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Cited by 161

The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.

Studies on lipid peroxidation in normal and tumour tissues. The Yoshida rat liver tumour

Open access
Cheeseman, K H, Emery, S, Maddix, S P, Slater, T F, Burton, G W, Ingold, K U·1988·Biochemical Journal

Abstract

Reduced rates of lipid peroxidation have been observed in Yoshida hepatoma cells and microsomes when compared with appropriate control tissue (normal rat liver) under the same pro-oxidant conditions. The pro-oxidant conditions used were incubation with NADPH+ADP+iron or ascorbate+iron or exposure to gamma-irradiation. As previously shown with the Novikoff hepatoma, the relative concentrations of alpha-tocopherol and polyunsaturated fatty acids are important in conferring resistance to lipid peroxidation in the Yoshida hepatoma. Furthermore, NADPH-cytochrome c reductase and the NADPH-cytochrome P-450 electron transport chain, which are involved in the initiation and propagation of certain types of lipid peroxidation, are found at very much reduced levels in the Yoshida hepatoma. The relative importance of these aberrations are discussed.

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Metadata

Authors
Cheeseman, K. H., Emery, S., Maddix, S. P., Slater, T. F., Burton, G. W., & Ingold, K. U.
Venue
Biochemical Journal
Published
1988-02-15
Volume
250
Issue
1
Pages
247-252
ISSN
  • 0264-6021 (ISSN-L)
  • 1470-8728
OpenAlex
W183553423
Cited by
161
References
1
FWCI
11.96
Type
journal article
Language
en
Open access
green

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CoChat. (2026). Citation check: Studies on lipid peroxidation in normal and tumour tissues. The Yoshida rat liver tumour. https://app.cochat.ai/papers/10.1042/bj2500247
The paper
Cheeseman, K. H., Emery, S., Maddix, S. P., Slater, T. F., Burton, G. W., & Ingold, K. U. (1988). Studies on lipid peroxidation in normal and tumour tissues. The Yoshida rat liver tumour. Biochemical Journal. https://doi.org/10.1042/bj2500247

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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