The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
BCR/ABL induces multiple abnormalities of cytoskeletal function
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Claims other papers make about this one
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In the citing paper
“p210-BCR-ABL-transduced Ba/F3 cells exhibit increased F-actin staining and an increased formation of filopodia and pseudopodia, reflecting elevated Rac1 activity (Salgia et al., 1997).”
From Rac Guanosine Triphosphatases Represent Integrating Molecular Therapeutic Targets for BCR-ABL-Induced Myeloproliferative Disease (2007) · cites this paper as Salgia et al., 1997
In the citing paper
“Expression of p210-BCR-ABL confers proliferative advantage to HSC, induces abnormal adhesion and migration of hematopoietic progenitors, and appears to be directly responsible for the development of a transformed phenotype.4,5”
From Rac2 GTPase deficiency depletes BCR-ABL+ leukemic stem cells and progenitors in vivo (2010) · cites this paper as [4]
In the citing paper
“Expression of p210-BCR-ABL confers proliferative advantage to HSC, induces abnormal adhesion and migration of hematopoietic progenitors, and appears to be directly responsible for the development of a transformed phenotype.4,5”
From Rac2 GTPase deficiency depletes BCR-ABL+ leukemic stem cells and progenitors in vivo (2010) · cites this paper as [4]
In the citing paper
“Expression of p210-BCR-ABL confers proliferative advantage to HSC, induces abnormal adhesion and migration of hematopoietic progenitors, and appears to be directly responsible for the development of a transformed phenotype.4,5”
From Rac2 GTPase deficiency depletes BCR-ABL+ leukemic stem cells and progenitors in vivo (2010) · cites this paper as [4]
Self-check
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Citations
Cites
ROWLEY, J. D. (1973) · Nature · doi:10.1038/243290a0 · Cited by 4,291
Research works printed but not linked: 1
- Daley, G., and D. Baltimore. 1988. Transformation of an interleu- kin-3-dependent hematopoietic cell line by the chronic myelogenous leukemia- specific p210 BCR/ABL protein. Proc. Natl. Acad. Sci. USA. 85:9312-9316.
Cited by
Thomas, E. K., Cancelas, J. A., Chae, H.-D., Cox, A. D., Keller, P. J., Perrotti, D., Neviani, P., Druker, B. J., Setchell, K. D. R., Zheng, Y., et al. (2007) · Cancer Cell · doi:10.1016/j.ccr.2007.10.015 · Cited by 147
Rac2 GTPase deficiency depletes BCR-ABL+ leukemic stem cells and progenitors in vivo
Sengupta, A., Arnett, J., Dunn, S., Williams, D. A., & Cancelas, J. A. (2010) · Blood · doi:10.1182/blood-2009-10-247437 · Cited by 52
Metadata
- Authors
- Salgia, R., Li, J. L., Ewaniuk, D. S., Pear, W., Pisick, E., Burky, S. A., Ernst, T., Sattler, M., Chen, L. B., & Griffin, J. D.
- Venue
- Journal of Clinical Investigation
- Published
- 1997-07-01
- Volume
- 100
- Issue
- 1
- Pages
- 46-57
- ISSN
- 0021-9738
- Cited by
- 147
- References
- 1
- Language
- en
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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- CoChat. (2026). Citation check: BCR/ABL induces multiple abnormalities of cytoskeletal function. https://app.cochat.ai/papers/10.1172/jci119520
- The paper
- Salgia, R., Li, J. L., Ewaniuk, D. S., Pear, W., Pisick, E., Burky, S. A., Ernst, T., Sattler, M., Chen, L. B., & Griffin, J. D. (1997). BCR/ABL induces multiple abnormalities of cytoskeletal function. Journal of Clinical Investigation. https://doi.org/10.1172/jci119520
