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Cited by 11T-cell and Retrovirus StudiesViral-associated cancers and disordersHIV Research and Treatment

The DOI matches its Crossref record.

Feedback Loop Regulation between Pim Kinases and Tax Keeps Human T-Cell Leukemia Virus Type 1 Viral Replication in Check

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Bellon, Marcia, Nicot, Christophe·2022·Journal of Virology
DOI 10.1128/jvi.01960-21PMC PMC8826812
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Abstract

The Pim family of protein kinases have established pro-oncogenic functions. They are often upregulated in cancer; especially leukemias and lymphomas.

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Crossref · 10.1128/jvi.01960-21

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Metadata

Authors
Bellon, M., & Nicot, C.
Affiliations
University of Kansas Medical Center (US)
Corresponding author
Bellon, Marcia
ORCID
Nicot, Christophe
Venue
Journal of Virology
Publisher
American Society for Microbiology
Published
2022-02-09
Volume
96
Issue
3
Pages
e0196021-e0196021
ISSN
  • 0022-538X (ISSN-L)
  • 1098-5514
DOI
10.1128/jvi.01960-21
PMC
PMC8826812
OpenAlex
W3215172558
Cited by
11
References
0
Citations by year
2026: 1 · 2025: 2 · 2024: 4 · 2023: 3 · 2022: 1
FWCI
0.75
Type
journal article
Language
en
Open access
Licence
Locations
  • Journal of Virology (publishedVersion)
  • PubMed Central (publishedVersion)
  • PubMed (publishedVersion)
Funders
HHS | NIH | National Cancer Institute (CA201309)
Topics
  • Life Sciences › Immunology and Microbiology › Immunology › T-cell and Retrovirus Studies
  • Health Sciences › Medicine › Oncology › Viral-associated cancers and disorders
  • Life Sciences › Immunology and Microbiology › Virology › HIV Research and Treatment
Keywords
PIM kinases, HTLV-1, taxation, PIM-1, PIM2, PIM3, T-cell leukemia, viral replication, viral latency, protein half-life, heat shock, immune evasion, oncogenic transformation, viral persistence
MeSH terms
Cell Line, Disease Susceptibility, Humans, Models, Molecular, Transcription Factors, Virus Replication, Human T-lymphotropic virus 1, HTLV-I Infections, Gene Expression Regulation, Viral, Gene Products, tax, HSP90 Heat-Shock Proteins, Terminal Repeat Sequences, Proteasome Endopeptidase Complex, Proto-Oncogene Proteins c-pim-1, Host-Pathogen Interactions, Proteolysis

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