Hypermetabolism and energetic constraints in mitochondrial disorders
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Sercel and colleagues wrote that it was long thought that mitochondrial diseases are linked to ATP deficiency, but that several studies now rather link mitochondrial and OXPHOS deficiency to hypermetabolism.
“In a recent comment, Sercel and colleagues wrote that it was long thought that mitochondrial diseases are linked to ATP deficiency, but that several studies now rather link mitochondrial and OXPHOS deficiency to hypermetabolism [50].”
From APOE4 Increases Energy Metabolism in APOE-Isogenic iPSC-Derived Neurons (2024) · cites this paper as [50]
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Forsström, S., Jackson, C. B., Carroll, C. J., Kuronen, M., Pirinen, E., Pradhan, S., Marmyleva, A., Auranen, M., Kleine, I.-M., Khan, N. A., et al. (2019) · Cell Metabolism · doi:10.1016/j.cmet.2019.08.019 · Cited by 252 · see citing works
A standard calculation methodology for human doubly labeled water studies
Speakman, J. R., Yamada, Y., Sagayama, H., Berman, E. S. F., Ainslie, P. N., Andersen, L. F., Anderson, L. J., Arab, L., Baddou, I., Bedu-Addo, K., et al. (2021) · Cell Reports Medicine · doi:10.1016/j.xcrm.2021.100203 · Cited by 143 · see citing works
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Research works printed but not linked: 15
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APOE4 Increases Energy Metabolism in APOE-Isogenic iPSC-Derived Neurons
Budny, V., Knöpfli, Y., Meier, D., Zürcher, K., Bodenmann, C., Peter, S. L., Müller, T., Tardy, M., Cortijo, C., & Tackenberg, C. (2024) · Cells · doi:10.3390/cells13141207 · Cited by 15 · see citing works
- Authors
- Sercel, A. J., Sturm, G., Gallagher, D., St-Onge, M.-P., Kempes, C. P., Pontzer, H., Hirano, M., & Picard, M.
- Affiliations
- Columbia University Irving Medical Center (US)
- University of California, San Francisco (US)
- Santa Fe Institute (US)
- Duke University (US)
- Corresponding author
- Picard, Martin
- ORCID
- Venue
- Nature Metabolism
- Published
- 2024-02-09
- Volume
- 6
- Issue
- 2
- Pages
- 192-195
- ISSN
- 2522-5812 (ISSN-L)
- PMC
- PMC12066245
- OpenAlex
- W4391680617
- Cited by
- 34
- References
- 2
- Citations by year
- 2026: 13 · 2025: 14 · 2024: 7
- FWCI
- 4.39
- Type
- journal article
- Language
- en
- Open access
- green
- www.ncbi.nlm.nih.gov
- Licence
- Locations
- Nature Metabolism (publishedVersion)
- PubMed (publishedVersion)
- PubMed Central (acceptedVersion)
- Funders
- Nathaniel Wharton Fund
- National Institutes of Health (DK26687)
- National Institute on Aging (R01AG066828)
- National Heart, Lung, and Blood Institute (R01HL142648, R35HL155670)
- National Institute of Mental Health (R01MH122706, R01MH119336)
- National Institute of Diabetes and Digestive and Kidney Diseases (P30DK026687)
- National Institute of Neurological Disorders and Stroke (U54NS078059)
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Mitochondrial Function and Pathology
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Clinical Biochemistry › Metabolism and Genetic Disorders
- Health Sciences › Medicine › Epidemiology › Autophagy in Disease and Therapy
- Keywords
- mitochondrial disease, hypermetabolism, oxidative phosphorylation defects, ATP deficiency, stress response, energy metabolism, energetic constraints, pathophysiology
- MeSH terms
- Humans, Mitochondria, Mitochondrial Diseases
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