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IL-33: a tissue derived cytokine pathway involved in allergic inflammation and asthma

Smith, D. E.·2010·Clinical & Experimental Allergy
DOI 10.1111/j.1365-2222.2009.03384.x
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Abstract

Summary: Besides classic T cell‐derived T‐helper type 2 (Th2) cytokines such as IL‐4, IL‐5 and IL‐13, tissue‐produced cytokines such as thymic stromal lymphopoietin, IL‐25 and IL‐33 are now recognized as important contributors to allergic inflammation. IL‐33 is produced by various tissue dwelling cells and broadly enhances allergic inflammation through its effects on hematopoietic cell types. The environmental or endogenous triggers that provoke IL‐33 cellular release may be associated with infection, inflammation or tissue damage. This review summarizes the wide range of documented IL‐33 activities on human cellular mediators of inflammation as well as genetic evidence that IL‐33 contributes to disease. Finally, there will be a discussion of still unanswered questions regarding the mechanisms by which cytokine activity is generated and IL‐33's relationship with other Th2‐associated cytokines. Cite this as: D. E. Smith, Clinical & Experimental Allergy, 2010 (40) 200–208.

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Metadata

Authors
Smith, D. E.
Venue
Clinical & Experimental Allergy
Publisher
Wiley
Published
2010-01-11
Volume
40
Issue
2
Pages
200-208
ISSN
  • 0954-7894
  • 1365-2222
DOI
10.1111/j.1365-2222.2009.03384.x
Cited by
173
References
4
Language
en
Licence
onlinelibrary.wiley.com/termsAndConditions#vor (vor)

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