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Carboxyhemoglobin Levels in Patients with Sickle-Cell Anemia: Relationship to Hemolytic and Vasoocclusive Severity
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In the citing paper
“The elevated HbCO level in SCD, as in other haemolytic diseases, is caused by an increased haem catabolism (Sears, Udden & Thomas, 2001).”
From Anaemia and cigarette smoking (2008) · cites this paper as Sears, Udden & Thomas, 2001
In the citing paper
“This is supported by findings that cigarette smokers have an elevated level of HbCO compared with nonsmokers but no correlation between HbCO levels and vaso-occlusive severity could be detected (Sears, Udden & Thomas, 2001).”
From Anaemia and cigarette smoking (2008) · cites this paper as Sears, Udden & Thomas, 2001
In the citing paper
“Sears, Udden and Thomas (2001) described a positive correlation between elevated HbCO levels and percentage of irreversibly sickled cells.”
From Anaemia and cigarette smoking (2008) · cites this paper as Sears, Udden & Thomas, 2001
In the citing paper
“The increase in endogenous HbCO production (Sjostrand, 1949) is in close correlation with bilirubin or number of reticulocytes and therefore correlates with the extent of haemolysis (Sears, Udden & Thomas, 2001).”
From Anaemia and cigarette smoking (2008) · cites this paper as Sears, Udden & Thomas, 2001
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Research works printed but not linked: 16
- Ernst, 1998, Carbon monoxide poisoning, N Engl J Med, 339, 1603, 10.1056/NEJM199811263392206 · DOI
- Roughton, 1944, The effect of carbon monoxide on the oxyhemoglobin dissociation curve, Am J Physiol, 141, 17, 10.1152/ajplegacy.1944.141.1.17 · DOI · Paper page
- Coburn, 1965, Considerations of the physiological variables that determine the blood carboxyhemoglobin concentration in man, J Clin Invest, 44, 1899, 10.1172/JCI105296 · DOI
- Nordenberg, 1990, The effect of cigarette smoking on hemoglobin levels and anemia screening, JAMA, 264, 1556, 10.1001/jama.1990.03450120068031 · DOI · Paper page
- Sjostrand, 1949, Endogenous formation of carbon monoxide in man under normal and pathological conditions, Scand J Clin Lab Invest, 1, 201, 10.3109/00365514909069943 · DOI
- Dinman, 1970, Effects of carbon monoxide on DPG concentrations in the erythrocyte, Ann NY Acad Sci, 174, 246, 10.1111/j.1749-6632.1970.tb49791.x · DOI
- McCurdy, 1978, Irreversibly sickled cells and red cell survival in sickle cell anemia. A study with both DF32P and 51Cr, Am J Med, 64, 253, 10.1016/0002-9343(78)90053-0 · DOI
- Steinberg, 1973, Mild sickle cell disease. Clinical and laboratory studies, JAMA, 224, 317, 10.1001/jama.1973.03220160021005 · DOI
- Lux, 1976, Irreversible deformation of the spectrin-actin lattice in irreversibly sickled cells, J Clin Invest, 58, 955, 10.1172/JCI108549 · DOI
- Zwart, 1981, Determination of hemoglobin derivatives with the IL 282 co-oximeter as compared with a manual spectrophotometric five-wavelength method, Clin Chem, 27, 1903, 10.1093/clinchem/27.11.1903 · DOI
- Bensinger, 1974, Hemolysis in sickle cell disease, Arch Intern Med, 133, 624, 10.1001/archinte.1974.00320160118010 · DOI
- Beutler, 1975, The effect of carbon monoxide on red cell life span in sickle cell disease, Blood, 46, 253, 10.1182/blood.V46.2.253.253 · DOI
- Young, 1988, Lung function in sickle cell hemoglobinpoathy patients compared with healthy subjects, J Natl Med Assn, 80, 599
- Sirs, 1963, The use of carbon monoxide to prevent sickle-cell formation, Lancet, 1, 971, 10.1016/S0140-6736(63)91751-3 · DOI
- Purugganan, 1964, Failure of carbonmonoxy sickle-cell haemoglobin to alter the sickle state, Lancet, 1, 79, 10.1016/S0140-6736(64)91394-7 · DOI
- Beutler, 1974, Hypothesis: Changes in the O2 dissociation curve and sickling: a general formulation and therapeutic strategy, Blood, 43, 297, 10.1182/blood.V43.2.297.297 · DOI
Cited by
Sickle Hemoglobin Confers Tolerance to Plasmodium Infection
Ferreira, A., Marguti, I., Bechmann, I., Jeney, V., Chora, Â., Palha, N. R., Rebelo, S., Henri, A., Beuzard, Y., & Soares, M. P. (2011) · Cell · doi:10.1016/j.cell.2011.03.049 · Cited by 340
LEIFERT, J. A. (2008) · International Journal of Laboratory Hematology · doi:10.1111/j.1751-553x.2008.01067.x · Cited by 83
Metadata
- Authors
- Sears, D. A., Udden, M. M., & Thomas, L. J.
- Venue
- The American Journal of the Medical Sciences
- Publisher
- Elsevier BV
- Published
- 2001-12
- Volume
- 322
- Issue
- 6
- Pages
- 345-348
- ISSN
- 0002-9629
- Cited by
- 33
- References
- 0
- Language
- en
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.