The DOI matches its Crossref record; 41 of the 41 references it cites were found in a registry or an index.
Prostaglandin E2 Stimulates Aromatase Expression in Endometriosis-Derived Stromal Cells
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In the citing paper
“which have the capacity to stimulate expression of the prostaglandin synthesis enzyme COX-2 and increase secretion of prostaglandin E2 (PGE2) by endometriotic cells and peritoneal macrophages [72,73].”
From Endometrial Intracrinology: Oestrogens, Androgens and Endometrial Disorders (2018) · cites this paper as [72]
In the citing paper
“PGE2 in turn stimulates the production of cyclic AMP (cAMP), which, together with steroidogenic factor 1 (SF1), whose expression is aberrantly upregulated in endometriotic tissue compared to endometrial tissue, induces expression of mRNAs that encode enzymes that play a critical role in the steroidogenic enzyme machinery, including STAR, 17-hydroxylase/17,20-lyase, 3β-HSD and aromatase, which may”
From Endometrial Intracrinology: Oestrogens, Androgens and Endometrial Disorders (2018) · cites this paper as [72]
In the citing paper
“PGE2 activates the protein kinase A (PKA) signaling pathway via raising the intracellular levels of cyclic adenosine 3′,5′-monophosphate (cAMP)12–14, which can enhance the binding of steroidogenic factor-1 (SF-1) to promoters of these steroidogenic genes15, and induce phosphorylation of the transcriptional activator cAMP-response element-binding protein (CREB)16.”
From Platelets induce increased estrogen production through NF-κB and TGF-β1 signaling pathways in endometriotic stromal cells (2020) · cites this paper as [12]
In the citing paper
“endometriotic stromal cells, in contrast, do produce a much higher level of estrogens, due to demethylated aromatase42, and overexpression of StAR, HSD3B2 and HSD17B112,43,45,47.”
From Platelets induce increased estrogen production through NF-κB and TGF-β1 signaling pathways in endometriotic stromal cells (2020) · cites this paper as [12]
In the citing paper
“as it has been demonstrated that prostaglandins are among the most potent inducers of aromatase activity in endometriotic cells (Noble et al., 1997)”
From “Waiting for Godot”: a commonsense approach to the medical treatment of endometriosis (2011) · cites this paper as Noble et al., 1997
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Metadata
- Authors
- Noble, L. S.
- Corresponding author
- Noble, L. S.
- Venue
- Journal of Clinical Endocrinology & Metabolism
- Publisher
- The Endocrine Society
- Published
- 1997-02-01
- Volume
- 82
- Issue
- 2
- Pages
- 600-606
- ISSN
- 0021-972X (ISSN-L)
- OpenAlex
- W4232127353
- Cited by
- 301
- References
- 0
- Citations by year
- 2025: 8 · 2024: 7 · 2023: 8 · 2022: 13 · 2021: 9 · 2020: 10 · 2019: 9 · 2018: 6 · 2017: 7 · 2016: 13 · 2015: 15 · 2014: 8 · 2013: 12 · 2012: 14
- FWCI
- 5.74
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- The Journal of Clinical Endocrinology & Metabolism (publishedVersion)
- Topics
- Health Sciences › Medicine › Reproductive Medicine › Endometriosis Research and Treatment
- Health Sciences › Medicine › Pharmacology (medical) › Inflammatory mediators and NSAID effects
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Endocrinology › Estrogen and related hormone effects
- Keywords
- prostaglandin E2, aromatase expression, endometriosis, aromatase induction, eutopic endometrium
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
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