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Cited by 318

The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

Pathogenesis of Diseases Induced by Human Lymphotropic Virus Type I Infection

Flier, Jeffrey S., Underhill, Lisa H., Hollsberg, Per, Hafler, David A.·1993·New England Journal of Medicine

Abstract

Although retroviruses were among the earliest viruses discovered,1,2 they were linked to human disease only in the early 1980s,35 with the study of T-cell lines from patients with the lymphoproliferative disorder adult T-cell leukemia6. T cells from these patients were shown to release a retrovirus, later known as human T-cell lymphotropic virus type I (HTLV-I), the first member of a new class of human viruses7. This led to the investigation of a number of idiopathic disorders. In 1985, tropical spastic paraparesis, an endemic disease in the West Indies,8 was associated with immune reactivity to HTLV-I in . . .

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Crossref · 10.1056/nejm199304223281608

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Metadata

Authors
Flier, J. S., Underhill, L. H., Hollsberg, P., & Hafler, D. A.
Venue
New England Journal of Medicine
Published
1993-04-22
Volume
328
Issue
16
Pages
1173-1182
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W2336211911
Cited by
318
References
3
FWCI
10.35
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Pathogenesis of Diseases Induced by Human Lymphotropic Virus Type I Infection. https://app.cochat.ai/papers/10.1056/nejm199304223281608
The paper
Flier, J. S., Underhill, L. H., Hollsberg, P., & Hafler, D. A. (1993). Pathogenesis of Diseases Induced by Human Lymphotropic Virus Type I Infection. New England Journal of Medicine. https://doi.org/10.1056/nejm199304223281608

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.