Cited by 109Gut microbiota and healthDrug Transport and Resistance MechanismsLiver Disease and Transplantation

Gut microbiota, cirrhosis, and alcohol regulate bile acid metabolism in the gut

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Ridlon, Jason M., Kang, Dae-Joong, Hylemon, Phillip B., Bajaj, Jasmohan S.·2015·Digestive Diseases

Abstract

The understanding of the complex role of the bile acid-gut microbiome axis in health and disease processes is evolving rapidly. Our focus revolves around the interaction of the gut microbiota with liver diseases, especially cirrhosis. The bile acid pool size has recently been shown to be a function of microbial metabolism of bile acid, and regulation of the microbiota by bile acids is important in the development and progression of several liver diseases. Humans produce a large, conjugated hydrophilic bile acid pool, maintained through positive-feedback antagonism of farnesoid X receptor (FXR) in the intestine and liver. Microbes use bile acids, and via FXR signaling this results in a smaller, unconjugated hydrophobic bile acid pool. This equilibrium is critical to maintain health. The challenge is to examine the manifold functions of gut bile acids as modulators of antibiotic, probiotic, and disease progression in cirrhosis, metabolic syndrome, and alcohol use. Recent studies have shown potential mechanisms explaining how perturbations in the microbiome affect bile acid pool size and composition. With advancing liver disease and cirrhosis, there is dysbiosis in the fecal, ileal, and colonic mucosa, in addition to a decrease in bile acid concentration in the intestine due to the liver problems. This results in a dramatic shift toward the Firmicutes, particularly Clostridium cluster XIVa, and increasing production of deoxycholic acid. Alcohol intake speeds up these processes in the subjects with and without cirrhosis without significant FXR feedback. Taken together, these pathways can impact intestinal and systemic inflammation while worsening dysbiosis. The interaction between bile acids, alcohol, cirrhosis, and dysbiosis is an important relationship that influences intestinal and systemic inflammation, which in turn determines progression of the overall disease process. These interactions and the impact of commonly used therapies for liver disease can provide insight into the pathogenesis of inflammation in humans.

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  • Clostridium scindens belongs to the genus Lachnoclostridium and is known to convert CDCA to UDCA by oxidation and epimerization of specific hydroxy groups.

    “Clostridium scindens belongs to the genus Lachnoclostridium,46 and is known to convert CDCA to UDCA by oxidation and epimerization of specific hydroxy groups47; it may explain our findings in correlation analysis.”

    From Gut microbiota and metabolites associate with outcomes of immune checkpoint inhibitor–treated unresectable hepatocellular carcinoma (2022) · cites this paper as [46]

    The section “Modification of Bile Acids by Microbiota in Healthy Individuals” (p. 4) describes conversion of chenodeoxycholic acid (CDCA) to UDCA by epimerization of its 7α-hydroxy group. It identifies Clostridium absonum as a species with both relevant enzyme activities, but does not say that C. scindens performs this conversion. The separate genus claim is attached to citation 46, not the citation being audited. (LLM, conf=0.85)

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References 38 · Cited by 109

Research works printed but not linked: 1

  1. 8 Reddy BS, Wynder EL: Metabolic epidemiology of colon cancer: fecal bile acids and neutral sterols in colon cancer patients with adenomatous polyps. Cancer 1977; 39: 25332539.

Authors
Ridlon, J. M., Kang, D.-J., Hylemon, P. B., & Bajaj, J. S.
Affiliations
  • Virginia Commonwealth University (US)
  • Hunter Holmes McGuire VA Medical Center (US)
Corresponding author
Bajaj, Jasmohan S.
Venue
Digestive Diseases
Publisher
S. Karger AG
Published
2015
Volume
33
Issue
3
Pages
338-345
ISSN
  • 0257-2753 (ISSN-L)
  • 1421-9875
OpenAlex
W1605734368
Cited by
109
References
38
Citations by year
2026: 9 · 2025: 4 · 2024: 12 · 2023: 14 · 2022: 10 · 2021: 14 · 2020: 12 · 2019: 10 · 2018: 9 · 2017: 8 · 2016: 7
FWCI
3.47
Type
journal article
Language
en
Open access
Locations
  • Digestive Diseases (publishedVersion)
  • PubMed (publishedVersion)
  • Europe PMC (PubMed Central) (acceptedVersion)
  • PubMed Central (acceptedVersion)
Funders
  • U.S. Department of Veterans Affairs (I01 CX001076)
  • National Institute on Alcohol Abuse and Alcoholism (R01AA020203)
  • National Institute of Diabetes and Digestive and Kidney Diseases (R01DK087913)
Topics
  • Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Gut microbiota and health
  • Health Sciences › Medicine › Oncology › Drug Transport and Resistance Mechanisms
  • Health Sciences › Medicine › Hepatology › Liver Disease and Transplantation
Keywords
gut microbiota, liver cirrhosis, bile acid metabolism, farnesoid X receptor, dysbiosis, alcohol consumption, deoxycholic acid, Clostridium cluster XIVa, Firmicutes, intestinal inflammation, systemic inflammation, liver disease progression, probiotics, antibiotic therapy
MeSH terms
Gastrointestinal Microbiome, Alcohol Drinking, Ethanol, Bile Acids and Salts, Feces, Humans, Liver Cirrhosis, Signal Transduction, Probiotics, Dysbiosis

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