The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
A new model for the interaction of dystrophin with F-actin.
Open accessAbstract
The F-actin binding and cross-linking properties of skeletal muscle dystrophin-glycoprotein complex were examined using high and low speed cosedimentation assays, microcapillary falling ball viscometry, and electron microscopy. Dystrophin-glycoprotein complex binding to F-actin saturated near 0.042 +/- 0.005 mol/ mol, which corresponds to one dystrophin per 24 actin monomers. Dystrophin-glycoprotein complex bound to F-actin with an average apparent Kd for dystrophin of 0.5 microM. These results demonstrate that native, full-length dystrophin in the glycoprotein complex binds F-actin with some properties similar to those measured for several members of the actin cross-linking super-family of proteins. However, we failed to observe dystrophin-glycoprotein complex-induced cross-linking of F-actin by three different methods, each positively controlled with alpha-actinin. Furthermore, high speed cosedimentation analysis of dystrophin-glycoprotein complex digested with calpain revealed a novel F-actin binding site located near the middle of the dystrophin rod domain. Recombinant dystrophin fragments corresponding to the novel actin binding site and the first 246 amino acids of dystrophin both bound F-actin but with significantly lower affinity and higher capacity than was observed with purified dystrophin-glycoprotein complex. Finally, dystrophin-glycoprotein complex was observed to significantly slow the depolymerization of F-actin, Suggesting that dystrophin may lie along side an actin filament through interaction with multiple actin monomers. These data suggest that although dystrophin is most closely related to the actin cross-linking superfamily based on sequence homology, dystrophin binds F-actin in a manner more analogous to actin side-binding proteins.
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Cites
Very mild muscular dystrophy associated with the deletion of 46% of dystrophin
England, S. B., Nicholson, L. V. B., Johnson, M. A., Forrest, S. M., Love, D. R., Zubrzycka-Gaarn, E. E., Bulman, D. E., Harris, J. B., & Davies, K. E. (1990) · Nature · doi:10.1038/343180a0 · Cited by 623
Cited by
Function and Genetics of Dystrophin and Dystrophin-Related Proteins in Muscle
Blake, D. J., Weir, A., Newey, S. E., & Davies, K. E. (2002) · Physiological Reviews · doi:10.1152/physrev.00028.2001 · Cited by 1,242
Spectrin and Ankyrin-Based Pathways: Metazoan Inventions for Integrating Cells Into Tissues
Bennett, V., & Baines, A. J. (2001) · Physiological Reviews · doi:10.1152/physrev.2001.81.3.1353 · Cited by 930
Synaptopodin: An Actin-associated Protein in Telencephalic Dendrites and Renal Podocytes
Mundel, P., Heid, H. W., Mundel, T. M., Krüger, M., Reiser, J., & Kriz, W. (1997) · The Journal of Cell Biology · doi:10.1083/jcb.139.1.193 · Cited by 592
Animal models for muscular dystrophy show different patterns of sarcolemmal disruption
Straub, V., Rafael, J. A., Chamberlain, J. S., & Campbell, K. P. (1997) · The Journal of Cell Biology · doi:10.1083/jcb.139.2.375 · Cited by 488
Molecular mechanisms of muscular dystrophies: old and new players
Davies, K. E., & Nowak, K. J. (2006) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm2024 · Cited by 349
Metadata
- Authors
- Rybakova, I. N., Amann, K. J., & Ervasti, J. M.
- Venue
- The Journal of cell biology
- Publisher
- Rockefeller University Press
- Published
- 1996-11-01
- Volume
- 135
- Issue
- 3
- Pages
- 661-672
- ISSN
- 0021-9525 (ISSN-L)
- 1540-8140
- OpenAlex
- W1998841070
- Cited by
- 220
- References
- 1
- FWCI
- 4.33
- Type
- journal article
- Language
- en
- Open access
- green
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- Rybakova, I. N., Amann, K. J., & Ervasti, J. M. (1996). A new model for the interaction of dystrophin with F-actin. The Journal of cell biology. https://doi.org/10.1083/jcb.135.3.661
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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