A Comprehensive Screen for TWIST Mutations in Patients with Craniosynostosis Identifies a New Microdeletion Syndrome of Chromosome Band 7p21.1
Open accessAbstract
Mutations in the coding region of the TWIST gene (encoding a basic helix-loop-helix transcription factor) have been identified in some cases of Saethre-Chotzen syndrome. Haploinsufficiency appears to be the pathogenic mechanism involved. To investigate the possibility that complete deletions of the TWIST gene also contribute to this disorder, we have developed a comprehensive strategy to screen for coding-region mutations and for complete gene deletions. Heterozygous TWIST mutations were identified in 8 of 10 patients with Saethre-Chotzen syndrome and in 2 of 43 craniosynostosis patients with no clear diagnosis. In addition to six coding-region mutations, our strategy revealed four complete TWIST deletions, only one of which associated with a translocation was suspected on the basis of conventional cytogenetic analysis. This case and two interstitial deletions were detectable by analysis of polymorphic microsatellite loci, including a novel (CA)n locus 7.9 kb away from TWIST, combined with FISH; these deletions ranged in size from 3.5 Mb to >11.6 Mb. The remaining, much smaller deletion was detected by Southern blot analysis and removed 2,924 bp, with a 2-bp orphan sequence at the breakpoint. Significant learning difficulties were present in the three patients with megabase-sized deletions, which suggests that haploinsufficiency of genes neighboring TWIST contributes to developmental delay. Our results identify a new microdeletion disorder that maps to chromosome band 7p21.1 and that causes a significant proportion of Saethre-Chotzen syndrome.
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No genotype-phenotype correlation is described for Saethre-Chotzen syndrome, except that large deletions are associated with learning disability and occasional C-terminal Twist box missense mutations may confer increased risk of a more non-specific synostosis phenotype with unknown penetrance.
“No genotype-phenotype correlation is described, except that large deletions are associated with learning disability (not generally a feature of Saethre-Chotzen syndrome), 26,27 and occasional missense mutations in the C-terminal Twist box apparently confer increased risk of a more non-specific synostosis phenotype (with unknown penetrance).28”
From Craniosynostosis (2011) · cites this paper as [27]
The clearest causal link between chromosome abnormalities and craniosynostosis is with deletions of 7p21.1, which includes the TWIST1 gene.
“The clearest causal link is with deletions of 7p21.1, which includes the TWIST1 gene.26,27”
From Craniosynostosis (2011) · cites this paper as [27]
Large TWIST1 deletions are linked to a higher risk of learning disability.
“large TWIST1 deletions are associated with a higher risk of learning disability.26,27”
From Craniosynostosis (2011) · cites this paper as [27]
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References 43 · Cited by 230
- Authors
- Johnson, D., Horsley, S. W., Moloney, D. M., Oldridge, M., Twigg, S. R. F., Walsh, S., Barrow, M., Njølstad, P. R., Kunz, J., Ashworth, G. J., Wall, S. A., Kearney, L., & Wilkie, A. O. M.
- Affiliations
- John Radcliffe Hospital (GB)
- Leicester Royal Infirmary (GB)
- Haukeland University Hospital (NO)
- Philipps University of Marburg (DE)
- Corresponding author
- Wilkie, Andrew O.M.
- Venue
- The American Journal of Human Genetics
- Publisher
- Elsevier BV
- Published
- 1998-11
- Volume
- 63
- Issue
- 5
- Pages
- 1282-1293
- ISSN
- 0002-9297 (ISSN-L)
- PMC
- PMC1377539
- OpenAlex
- W2074130385
- Cited by
- 230
- References
- 43
- Citations by year
- 2026: 4 · 2025: 2 · 2024: 1 · 2023: 5 · 2021: 3 · 2020: 3 · 2019: 6 · 2018: 5 · 2017: 8 · 2016: 6 · 2015: 8 · 2014: 3 · 2013: 6 · 2012: 7
- FWCI
- 5.33
- Type
- journal article
- Language
- en
- Open access
- bronze
- doi.org
- Licence
- Locations
- The American Journal of Human Genetics (publishedVersion)
- Europe PMC (PubMed Central) (publishedVersion)
- PubMed Central (publishedVersion)
- PubMed (publishedVersion)
- Funders
- Wellcome Trust
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Craniofacial Disorders and Treatments
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genomic variations and chromosomal abnormalities
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genomics and Rare Diseases
- Keywords
- twist gene, Saethre-Chotzen syndrome, craniosynostosis, haploinsufficiency, gene deletion, microsatellite markers, interstitial deletion, learning difficulties, developmental delay, bHLH transcription factors
- MeSH terms
- Acrocephalosyndactylia, Chromosome Banding, Chromosome Mapping, Chromosomes, Human, Pair 7, Craniosynostoses, Female, Genetic Markers, Humans, Karyotyping, Male, Mass Screening, Molecular Sequence Data, Nuclear Proteins, Transcription Factors, Polymerase Chain Reaction, Sequence Deletion, DNA Primers, Helix-Loop-Helix Motifs, Twist-Related Protein 1
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