Fueling thought: Management of glycolysis and oxidative phosphorylation in neuronal metabolism

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Items 101–200 of 368

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5. Alternative substrate metabolism depends on cerebral metabolic state following traumatic brain injury
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7. Glycolysis Paradigm Shift Dictates a Reevaluation of Glucose and Oxygen Metabolic Rates of Activated Neural Tissue
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8. Stuttering: A Disorder of Energy Supply to Neurons?
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9. Altered Energy Metabolism During Early Optic Nerve Crush Injury: Implications of Warburg-Like Aerobic Glycolysis in Facilitating Retinal Ganglion Cell Survival
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14. Astrocytic aerobic glycolysis provides lactate to support neuronal oxidative metabolism in the hippocampus
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15. Dysregulated Glucose Metabolism as a Therapeutic Target to Reduce Post-traumatic Epilepsy
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16. Glycogenolysis in Cerebral Cortex During Sensory Stimulation, Acute Hypoglycemia, and Exercise: Impact on Astrocytic Energetics, Aerobic Glycolysis, and Astrocyte-Neuron Interactions
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17. What is bipolar disorder? A disease model of dysregulated energy expenditure
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19. Major Advances in Brain Glycogen Research: Understanding of the Roles of Glycogen Have Evolved from Emergency Fuel Reserve to Dynamic, Regulated Participant in Diverse Brain Functions
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22. NMNAT2 supports vesicular glycolysis via NAD homeostasis to fuel fast axonal transport
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23. What Is the Role of Lactate in Brain Metabolism, Plasticity, and Neurodegeneration?
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24. Activation of proline biosynthesis is critical to maintain glutamate homeostasis during acute methamphetamine exposure
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26. A Warburg-like metabolic program coordinates Wnt, AMPK, and mTOR signaling pathways in epileptogenesis
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27. A coarse-grained NADH redox model enables inference of subcellular metabolic fluxes from fluorescence lifetime imaging
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28. LRRK2-mediated mitochondrial dysfunction in Parkinson’s disease
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29. Axin2 coupled excessive Wnt‐glycolysis signaling mediates social defect in autism spectrum disorders
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30. The bioenergetics of neuronal morphogenesis and regeneration: Frontiers beyond the mitochondrion
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31. Astrocyte heterogeneity within white matter tracts and a unique subpopulation of optic nerve head astrocytes
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32. Extracellular Vesicles Secreted in Response to Cytokine Exposure Increase Mitochondrial Oxygen Consumption in Recipient Cells
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33. Salidroside orchestrates metabolic reprogramming by regulating the Hif-1α signalling pathway in acute mountain sickness
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34. Metabolic adaptation to the chronic loss of Ca2+ signaling induced by KO of IP3 receptors or the mitochondrial Ca2+ uniporter
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35. Bidirectional Microbiome-Gut-Brain-Axis Communication Influences Metabolic Switch-Associated Responses in the Mosquito Anopheles culicifacies
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38. L-Glucose: Another Path to Cancer Cells
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39. Metabolomic Fingerprint of Mecp2-Deficient Mouse Cortex: Evidence for a Pronounced Multi-Facetted Metabolic Component in Rett Syndrome
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40. Unlocking the potential of photobiomodulation therapy for brain neurovascular coupling: The biological effects and medical applications
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41. Glucose-Sparing Action of Ketones Boosts Functions Exclusive to Glucose in the Brain
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42. Baseline oxygen consumption decreases with cortical depth
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43. Metabolic contributions to neuronal deficits caused by genomic disruption of schizophrenia risk gene SETD1A
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44. Platinized carbon fiber-based glucose microbiosensor designed for metabolic studies in brain slices
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45. Conservation and divergence of vulnerability and responses to stressors between human and mouse astrocytes
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46. Essential role of sulfide oxidation in brain health and neurological disorders
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47. Developmental shift to mitochondrial respiration for energetic support of sustained transmission during maturation at the calyx of Held
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48. Noncoding RNAs in the Glycolysis of Ovarian Cancer
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49. Fatty acid sensing in the brain: The role of glial-neuronal metabolic crosstalk and horizontal lipid flux
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50. The association of regional cerebral blood flow and glucose metabolism in normative ageing and insulin resistance
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51. Functional Consequences of Calcium-Dependent Synapse-to-Nucleus Communication: Focus on Transcription-Dependent Metabolic Plasticity
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52. β-amyloid monomers drive up neuronal aerobic glycolysis in response to energy stressors
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53. Metabolic partitioning in the brain and its hijacking by glioblastoma
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54. Brain microenvironment-remodeling nanomedicine improves cerebral glucose metabolism, mitochondrial activity and synaptic function in a mouse model of Alzheimer's disease
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55. Advances in research on mitochondrial dysfunction in neurodegenerative diseases
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56. Autophagy regulator ATG5 preserves cerebellar function by safeguarding its glycolytic activity
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57. A Bird's‐Eye View of Glycolytic Upregulation in Activated Brain: The Major Fate of Lactate Is Release From Activated Tissue, Not Shuttling to Nearby Neurons
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58. Metabolic interactions in the brain: the crucial roles of neurons, astrocytes, and microglia in health and disease
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59. A bioinspired carbon monoxide delivery system prevents acute kidney injury and the progression to chronic kidney disease
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60. Mechanistic stoichiometric relationship between the rates of neurotransmission and neuronal glucose oxidation: Reevaluation of and alternatives to the pseudo‐malate‐aspartate shuttle model
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61. Bioinspired nanoerythrocytes for metabolic microenvironment remodeling and long-term prognosis promoting of acute ischemic stroke
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62. Computational modeling of AMPK and mTOR crosstalk in glutamatergic synapse calcium signaling
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63. Chronic neuronal activation leads to elevated lactate dehydrogenase A through the AMP-activated protein kinase/hypoxia-inducible factor-1α hypoxia pathway
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64. Neuronal Mitochondrial Calcium Uniporter (MCU) Deficiency Is Neuroprotective in Hyperexcitability by Modulation of Metabolic Pathways and ROS Balance
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65. Energetic substrate availability regulates synchronous activity in an excitatory neural network
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76. Development of a Model to Test Whether Glycogenolysis Can Support Astrocytic Energy Demands of Na+, K+-ATPase and Glutamate-Glutamine Cycling, Sparing an Equivalent Amount of Glucose for Neurons
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83. The “protected” glucose transport through the astrocytic endoplasmic reticulum is too slow to serve as a quantitatively‐important highway for nutrient delivery
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