The DOI matches its Crossref record.
Increasing complexity of the dystrophin-associated protein complex.
Open accessAbstract
Duchenne muscular dystrophy is a severe X chromosome-linked, muscle-wasting disease caused by lack of the protein dystrophin. The exact function of dystrophin remains to be determined. However, analysis of its interaction with a large oligomeric protein complex at the sarcolemma and the identification of a structurally related protein, utrophin, is leading to the characterization of candidate genes for other neuromuscular disorders.
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Citations
Cited by
Three muscular dystrophies: loss of cytoskeleton-extracellular matrix linkage
Campbell, K. P. (1995) · Cell · doi:10.1016/0092-8674(95)90344-5 · Cited by 829
Niethammer, M., Kim, E., & Sheng, M. (1996) · The Journal of Neuroscience · doi:10.1523/jneurosci.16-07-02157.1996 · Cited by 750
Utrophin–dystrophin-deficient mice as a model for Duchenne muscular dystrophy
Deconinck, A. E., Rafael, J. A., Skinner, J. A., Brown, S. C., Potter, A. C., Metzinger, L., Watt, D. J., Dickson, J. G., Tinsley, J. M., & Davies, K. E. (1997) · Cell · doi:10.1016/s0092-8674(00)80532-2 · Cited by 752
Self-citationSupramolecular assembly of basement membranes
Timpl, R., & Brown, J. C. (1996) · BioEssays · doi:10.1002/bies.950180208 · Cited by 699
Sudol, M., Bork, P., Einbond, A., Kastury, K., Druck, T., Negrini, M., Huebner, K., & Lehman, D. (1995) · Journal of Biological Chemistry · doi:10.1074/jbc.270.24.14733 · Cited by 342
Metadata
- Authors
- Tinsley, J. M., Blake, D. J., Zuellig, R. A., & Davies, K. E.
- Venue
- Proceedings of the National Academy of Sciences
- Published
- 1994-08-30
- Volume
- 91
- Issue
- 18
- Pages
- 8307-8313
- ISSN
- 0027-8424 (ISSN-L)
- 1091-6490
- OpenAlex
- W2072336811
- Cited by
- 193
- References
- 0
- FWCI
- 8.82
- Type
- journal article
- Language
- en
- Open access
- green
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- Tinsley, J. M., Blake, D. J., Zuellig, R. A., & Davies, K. E. (1994). Increasing complexity of the dystrophin-associated protein complex. Proceedings of the National Academy of Sciences. https://doi.org/10.1073/pnas.91.18.8307
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
