The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
The Origin and Development of Human Tumors Studied with Cell Markers
Abstract
AMONG the more remarkable but least understood homeostatic mechanisms of higher organisms are those that govern tissue development, growth and repair. Considering the trillions of cells involved and the complexity and duration of these processes from fertilization until death, it is surprising that functional derangements such as neoplasia do not occur more frequently. Even so, cancer is one of the chief causes of human suffering and death, and, as such, has been the subject of intense research. In animals, such factors as genetic predisposition, viruses, radiation, chemicals, dietary components and hormones are known carcinogens, but they have been conclusively associated . . .
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Cites
ROWLEY, J. D. (1973) · Nature · doi:10.1038/243290a0 · Cited by 4,291
Cited by
Nowell, P. C. (1976) · Science · doi:10.1126/science.959840 · Cited by 6,471
The Pathogenesis of Atherosclerosis: (Second of Two Parts)
Ross, R., & Glomset, J. A. (1976) · New England Journal of Medicine · doi:10.1056/nejm197608192950805 · Cited by 1,163
Cancer and ageing in mice and men
Peto, R., Roe, F. J., Lee, P. N., Levy, L., & Clack, J. (1975) · British Journal of Cancer · doi:10.1038/bjc.1975.242 · Cited by 425
Telomere Length Abnormalities Occur Early in the Initiation of Epithelial Carcinogenesis
Meeker, A. K., Hicks, J. L., Iacobuzio-Donahue, C. A., Montgomery, E. A., Westra, W. H., Chan, T. Y., Ronnett, B. M., & De Marzo, A. M. (2004) · Clinical Cancer Research · doi:10.1158/1078-0432.ccr-0984-03 · Cited by 306
Metadata
- Authors
- Fialkow, P. J.
- Venue
- New England Journal of Medicine
- Publisher
- Massachusetts Medical Society
- Published
- 1974-07-04
- Volume
- 291
- Issue
- 1
- Pages
- 26-35
- ISSN
- 0028-4793 (ISSN-L)
- 1533-4406
- OpenAlex
- W2340761814
- Cited by
- 209
- References
- 1
- FWCI
- 9.82
- Type
- journal article
- Language
- en
- Open access
- closed
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
