Cited by 813Advanced Breast Cancer TherapiesPI3K/AKT/mTOR signaling in cancerBreast Cancer Treatment Studies

Capivasertib in Hormone Receptor–Positive Advanced Breast Cancer

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Turner, Nicholas C., Oliveira, Mafalda, Howell, Sacha J., Dalenc, Florence, Cortes, Javier, Gomez Moreno, Henry L., Hu, Xichun, Jhaveri, Komal +15 more·2023·New England Journal of Medicine

Abstract

AKT pathway activation is implicated in endocrine-therapy resistance. Data on the efficacy and safety of the AKT inhibitor capivasertib, as an addition to fulvestrant therapy, in patients with hormone receptor–positive advanced breast cancer are limited. Download a PDF of the Research Summary. In a phase 3, randomized, double-blind trial, we enrolled eligible pre-, peri-, and postmenopausal women and men with hormone receptor–positive, human epidermal growth factor receptor 2–negative advanced breast cancer who had had a relapse or disease progression during or after treatment with an aromatase inhibitor, with or without previous cyclin-dependent kinase 4 and 6 (CDK4/6) inhibitor therapy. Patients were randomly assigned in a 1:1 ratio to receive capivasertib plus fulvestrant or placebo plus fulvestrant. The dual primary end point was investigator-assessed progression-free survival assessed both in the overall population and among patients with AKT pathway–altered (PIK3CA, AKT1, or PTEN) tumors. Safety was assessed. Overall, 708 patients underwent randomization; 289 patients (40.8%) had AKT pathway alterations, and 489 (69.1%) had received a CDK4/6 inhibitor previously for advanced breast cancer. In the overall population, the median progression-free survival was 7.2 months in the capivasertib–fulvestrant group, as compared with 3.6 months in the placebo–fulvestrant group (hazard ratio for progression or death, 0.60; 95% confidence interval [CI], 0.51 to 0.71; P<0.001). In the AKT pathway–altered population, the median progression-free survival was 7.3 months in the capivasertib–fulvestrant group, as compared with 3.1 months in the placebo–fulvestrant group (hazard ratio, 0.50; 95% CI, 0.38 to 0.65; P<0.001). The most frequent adverse events of grade 3 or higher in patients receiving capivasertib–fulvestrant were rash (in 12.1% of patients, vs. in 0.3% of those receiving placebo–fulvestrant) and diarrhea (in 9.3% vs. 0.3%). Adverse events leading to discontinuation were reported in 13.0% of the patients receiving capivasertib and in 2.3% of those receiving placebo. Capivasertib–fulvestrant therapy resulted in significantly longer progression-free survival than treatment with fulvestrant alone among patients with hormone receptor–positive advanced breast cancer whose disease had progressed during or after previous aromatase inhibitor therapy with or without a CDK4/6 inhibitor. (Funded by AstraZeneca and the National Cancer Institute; CAPItello-291 ClinicalTrials.gov number, NCT04305496.) QUICK TAKE VIDEO SUMMARYCapivasertib in Advanced Breast Cancer 02:14

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  • Use of fulvestrant as a single agent is now suboptimal in the second line.

    “the use of which as a single-agent is now suboptimal in the second line”

    From Abemaciclib plus a nonsteroidal aromatase inhibitor as initial therapy for HR+, HER2- advanced breast cancer: final overall survival results of MONARCH 3. (2024) · cites this paper as [25]

    The Discussion (page not provided) says single-agent endocrine therapy has poor outcomes after prior CDK4/6 inhibitor treatment. The Abstract Results also show longer progression-free survival with capivasertib–fulvestrant than with fulvestrant alone. But the claim applies the judgment broadly to second-line fulvestrant; the trial studied patients whose disease had progressed after aromatase inhibitor therapy, with or without a CDK4/6 inhibitor, and permitted up to two prior endocrine-therapy lines. (LLM, conf=0.83)

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Authors
Turner, N. C., Oliveira, M., Howell, S. J., Dalenc, F., Cortes, J., Gomez Moreno, H. L., Hu, X., Jhaveri, K., Krivorotko, P., Loibl, S., Morales Murillo, S., Okera, M., Park, Y. H., Sohn, J., Toi, M., Tokunaga, E., Yousef, S., Zhukova, L., de Bruin, E. C., Grinsted, L., Schiavon, G., Foxley, A., & Rugo, H. S.
Affiliations
  • AstraZeneca (United Kingdom) (GB)
  • From the Royal Marsden Hospital, Institute of Cancer Research, London (N.C.T.), the Christie NHS Foundation Trust, Manchester (S.J.H.), and Oncology Research and Development, AstraZeneca, Cambridge (E.C.B., L.G., G.S., A.F.) — all in the United Kingdom; the Department of Medical Oncology, Vall d’Hebron University Hospital (M. Oliveira), the Breast Cancer Unit, Vall d’Hebron Institute of Oncology (M. Oliveira), the Department of Oncology, International Breast Cancer Center, Pangaea Oncology, Quiron Group, Medica Scientia Innovation Research (J.C.), and Institut de Recerca Biomèdica (S.M.M.), Barcelona, and the Department of Medicine, Faculty of Biomedical and Health Sciences, Universidad Europea de Madrid, Madrid (J.C.) — all in Spain; Institut Claudius Regaud, Institut Universitaire du Cancer–Oncopole Toulouse, Toulouse, France (F.D.); Departamento de Oncología Médica, Instituto Nacional de Enfermedades Neoplásicas, and Universidad Ricardo Palma — both in Lima, Peru (H.L.G.M.); Shanghai Cancer Center, Fuda...
Venue
New England Journal of Medicine
Published
2023-06
Volume
388
Issue
22
Pages
2058-2070
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W4378801565
Cited by
813
References
2
Citations by year
2026: 269 · 2025: 282 · 2024: 217 · 2023: 45
FWCI
149.97
Type
journal article
Language
en
Open access
Locations
  • New England Journal of Medicine (publishedVersion)
  • PubMed (publishedVersion)
  • PubMed Central (acceptedVersion)
  • Research Explorer (The University of Manchester) (submittedVersion, cc-by)
  • Archivio Istituzionale della Ricerca - Università degli Studi della Campania "Luigi Vanvitelli" (submittedVersion)
  • IRIS UNIMORE (University of Modena and Reggio Emilia) (submittedVersion, cc-by)
  • LA Referencia (Red Federada de Repositorios Institucionales de Publicaciones Científicas) (submittedVersion, cc-by)
Funders
AstraZeneca
Topics
  • Health Sciences › Medicine › Pulmonary and Respiratory Medicine › Advanced Breast Cancer Therapies
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › PI3K/AKT/mTOR signaling in cancer
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Cancer Research › Breast Cancer Treatment Studies
Keywords
AKT inhibitors, hormone receptor-positive breast cancer, advanced breast cancer, fulvestrant, endocrine therapy resistance, PIK3CA, PTEN, CDK4/6 inhibitors, progression-free survival, adverse events, rash, diarrhea, drug safety, drug efficacy, aromatase inhibitor resistance, HER2-negative breast cancer
MeSH terms
Fulvestrant, Antineoplastic Combined Chemotherapy Protocols, Breast Neoplasms, Double-Blind Method, Female, Humans, Male, Neoplasm Recurrence, Local, Pyrimidines, Pyrroles, Erb-b2 Receptor Tyrosine Kinases, Receptor, ErbB-2, Aromatase Inhibitors, Proto-Oncogene Proteins c-akt

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