The DOI matches its Crossref record.
The Regulation of Physiological Changes During Mammalian Aging
Abstract
Much evidence suggests that intrinsic molecular or cellular aging mechanisms need not be invoked to explain most age-related cellular changes and pathologcical conditions. Analysis of a widely scattered literature indicates that hormones and neural factors regulate a great number of cellular aging phenomena of mammals. It is proposed that age-related changes after maturation result from an extension of the neural and endocrine mechanisms that control earlier development and that produce a regulatory cascade of changing neural, endocrine, and target-tissue interactions.
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Senescence is presently best understood by decline of maximal function of the vital organs.
“While the process of senescence certainly has biochemical and cellular underpinnings, it is presently best understood by decline of maximal function of the vital organs (Finch 1976;Shock 1960;Strehler 1960).”
From The Compression of Morbidity (2005) · cites this paper as Finch 1976
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Memory deficits associated with senescence: A neurophysiological and behavioral study in the rat.
Barnes, C. A. (1979) · Journal of Comparative and Physiological Psychology · doi:10.1037/h0077579 · Cited by 2,088
FRIES, J. F. (2005) · The Milbank Quarterly · doi:10.1111/j.1468-0009.2005.00401.x · Cited by 449
Mann, D. M. A. (1983) · Mechanisms of Ageing and Development · doi:10.1016/0047-6374(83)90100-8 · Cited by 135
Metadata
- Authors
- Finch, C. E.
- Venue
- The Quarterly Review of Biology
- Publisher
- University of Chicago Press
- Published
- 1976-03
- Volume
- 51
- Issue
- 1
- Pages
- 49-83
- ISSN
- 0033-5770
- 1539-7718
- Cited by
- 203
- References
- 0
- Language
- en
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