Cited by 176

The DOI matches its Crossref record.

The low density lipoprotein receptor is not required for normal catabolism of Lp(a) in humans.

Rader, D J, Mann, W A, Cain, W, Kraft, H G, Usher, D, Zech, L A, Hoeg, J M, Davignon, J, Lupien, P, Grossman, M·1995·Journal of Clinical Investigation

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Crossref · 10.1172/jci117794

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Research works printed but not linked: 3

  1. Utermann, G., F. Hoppichler, H. Dieplinger, M. Seed, G. Thompson, and E. Boerwinkle. 1989. Defects in the low density lipoprotein receptor gene affect lipoprotein (a) levels: multiplicative interaction of two gene loci associated with premature atherosclerosis. Proc. Natl. Acad. Sci. USA. 86:4171-4174.
  2. Brewer, H. B., Jr. 1990. Effectiveness of diet and drugs in the treatment of patients with elevated Lp(a) levels. In Lipoprotein(a). A. M. Scanu, editor. Academic Press, Inc., New York. 211-220.
  3. Eaton, D. L., G. M. Fless, W. J. Kohr, J. W. McLean, Q. T. Xu, C. G. Miller, R. M. Lawn, and A. M. Scanu. 1987. Partial amino acid sequence of apolipoprotein(a) shows that it is homologous to plasminogen. Proc. Natl. Acad. Sci. USA. 84:3224-3228.

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Metadata

Authors
Rader, D. J., Mann, W. A., Cain, W., Kraft, H. G., Usher, D., Zech, L. A., Hoeg, J. M., Davignon, J., Lupien, P., & Grossman, M.
Venue
Journal of Clinical Investigation
Published
1995-03-01
Volume
95
Issue
3
Pages
1403-1408
ISSN
0021-9738
Cited by
176
References
0
Language
en

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