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Cited by 226

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Tumor-Induced Osteomalacia -- Unveiling a New Hormone

Econs, Michael J., Drezner, Marc K.·1994·New England Journal of Medicine

Abstract

Phosphate plays a critical part in the regulation of cell metabolism, and phosphate homeostasis is closely regulated in normal humans. Indeed, like serum calcium, serum phosphate is maintained within a narrow range of values, and people with abnormal concentrations may have a predisposition to life-threatening conditions, such as hemolysis, myopathy, hypocalcemia, and nephrocalcinosis.The principal organ that regulates phosphate homeostasis is the kidney. Regulation is accomplished partly through variation in glomerular filtration of phosphate but primarily through variation in renal tubular reabsorption of phosphate. These variations are detectable within 24 hours after a change in dietary phosphate intake and even . . .

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Metadata

Authors
Econs, M. J., & Drezner, M. K.
Venue
New England Journal of Medicine
Published
1994-06-09
Volume
330
Issue
23
Pages
1679-1681
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W2020138048
Cited by
226
References
0
FWCI
7.67
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Tumor-Induced Osteomalacia -- Unveiling a New Hormone. https://app.cochat.ai/papers/10.1056/nejm199406093302310
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Econs, M. J., & Drezner, M. K. (1994). Tumor-Induced Osteomalacia -- Unveiling a New Hormone. New England Journal of Medicine. https://doi.org/10.1056/nejm199406093302310

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.