., Bakken, T. E., Budzillo, A., Dee, N., Ding, S.-L., Gouwens, N. W., Hodge, R. D., Kalmbach, B., Lee, C., Lee, B. R., Alfiler, L., Baker, K., Barkan, E., Beller, A., Berry, K., Bertagnolli, D., Bickley, K., Bomben, J., Braun, T., Brouner, K., Casper, T., Chong, P., Crichton, K., Dalley, R., de Frates, R., Desta, T., Lee, S. D., D’Orazi, F., Dotson, N., Egdorf, T., Enstrom, R., Farrell, C., Feng, D., Fong, O., Furdan, S., Galakhova, A. A., Gamlin, C., Gary, A., Glandon, A., Goldy, J., Gorham, M., Goriounova, N. A., Gratiy, S., Graybuck, L., Gu, H., Hadley, K., Hansen, N., Heistek, T. S., Henry, A. M., Heyer, D. B., Hill, D., Hill, C., Hupp, M., Jarsky, T., Kebede, S., Keene, L., Kim, L., Kim, M.-H., Kroll, M., Latimer, C., Levi, B. P., Link, K. E., Mallory, M., Mann, R., Marshall, D., Maxwell, M., McGraw, M., McMillen, D., Melief, E., Mertens, E. J., Mezei, L., Mihut, N., Mok, S., Molnar, G., Mukora, A., Ng, L., Ngo, K., Nicovich, P. R., Nyhus, J., Olah, G., Oldre, A., Omstead, V., Ozsvar, A., Park, D., Peng, H., Pham, T., Pom, C. A., Potekhina, L., Rajanbabu, R., Ransford, S., Reid, D., Rimorin, C., Ruiz, A., Sandman, D., Sulc, J., Sunkin, S. M., Szafer, A., Szemenyei, V., Thomsen, E. R., Tieu, M., Torkelson, A., Trinh, J., Tung, H., Wakeman, W., Waleboer, F., Ward, K., Wilbers, R., Williams, G., Yao, Z., Yoon, J.-G., Anastassiou, C., Arkhipov, A., Barzo, P., Bernard, A., Cobbs, C., de Witt Hamer, P. C., Ellenbogen, R. G., Esposito, L., Ferreira, M., Gwinn, R. P., Hawrylycz, M. J., Hof, P. R., Idema, S., Jones, A. R., Keene, C. D., Ko, A. L., Murphy, G. J., Ng, L., Ojemann, J. G., Patel, A. P., Phillips, J. W., Silbergeld, D. L., Smith, K., Tasic, B., Yuste, R., Segev, I., de Kock, C. P. J., Mansvelder, H. D., Tamas, G., Zeng, H., Koch, C., & Lein, E. S.
Affiliations
  • Allen Institute for Brain Science (US)
  • University of Washington (US)
  • University of Szeged (HU)
  • Vrije Universiteit Amsterdam (NL)
  • Swedish Medical Center (US)
  • Icahn School of Medicine at Mount Sinai (US)
  • Amsterdam University Medical Centers (NL)
  • Columbia University (US)
  • Hebrew University of Jerusalem (IL)
Corresponding author
Lein, Ed S.
ORCID
Venue
Nature
Published
2021-10-06
Volume
598
Issue
7879
Pages
151-158
ISSN
  • 0028-0836 (ISSN-L)
  • 1476-4687
OpenAlex
W3204722966
Cited by
285
References
70
Citations by year
2026: 37 · 2025: 57 · 2024: 61 · 2023: 72 · 2022: 44 · 2021: 13
FWCI
14.91
Type
journal article
Language
en
Open access
Locations
  • Nature (publishedVersion, cc-by)
  • PubMed Central (publishedVersion, cc-by)
  • PubMed (publishedVersion)
  • VU Research Portal (publishedVersion, other-oa)
  • Repository of the Academy's Library (Library of the Hungarian Academy of Sciences) (acceptedVersion)
  • RePEc: Research Papers in Economics (submittedVersion)
  • Pure Amsterdam UMC (submittedVersion, cc-by)
  • Amsterdam UMC (VU Amsterdam) - Institutional Repository (submittedVersion, other-oa)
  • Data Archiving and Networked Services (DANS) (submittedVersion, other-oa)
Funders
  • University of Washington
  • Amsterdam University Medical Centers
  • European Commission (HORIZON2020, 945539)
  • Agence Nationale de la Recherche (ANR-17-NEU3-0004)
  • Nederlandse Organisatie voor Wetenschappelijk Onderzoek (024.004.012, 013-17-001)
  • Magyar Tudományos Akadémia
  • Emberi Eroforrások Minisztériuma
  • Szegedi Tudományegyetem
  • Cancer Center Amsterdam
  • National Institutes of Health (5u01mh114812-03, 945539, U01MH114812)
  • Horizon 2020 Framework Programme (#945539, GINOP-2.3.2)
  • National Research, Development and Innovation Office
  • National Institute on Aging (P30AG066509)
  • National Institute of Mental Health (945539, R01MH116247, U01MH105982, U01MH114812, U19MH114830)
  • National Eye Institute (R01EY023173)
  • National Institute of Neurological Disorders and Stroke (R01NS120300)
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Single-cell and spatial transcriptomics
  • Life Sciences › Neuroscience › Cellular and Molecular Neuroscience › Neuroscience and Neuropharmacology Research
  • Life Sciences › Neuroscience › Developmental Neuroscience › Neurogenesis and neuroplasticity mechanisms
Keywords
Patch-seq, single-cell transcriptomics, neurofilaments, cortical cell types
MeSH terms
Alzheimer Disease, Animals, Collagen, Electrophysiology, Female, Humans, Lysine, Male, Neurons, Extracellular Matrix Proteins, Patch-Clamp Techniques, Glutamic Acid, Neocortex, Cell Shape, Mice, Transcriptome

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