The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.
The Biology of Chronic Myeloid Leukemia
Abstract
Chronic myeloid leukemia (CML) is a clonal myeloproliferative expansion of transformed, primitive hematopoietic progenitor cells. It involves myeloid, monocytic, erythroid, megakaryocytic, B-lymphoid, and occasionally T-lymphoid lineages.1 CML was the first human disease in which a specific abnormality of the karyotype — the Philadelphia (Ph) chromosome — could be linked to pathogenetic events of leukemogenesis.2 It was among the first neoplastic diseases in which therapy with a biologic agent (interferon) was found to suppress the leukemic clone and prolong survival.3 Although heterogeneous, CML is the best-characterized leukemia at a molecular level, and studies in recent years have helped to define further . . .
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ROWLEY, J. D. (1973) · Nature · doi:10.1038/243290a0 · Cited by 4,291
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Schlaepfer, D. D., Hanks, S. K., Hunter, T., & Geer, P. V. D. (1994) · Nature · doi:10.1038/372786a0 · Cited by 1,595
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Druker, B. J., Sawyers, C. L., Kantarjian, H., Resta, D. J., Reese, S. F., Ford, J. M., Capdeville, R., & Talpaz, M. (2001) · New England Journal of Medicine · doi:10.1056/nejm200104053441402 · Cited by 2,713
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Metadata
- Authors
- Faderl, S., Talpaz, M., Estrov, Z., O'Brien, S., Kurzrock, R., & Kantarjian, H. M.
- Venue
- New England Journal of Medicine
- Publisher
- Massachusetts Medical Society
- Published
- 1999-07-15
- Volume
- 341
- Issue
- 3
- Pages
- 164-172
- ISSN
- 0028-4793 (ISSN-L)
- 1533-4406
- OpenAlex
- W2314938037
- Cited by
- 1,302
- References
- 2
- FWCI
- 23.44
- Type
- journal article
- Language
- en
- Open access
- closed
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- Faderl, S., Talpaz, M., Estrov, Z., O'Brien, S., Kurzrock, R., & Kantarjian, H. M. (1999). The Biology of Chronic Myeloid Leukemia. New England Journal of Medicine. https://doi.org/10.1056/nejm199907153410306
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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