The DOI matches its Crossref record; 7 of the 7 references it cites were found in a registry or an index.
High level expression of ΔN-p63: a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)?
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Citations
Cites
Mdm2 promotes the rapid degradation of p53
Haupt, Y., Maya, R., Kazaz, A., & Oren, M. (1997) · Nature · doi:10.1038/387296a0 · Cited by 4,506
Regulation of p53 stability by Mdm2
Kubbutat, M. H. G., Jones, S. N., & Vousden, K. H. (1997) · Nature · doi:10.1038/387299a0 · Cited by 3,312 · cited 1×
“This is because hMdm2 is a negative regulator of p53 through its capacity to bind to the transactivation domain of p53 and suppress transcription and also to target p53 for ubiquitin-dependent, proteasome-mediated proteolysis (Oliner et al., 1993; Haupt et al., 1997; Kubbutat et al., 1997).”
BackgroundAmplification of a gene encoding a p53-associated protein in human sarcomas
Oliner, J. D., Kinzler, K. W., Meltzer, P. S., George, D. L., & Vogelstein, B. (1992) · Nature · doi:10.1038/358080a0 · Cited by 1,949
Zhang, Y., Xiong, Y., & Yarbrough, W. G. (1998) · Cell · doi:10.1016/s0092-8674(00)81401-4 · Cited by 1,626
Myc signaling via the ARF tumor suppressor regulates p53-dependent apoptosis and immortalization
Zindy, F., Eischen, C. M., Randle, D. H., Kamijo, T., Cleveland, J. L., Sherr, C. J., & Roussel, M. F. (1998) · Genes & Development · doi:10.1101/gad.12.15.2424 · Cited by 1,245
Disruption of the ARF-Mdm2-p53 tumor suppressor pathway in Myc-induced lymphomagenesis
Eischen, C. M., Weber, J. D., Roussel, M. F., Sherr, C. J., & Cleveland, J. L. (1999) · Genes & Development · doi:10.1101/gad.13.20.2658 · Cited by 819
The CDKN2A (p16) Gene and Human Cancer
Foulkes, W. D., Flanders, T. Y., Pollock, P. M., & Hayward, N. K. (1997) · Molecular Medicine · doi:10.1007/bf03401664 · Cited by 292
Cited by
Focus on nasopharyngeal carcinoma
Lo, K. W., To, K. F., & Huang, D. P. (2004) · Cancer Cell · doi:10.1016/s1535-6108(04)00119-9 · Cited by 559
p63 and p73: Roles in Development and Tumor Formation
Moll, U. M., & Slade, N. (2004) · Molecular Cancer Research · doi:10.1158/1541-7786.371.2.7 · Cited by 486
Gaiddon, C., Lokshin, M., Ahn, J., Zhang, T., & Prives, C. (2001) · Molecular and Cellular Biology · doi:10.1128/mcb.21.5.1874-1887.2001 · Cited by 464
The evolution of diverse biological responses to DNA damage: insights from yeast and p53
Wahl, G. M., & Carr, A. M. (2001) · Nature Cell Biology · doi:10.1038/ncb1201-e277 · Cited by 376
Dawson, C. W., Port, R. J., & Young, L. S. (2012) · Seminars in Cancer Biology · doi:10.1016/j.semcancer.2012.01.004 · Cited by 355
Metadata
- Authors
- Crook, T., Nicholls, J. M., Brooks, L., O'Nions, J., & Allday, M. J.
- Venue
- Oncogene
- Published
- 2000-07-13
- Volume
- 19
- Issue
- 30
- Pages
- 3439-3444
- ISSN
- 0950-9232 (ISSN-L)
- 1476-5594
- OpenAlex
- W1972334859
- Cited by
- 194
- References
- 7
- FWCI
- 5.54
- Type
- journal article
- Language
- en
- Open access
- closed
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- CoChat. (2026). Citation check: High level expression of ΔN-p63: a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)? https://app.cochat.ai/papers/10.1038/sj.onc.1203656
- The paper
- Crook, T., Nicholls, J. M., Brooks, L., O'Nions, J., & Allday, M. J. (2000). High level expression of ΔN-p63: a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)? Oncogene. https://doi.org/10.1038/sj.onc.1203656
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.