Cited by 158Metabolism and Genetic DisordersLipid metabolism and biosynthesisDrug Transport and Resistance Mechanisms

Intrinsic acyl-CoA thioesterase activity of a peroxisomal ATP binding cassette transporter is required for transport and metabolism of fatty acids

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De Marcos Lousa, Carine, van Roermund, Carlo W. T., Postis, Vincent L. G., Dietrich, Daniela, Kerr, Ian D., Wanders, Ronald J. A., Baldwin, Stephen A., Baker, Alison +1 more·2013·Proceedings of the National Academy of Sciences

Abstract

Peroxisomes are organelles that perform diverse metabolic functions in different organisms, but a common function is β-oxidation of a variety of long chain aliphatic, branched, and aromatic carboxylic acids. Import of substrates into peroxisomes for β-oxidation is mediated by ATP binding cassette (ABC) transporter proteins of subfamily D, which includes the human adrenoleukodystropy protein (ALDP) defective in X-linked adrenoleukodystrophy (X-ALD). Whether substrates are transported as CoA esters or free acids has been a matter of debate. Using COMATOSE (CTS), a plant representative of the ABCD family, we demonstrate that there is a functional and physical interaction between the ABC transporter and the peroxisomal long chain acyl-CoA synthetases (LACS)6 and -7. We expressed recombinant CTS in insect cells and showed that membranes from infected cells possess fatty acyl-CoA thioesterase activity, which is stimulated by ATP. A mutant, in which Serine 810 is replaced by asparagine (S810N) is defective in fatty acid degradation in vivo, retains ATPase activity but has strongly reduced thioesterase activity, providing strong evidence for the biological relevance of this activity. Thus, CTS, and most likely the other ABCD family members, represent rare examples of polytopic membrane proteins with an intrinsic additional enzymatic function that may regulate the entry of substrates into the β-oxidation pathway. The cleavage of CoA raises questions about the side of the membrane where this occurs and this is discussed in the context of the peroxisomal coenzyme A (CoA) budget.

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  • By binding to ATP, ALDP transports VLCFAcyl-CoA into the peroxisomal matrix, where they are β-oxidized into medium-chain fatty acids (MCFAs) and MCFAcyl-carnitines, propionyl-CoA, and acetyl-CoA.

    “By binding to ATP, ALDP transports VLCFAcyl-CoA into the peroxisomal matrix [143,159,160], where they are β-oxidized into medium-chain fatty acids (MCFAs) and MCFAcyl-carnitines [161], propionyl-CoA, and acetyl-CoA.”

    From Revisiting the Pathogenesis of X-Linked Adrenoleukodystrophy (2025) · cites this paper as [159]

    The Introduction (p. 2) identifies ALDP as a peroxisomal ABC transporter involved in very-long-chain fatty-acid metabolism, and Fig. 4 (p. 4) describes ATP-powered transport for the related plant transporter CTS. But the paper's model says acyl-CoA is cleaved and free fatty acid enters the peroxisome; it does not establish that ALDP transports intact VLCFA-acyl-CoA into the matrix. The listed β-oxidation products are outside the clause citing this source. (LLM, conf=0.88)

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Authors
De Marcos Lousa, C., van Roermund, C. W. T., Postis, V. L. G., Dietrich, D., Kerr, I. D., Wanders, R. J. A., Baldwin, S. A., Baker, A., & Theodoulou, F. L.
Affiliations
  • Centre for Plant Sciences, School of Biology, University of Leeds, Leeds LS2 9JT, United Kingdom;
  • Laboratory of Genetic Metabolic Diseases, Departments of Pediatrics and Clinical Chemistry, Academic Medical Centre, University of Amsterdam, 1105 AZ Amsterdam, The Netherlands;
  • University of Leeds (GB)
  • Rothamsted Research (GB)
  • University of Nottingham (GB)
  • Amsterdam UMC Location University of Amsterdam (NL)
Corresponding author
Baker, Alison
Venue
Proceedings of the National Academy of Sciences
Published
2013-01-03
Volume
110
Issue
4
Pages
1279-1284
ISSN
  • 0027-8424 (ISSN-L)
  • 1091-6490
OpenAlex
W2164818222
Cited by
158
References
1
Citations by year
2026: 5 · 2025: 6 · 2024: 5 · 2023: 6 · 2022: 13 · 2021: 9 · 2020: 25 · 2019: 12 · 2018: 11 · 2017: 9 · 2016: 16 · 2015: 14 · 2014: 15 · 2013: 11
FWCI
12.48
Type
journal article
Language
en
Open access
Locations
  • Proceedings of the National Academy of Sciences (publishedVersion)
  • Europe PMC (PubMed Central) (publishedVersion)
  • PubMed Central (publishedVersion)
  • PubMed (publishedVersion)
  • Discovery Research Portal (University of Dundee) (publishedVersion, other-oa)
  • Pure Amsterdam UMC (publishedVersion)
  • White Rose Research Online (University of Leeds, The University of Sheffield, University of York) (acceptedVersion)
  • Rothamsted Repository (Rothamsted Repository) (submittedVersion)
  • Data Archiving and Networked Services (DANS) (submittedVersion)
  • Explore Bristol Research (submittedVersion)
Funders
  • Directorate for Biological Sciences
  • Biotechnology and Biological Sciences Research Council (BB/F007299/1, BBS/E/C/00004948, BB/F007108/1, BBS/E/C/00005207)
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Clinical Biochemistry › Metabolism and Genetic Disorders
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Biochemistry › Lipid metabolism and biosynthesis
  • Health Sciences › Medicine › Oncology › Drug Transport and Resistance Mechanisms
Keywords
acyl-CoA thioesterase, peroxisomal ABC transporters, long-chain acyl-CoA synthetase, fatty acid beta-oxidation, X-linked adrenoleukodystrophy, polytopic membrane proteins, coenzyme A metabolism, ATPase activity
MeSH terms
Acyl Coenzyme A, Adenosine Triphosphatases, Animals, Biological Transport, Active, Coenzyme A Ligases, Fatty Acids, Humans, Models, Biological, Recombinant Proteins, Saccharomyces cerevisiae, Thiolester Hydrolases, Mutagenesis, Site-Directed, Arabidopsis, ATP-Binding Cassette Transporters, Amino Acid Substitution, Peroxisomes, Arabidopsis Proteins, Saccharomyces cerevisiae Proteins, Plants, Genetically Modified, Fatty Acid Transport Proteins

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